Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration. - Wikidata - wikimedia - TriplyDB

Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration.

Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration.

http://www.wikidata.org/entity/Q30436774

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Mammalian Krüppel-like factors in health and diseases Regulation of an inflammatory disease: Krüppel-like factors and atherosclerosis Implications of autophagy for vascular smooth muscle cell function and plasticity Txnip ablation reduces vascular smooth muscle cell inflammation and ameliorates atherosclerosis in apolipoprotein E knockout mice Lipid peroxidation generates biologically active phospholipids including oxidatively N-modified phospholipids Generation and biological activities of oxidized phospholipids Smooth muscle cell-specific deletion of Col15a1 unexpectedly leads to impaired development of advanced atherosclerotic lesions.The effect of oxidized phospholipids on phenotypic polarization and function of macrophages.Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective.KLF4-dependent phenotypic modulation of smooth muscle cells has a key role in atherosclerotic plaque pathogenesis.Epigenetic regulation of COL15A1 in smooth muscle cell replicative aging and atherosclerosis KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Smooth muscle cell phenotypic switching in atherosclerosis Cyclosporine up-regulates Krüppel-like factor-4 (KLF4) in vascular smooth muscle cells and drives phenotypic modulation in vivo.Complex regulation and function of the inflammatory smooth muscle cell phenotype in atherosclerosis Upregulation of distinct collagen transcripts in post-surgery scar tissue: a study of conjunctival fibrosis Krüppel-like factor 4 mediates lysophosphatidic acid-stimulated migration and proliferation of PC3M prostate cancer cells.Kruppel-like factor-4 transcriptionally regulates VE-cadherin expression and endothelial barrier function Pathological aspects of lipid peroxidation.Environment and vascular bed origin influence differences in endothelial transcriptional profiles of coronary and iliac arteries.Gene expression changes in the injured spinal cord following transplantation of mesenchymal stem cells or olfactory ensheathing cells.Autophagic regulation of smooth muscle cell biology The effect of PPE-induced emphysema and chronic LPS-induced pulmonary inflammation on atherosclerosis development in APOE*3-LEIDEN mice.In vivo noninvasive measurement of skin autofluorescence biomarkers relate to cardiovascular disease in mice.Inflammatory Ly-6C(hi) monocytes play an important role in the development of severe transplant arteriosclerosis in hyperlipidemic recipients Emerging applications for zebrafish as a model organism to study oxidative mechanisms and their roles in inflammation and vascular accumulation of oxidized lipids TIPE2 deficiency accelerates neointima formation by downregulating smooth muscle cell differentiation.Type VIII collagen mediates vessel wall remodeling after arterial injury and fibrous cap formation in atherosclerosis.Nkx2-5 Is Expressed in Atherosclerotic Plaques and Attenuates Development of Atherosclerosis in Apolipoprotein E-Deficient Mice.Zebrafish models of dyslipidemia: relevance to atherosclerosis and angiogenesis.Kruppel-like factors and vascular inflammation: implications for atherosclerosis.Role of phospholipid oxidation products in atherosclerosis.All-trans retinoic acid protects hepatocellular carcinoma cells against serum-starvation-induced cell death by upregulating collagen 8A2.Vascular smooth muscle cell in atherosclerosis.Lysophosphatidic acid-induced vascular neointimal formation in mouse carotid arteries is mediated by the matricellular protein CCN1/Cyr61.Collagen isoform shift during the early phase of reverse left ventricular remodelling after relief of pressure overload.Short-term stimulation of calcium-permeable transient receptor potential canonical 5-containing channels by oxidized phospholipids.Kruppel-like factor 4 contributes to high phosphate-induced phenotypic switching of vascular smooth muscle cells into osteogenic cells.Lack of collagen VIII reduces fibrosis and promotes early mortality and cardiac dilatation in pressure overload in mice.

P2860

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P2860

Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration.

http://www.wikidata.org/entity/Q30436774

description

2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009 թվականի հունվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Oxidized phospholipids induce ...... smooth muscle cell migration.

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Circulation Research

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Oxidized phospholipids induce ...... smooth muscle cell migration.

@en

P2093

Eser Adiguzel

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Gary K Owens

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Judith Berliner

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Michelle P Bendeck

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Nataliya A Pidkovka

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Olga A Cherepanova

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Olga F Sarmento

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Qiong Gan

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Tadashi Yoshida

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P2860

Type VIII collagen stimulates smooth muscle cell migration and matrix metalloproteinase synthesis after arterial injury

Vascular collagens: spotlight on the role of type VIII collagen in atherogenesis

Molecular regulation of vascular smooth muscle cell differentiation in development and disease

Positive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.

Kruppel-like factor 4 abrogates myocardin-induced activation of smooth muscle gene expression.

Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injury

Oxidized phospholipids induce phenotypic switching of vascular smooth muscle cells in vivo and in vitro.

Krüppel-like factor 4 is transactivated by butyrate in colon cancer cells.

Sp1 and krüppel-like factor family of transcription factors in cell growth regulation and cancer.

The zinc-finger transcription factor Klf4 is required for terminal differentiation of goblet cells in the colon.

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5

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104

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Norbert Leitinger

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2009-01-22T00:00:00Z

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19168440

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2758767

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