In vivo silencing of the Ca(V)3.2 T-type calcium channels in sensory neurons alleviates hyperalgesia in rats with streptozocin-induced diabetic neuropathy.
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The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialDiabetic neuropathic pain: Physiopathology and treatmentRedox-Dependent Modulation of T-Type Ca(2+) Channels in Sensory Neurons Contributes to Acute Anti-Nociceptive Effect of Substance PT-type calcium channels contribute to colonic hypersensitivity in a rat model of irritable bowel syndromeTargeting of CaV3.2 T-type calcium channels in peripheral sensory neurons for the treatment of painful diabetic neuropathyCellular and Molecular Mechanisms of PainNew Horizons in Diabetic Neuropathy: Mechanisms, Bioenergetics, and Pain.Painful Diabetic Neuropathy: Prevention or Suppression?Reversal of neuropathic pain in diabetes by targeting glycosylation of Ca(V)3.2 T-type calcium channelsImmunohistological demonstration of CaV3.2 T-type voltage-gated calcium channel expression in soma of dorsal root ganglion neurons and peripheral axons of rat and mouse.CaV3.2 T-type calcium channels in peripheral sensory neurons are important for mibefradil-induced reversal of hyperalgesia and allodynia in rats with painful diabetic neuropathy.Presynaptic Cav3.2 channels regulate excitatory neurotransmission in nociceptive dorsal horn neuronsTTA-P2 is a potent and selective blocker of T-type calcium channels in rat sensory neurons and a novel antinociceptive agent.T-type voltage-gated calcium channels as targets for the development of novel pain therapies.Neuropathic pain: role for presynaptic T-type channels in nociceptive signaling.CRMP-2 peptide mediated decrease of high and low voltage-activated calcium channels, attenuation of nociceptor excitability, and anti-nociception in a model of AIDS therapy-induced painful peripheral neuropathyAnalgesic effect of a mixed T-type channel inhibitor/CB2 receptor agonist.NMP-7 inhibits chronic inflammatory and neuropathic pain via block of Cav3.2 T-type calcium channels and activation of CB2 receptors.Small organic molecule disruptors of Cav3.2 - USP5 interactions reverse inflammatory and neuropathic pain.Diabetic neuropathy enhances voltage-activated Ca2+ channel activity and its control by M4 muscarinic receptors in primary sensory neurons.Upregulation of T-type Ca2+ channels in long-term diabetes determines increased excitability of a specific type of capsaicin-insensitive DRG neurons.Pain modality and spinal glia expression by streptozotocin induced diabetic peripheral neuropathy in ratsTopical application of disodium isostearyl 2-O-L-ascorbyl phosphate, an amphiphilic ascorbic acid derivative, reduces neuropathic hyperalgesia in rats.LncRNA uc.48+ is involved in diabetic neuropathic pain mediated by the P2X3 receptor in the dorsal root ganglia.Involvement of the endogenous hydrogen sulfide/Ca(v) 3.2 T-type Ca2+ channel pathway in cystitis-related bladder pain in mice.Pathophysiological implication of CaV3.1 T-type Ca2+ channels in trigeminal neuropathic pain.Inflammatory mediator bradykinin increases population of sensory neurons expressing functional T-type Ca(2+) channels.Assessment of the effectiveness and safety of Ethosuximide in the Treatment of non-Diabetic Peripheral Neuropathic Pain: EDONOT-protocol of a randomised, parallel, controlled, double-blinded and multicentre clinical trial.Nociceptive neurons differentially express fast and slow T-type Ca²⁺ currents in different types of diabetic neuropathy.Abnormal calcium homeostasis in peripheral neuropathies.Calcium channel functions in pain processingPharmacological treatment of diabetic neuropathic pain.The design and discovery of T-type calcium channel inhibitors for the treatment of central nervous system disorders.T-type Ca2+ channels in normal and abnormal brain functions.Calcium-permeable ion channels in pain signaling.T-type calcium channels in chronic pain: mouse models and specific blockers.Neuronal calcium signaling in chronic pain.A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain.Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociception.A Randomized, Double-Blind, Placebo-Controlled, Crossover Study of the T-Type Calcium Channel Blocker ABT-639 in an Intradermal Capsaicin Experimental Pain Model in Healthy Adults.
P2860
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P2860
In vivo silencing of the Ca(V)3.2 T-type calcium channels in sensory neurons alleviates hyperalgesia in rats with streptozocin-induced diabetic neuropathy.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@ast
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@en
type
label
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@ast
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@en
prefLabel
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@ast
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@en
P2093
P2860
P1433
P1476
In vivo silencing of the Ca(V) ...... n-induced diabetic neuropathy.
@en
P2093
Ajit K Naik
Janelle R Latham
Michael T Nelson
Miljen M Jagodic
Peihan Orestes
Richard B Messinger
Slobodan M Todorovic
Woo Yong Lee
P2860
P304
P356
10.1016/J.PAIN.2009.06.012
P407
P577
2009-07-03T00:00:00Z