A G/C element mediates repression of the SM22alpha promoter within phenotypically modulated smooth muscle cells in experimental atherosclerosis.
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The LIM protein leupaxin is enriched in smooth muscle and functions as an serum response factor cofactor to induce smooth muscle cell gene transcriptionSpecificity protein-1 as a critical regulator of human cystathionine gamma-lyase in smooth muscle cellsKruppel-like factor 4, Elk-1, and histone deacetylases cooperatively suppress smooth muscle cell differentiation markers in response to oxidized phospholipids.KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis.Recent insights into the cellular biology of atherosclerosisTNF-α induces phenotypic modulation in cerebral vascular smooth muscle cells: implications for cerebral aneurysm pathology.KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formationSmooth muscle cell plasticity: fact or fiction?Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Cigarette smoke modulates vascular smooth muscle phenotype: implications for carotid and cerebrovascular diseaseSmooth muscle cell phenotypic switching in atherosclerosisInterleukin-1β modulates smooth muscle cell phenotype to a distinct inflammatory state relative to PDGF-DD via NF-κB-dependent mechanisms.Genome-wide microarray analyses identify the protein C receptor as a novel calcineurin/nuclear factor of activated T cells-dependent gene in vascular smooth muscle cell phenotypic modulation.Nuclear factor of activated T cells 5 regulates vascular smooth muscle cell phenotypic modulation.Chromatin immunoprecipitation (ChIP): revisiting the efficacy of sample preparation, sonication, quantification of sheared DNA, and analysis via PCR.Cyclosporine up-regulates Krüppel-like factor-4 (KLF4) in vascular smooth muscle cells and drives phenotypic modulation in vivo.Smooth muscle phenotypic modulation is an early event in aortic aneurysmsSp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.PDGF-DD, a novel mediator of smooth muscle cell phenotypic modulation, is upregulated in endothelial cells exposed to atherosclerosis-prone flow patternsControl of SRF binding to CArG box chromatin regulates smooth muscle gene expression in vivo.Molecular mechanisms of collagen isotype-specific modulation of smooth muscle cell phenotypeIncreased smooth muscle cell activation and neointima formation in response to injury in AIF-1 transgenic miceMultiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells.Vascular smooth muscle cells in cerebral aneurysm pathogenesis.Smooth muscle cell-specific Notch1 haploinsufficiency restricts the progression of abdominal aortic aneurysm by modulating CTGF expression.Vascular-directed tissue factor pathway inhibitor overexpression regulates plasma cholesterol and reduces atherosclerotic plaque developmentA proteomic focus on the alterations occurring at the human atherosclerotic coronary intima.Disruption of SM22 promotes inflammation after artery injury via nuclear factor kappaB activation.Role of somatic mutations in vascular disease formation.Store-operated Ca(2+) entry is not essential for PDGF-BB induced phenotype modulation in rat aortic smooth muscleTransgenic overexpression of pregnancy-associated plasma protein-A in murine arterial smooth muscle accelerates atherosclerotic lesion developmentVascular wall extracellular matrix proteins and vascular diseasesArterial injury promotes medial chondrogenesis in Sm22 knockout mice.Attenuation of chondrogenic transformation in vascular smooth muscle by dietary quercetin in the MGP-deficient mouse modelDevelopment of viral vectors for use in cardiovascular gene therapy.Prevention of atherosclerosis by Yindan Xinnaotong capsule combined with swimming in ratsS100A12 in vascular smooth muscle accelerates vascular calcification in apolipoprotein E-null mice by activating an osteogenic gene regulatory programDefining smooth muscle cells and smooth muscle injury.Reciprocal regulation controlling the expression of CPI-17, a specific inhibitor protein for the myosin light chain phosphatase in vascular smooth muscle cells.Constitutive expression of pregnancy-associated plasma protein-A in arterial smooth muscle reduces the vascular response to injury in vivo.
P2860
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P2860
A G/C element mediates repression of the SM22alpha promoter within phenotypically modulated smooth muscle cells in experimental atherosclerosis.
description
2004 nî lūn-bûn
@nan
2004 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
A G/C element mediates repress ...... experimental atherosclerosis.
@ast
A G/C element mediates repress ...... experimental atherosclerosis.
@en
type
label
A G/C element mediates repress ...... experimental atherosclerosis.
@ast
A G/C element mediates repress ...... experimental atherosclerosis.
@en
prefLabel
A G/C element mediates repress ...... experimental atherosclerosis.
@ast
A G/C element mediates repress ...... experimental atherosclerosis.
@en
P2093
P1433
P1476
A G/C element mediates repress ...... n experimental atherosclerosis
@en
P2093
B R Wamhoff
O G McDonald
P304
P356
10.1161/01.RES.0000147961.09840.FB
P577
2004-10-14T00:00:00Z