The subendothelial extracellular matrix modulates JNK activation by flow - Wikidata - wikimedia - TriplyDB

The subendothelial extracellular matrix modulates JNK activation by flow

The subendothelial extracellular matrix modulates JNK activation by flow

http://www.wikidata.org/entity/Q30438150

about

The atypical structure and function of newborn arterial endothelium is mediated by Rho/Rho kinase signaling Altered nitric oxide production mediates matrix-specific PAK2 and NF-κB activation by flow Agonist-directed desensitization of the β2-adrenergic receptor Flow-dependent cellular mechanotransduction in atherosclerosis.The role of p21-activated kinase in the initiation of atherosclerosis.Hemodynamic activation of beta-catenin and T-cell-specific transcription factor signaling in vascular endothelium regulates fibronectin expression.JNK2 promotes endothelial cell alignment under flow Atheroprone hemodynamics regulate fibronectin deposition to create positive feedback that sustains endothelial inflammation Matrix-specific protein kinase A signaling regulates p21-activated kinase activation by flow in endothelial cells Fibulin-2 and fibulin-5 cooperatively function to form the internal elastic lamina and protect from vascular injury.The arterial microenvironment: the where and why of atherosclerosis.ACE as a mechanosensor to shear stress influences the control of its own regulation via phosphorylation of cytoplasmic Ser(1270)The gene expression of human endothelial cells is modulated by subendothelial extracellular matrix proteins: short-term response to laminar shear stress.Effects of disturbed flow on vascular endothelium: pathophysiological basis and clinical perspectives Identification of small proline-rich repeat protein 3 as a novel atheroprotective factor that promotes adaptive Akt signaling in vascular smooth muscle cells.The role of blood flow in determining the sites of atherosclerotic plaques.Intramembrane binding of VE-cadherin to VEGFR2 and VEGFR3 assembles the endothelial mechanosensory complex.Shear stress inhibits homocysteine-induced stromal cell-derived factor-1 expression in endothelial cells.Interaction between integrin α5 and PDE4D regulates endothelial inflammatory signalling.The effects of acute and chronic exercise on the vasculature Linking cell structure to gene regulation: signaling events and expression controls on the model genes PAI-1 and CTGF.Extracellular matrix proteomics identifies molecular signature of symptomatic carotid plaques JNKs, insulin resistance and inflammation: A possible link between NAFLD and coronary artery disease.Inhibition of the mTOR pathway: a possible protective role in coronary artery disease.Molecular biology of atherosclerosis.Shear stress-initiated signaling and its regulation of endothelial function.Endothelial fluid shear stress sensing in vascular health and disease.Effects of shear stress on endothelial cells: go with the flow.Blood Brothers: Hemodynamics and Cell-Matrix Interactions in Endothelial Function.Integrin signaling in atherosclerosis.In Development-A New Paradigm for Understanding Vascular Disease.Label-free optical biosensor with microfluidics for sensing ligand-directed functional selectivity on trafficking of thrombin receptor.High Pulsatility Flow Induces Acute Endothelial Inflammation through Overpolarizing Cells to Activate NF-κB.Providing insights into atherosclerosis: A case of multivessel coronary artery disease with an anomalous straightforward vessel spared from atherosclerosis.Oscillatory flow accelerates autocrine signaling due to nonlinear effect of convection on receptor-related actions.Unveiling LOX-1 receptor interplay with nanotopography: mechanotransduction and atherosclerosis onset.Plasma fibronectin deficiency impedes atherosclerosis progression and fibrous cap formation Extracellular matrix differentiating good flow versus bad flow.Differential regulations of fibronectin and laminin in Smad2 activation in vascular endothelial cells in response to disturbed flow.Exercise training protects against atherosclerotic risk factors through vascular NADPH oxidase, extracellular signal-regulated kinase 1/2 and stress-activated protein kinase/c-Jun N-terminal kinase downregulation in obese rats.

P2860

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P2860

The subendothelial extracellular matrix modulates JNK activation by flow

http://www.wikidata.org/entity/Q30438150

description

2009 nî lūn-bûn

@nan

2009 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի մարտին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

The subendothelial extracellular matrix modulates JNK activation by flow

@ast

The subendothelial extracellular matrix modulates JNK activation by flow

@en

type

label

The subendothelial extracellular matrix modulates JNK activation by flow

@ast

The subendothelial extracellular matrix modulates JNK activation by flow

@en

prefLabel

The subendothelial extracellular matrix modulates JNK activation by flow

@ast

The subendothelial extracellular matrix modulates JNK activation by flow

@en

P1433

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P356

CIRCRESAHA.108.186486

P1433

Circulation Research

P1476

The subendothelial extracellular matrix modulates JNK activation by flow

@en

P2093

A Wayne Orr

http://www.w3.org/2001/XMLSchema#string

Cornelia Hahn

http://www.w3.org/2001/XMLSchema#string

John M Sanders

http://www.w3.org/2001/XMLSchema#string

Krishna A Jhaveri

http://www.w3.org/2001/XMLSchema#string

Martin Alexander Schwartz

http://www.w3.org/2001/XMLSchema#string

P2860

MEK kinases are regulated by EGF and selectively interact with Rac/Cdc42

Fluid shear stress inhibits vascular inflammation by decreasing thioredoxin-interacting protein in endothelial cells

Atherosclerosis

Integrins as receptors for laminins

Signal transduction by the JNK group of MAP kinases

Fluid shear stress activation of focal adhesion kinase. Linking to mitogen-activated protein kinases

Activation of integrins in endothelial cells by fluid shear stress mediates Rho-dependent cytoskeletal alignment

Biology of the p21-activated kinases

A dominant-negative p65 PAK peptide inhibits angiogenesis.

p21-activated kinase regulates endothelial permeability through modulation of contractility.

P304

995-1003

http://www.w3.org/2001/XMLSchema#string

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scholarly article

P356

10.1161/CIRCRESAHA.108.186486

http://www.w3.org/2001/XMLSchema#string

P433

8

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104

http://www.w3.org/2001/XMLSchema#string

P577

2009-03-12T00:00:00Z

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P5875

24201194

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P698

19286608

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P921

extracellular matrix

P932

2702158

http://www.w3.org/2001/XMLSchema#string