MHC class I Dk locus and Ly49G2+ NK cells confer H-2k resistance to murine cytomegalovirus - Wikidata - wikimedia - TriplyDB

MHC class I Dk locus and Ly49G2+ NK cells confer H-2k resistance to murine cytomegalovirus

MHC class I Dk locus and Ly49G2+ NK cells confer H-2k resistance to murine cytomegalovirus

http://www.wikidata.org/entity/Q30435996

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Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience Genomic Modifiers of Natural Killer Cells, Immune Responsiveness and Lymphoid Tissue Remodeling Together Increase Host Resistance to Viral Infection Genome-Wide Exome Analysis of Cmv5-Disparate Mouse Strains that Differ in Host Resistance to Murine Cytomegalovirus Infection.Murine Cytomegalovirus Disrupts Splenic Dendritic Cell Subsets via Type I Interferon-Dependent and -Independent Mechanisms.Acute Virus Control Mediated by Licensed NK Cells Sets Primary CD8+ T Cell Dependence on CD27 Costimulation.Multiparametric analysis of host response to murine cytomegalovirus in MHC class I-disparate mice reveals primacy of Dk-licensed Ly49G2+ NK cells in viral control.Know Thyself: NK-Cell Inhibitory Receptors Prompt Self-Tolerance, Education, and Viral Control.Rapid discrimination of MHC class I and killer cell lectin-like receptor allele variants by high-resolution melt analysis.Self MHC class I-licensed NK cells enhance adaptive CD8 T-cell viral immunity.NK gene complex and chromosome 19 loci enhance MHC resistance to murine cytomegalovirus infection MHC class I D(k) expression in hematopoietic and nonhematopoietic cells confers natural killer cell resistance to murine cytomegalovirus Genetic approach to study lupus glomerulonephritis.Cytomegalovirus immunoevasin reveals the physiological role of "missing self" recognition in natural killer cell dependent virus control in vivo.Distinct MHC class I-dependent NK cell-activating receptors control cytomegalovirus infection in different mouse strains.Mouse Ly49G2+ NK cells dominate early responses during both immune reconstitution and activation independently of MHC The Attenuated Live Yellow Fever Virus 17D Infects the Thymus and Induces Thymic Transcriptional Modifications of Immunomodulatory Genes in C57BL/6 and BALB/C Mice.The impact of Ly49-NK cell-dependent recognition of MCMV infection on innate and adaptive immune responses All is fair in virus-host interactions: NK cells and cytomegalovirus.MCMV avoidance of recognition and control by NK cells.

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P2860

MHC class I Dk locus and Ly49G2+ NK cells confer H-2k resistance to murine cytomegalovirus

http://www.wikidata.org/entity/Q30435996

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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type

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MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

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P356

JIMMUNOL.0803933

P1433

Journal of Immunology

P1476

MHC class I Dk locus and Ly49G ...... ance to murine cytomegalovirus

@en

P2093

Michael D Stadnisky

http://www.w3.org/2001/XMLSchema#string

Michael G Brown

http://www.w3.org/2001/XMLSchema#string

Xuefang Xie

http://www.w3.org/2001/XMLSchema#string

P2860

Natural killer cells promote early CD8 T cell responses against cytomegalovirus

Class I MHC-binding characteristics of the 129/J Ly49 repertoire

NK cell recognition

Natural killer gene complex (Nkc) allelic variability in inbred mice: evidence for Nkc haplotypes.

Cmv1-independent antiviral role of NK cells revealed in murine cytomegalovirus-infected New Zealand White mice.

NK gene complex dynamics and selection for NK cell receptors.

Requisite H2k role in NK cell-mediated resistance in acute murine cytomegalovirus-infected MA/My mice.

Deficient major histocompatibility complex-linked innate murine cytomegalovirus immunity in MA/My.L-H2b mice and viral downregulation of H-2k class I proteins.

Cytomegaloviral control of MHC class I function in the mouse.

Epistasis between mouse Klra and major histocompatibility complex class I loci is associated with a new mechanism of natural killer cell-mediated innate resistance to cytomegalovirus infection.

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7163-7171

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scholarly article

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10.4049/JIMMUNOL.0803933

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11

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182

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2009-06-01T00:00:00Z

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24439378

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19454713

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Cytomegalovirus

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2730597

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