ICAM-1 and LFA-1 play critical roles in LPS-induced neutrophil recruitment into the alveolar space.
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Neutrophils in type 1 diabetesDistribution and expression of CD200 in the rat respiratory system under normal and endotoxin-induced pathological conditionsPreventive and therapeutic euphol treatment attenuates experimental colitis in miceEndothelial cell heterogeneityEliminating or blocking 12/15-lipoxygenase reduces neutrophil recruitment in mouse models of acute lung injuryPhosphoinositide 3-kinase gamma required for lipopolysaccharide-induced transepithelial neutrophil trafficking in the lung.The role of platelets in acute lung injury (ALI)Blocking p21-activated kinase reduces lipopolysaccharide-induced acute lung injury by preventing polymorphonuclear leukocyte infiltration.Anchoring fusion thrombomodulin to the endothelial lumen protects against injury-induced lung thrombosis and inflammation.Defective neutrophil recruitment in leukocyte adhesion deficiency type I disease causes local IL-17-driven inflammatory bone loss.Distamycin A inhibits HMGA1-binding to the P-selectin promoter and attenuates lung and liver inflammation during murine endotoxemia.Blockade of ICAM-1 improves the outcome of polymicrobial sepsis via modulating neutrophil migration and reversing immunosuppression.Leukocyte-endothelial interactions in inflammation.Expression, production, and renaturation of a functional single-chain variable antibody fragment (scFv) against human ICAM-1Acrolein inhalation suppresses lipopolysaccharide-induced inflammatory cytokine production but does not affect acute airways neutrophiliaCritical role of non-muscle myosin light chain kinase in thrombin-induced endothelial cell inflammation and lung PMN infiltrationInnate immune function of the adherens junction protein p120-catenin in endothelial response to endotoxinLeukocyte rolling and adhesion both contribute to regulation of microvascular permeability to albumin via ligation of ICAM-1.Dysregulation of neutrophil CXCR2 and pulmonary endothelial icam-1 promotes age-related pulmonary inflammation.Febrile-range hyperthermia modifies endothelial and neutrophilic functions to promote extravasation.Effects of JAM-A deficiency or blocking antibodies on neutrophil migration and lung injury in a murine model of ALIReduced neutrophil chemotaxis and infiltration contributes to delayed resolution of cutaneous wound infection with advanced age.Inflammatory response in microvascular endothelium in sepsis: role of oxidants.Siglec-E is a negative regulator of acute pulmonary neutrophil inflammation and suppresses CD11b β2-integrin-dependent signaling.Insult-dependent effect of bone marrow cell therapy on inflammatory response in a murine model of extrapulmonary acute respiratory distress syndromePotential utility of melatonin as an antioxidant during pregnancy and in the perinatal period.The mercurial nature of neutrophils: still an enigma in ARDS?Increased survival and reduced neutrophil infiltration of the liver in Rab27a- but not Munc13-4-deficient mice in lipopolysaccharide-induced systemic inflammation.Protein C concentrate controls leukocyte recruitment during inflammation and improves survival during endotoxemia after efficient in vivo activationCharacterizing cell subsets using marker enrichment modeling.Tumor-associated neutrophils (TAN) develop pro-tumorigenic properties during tumor progression.Anti-inflammatory functions of protein C require RAGE and ICAM-1 in a stimulus-dependent manner.Gram-Negative Pneumonia Alters Large-Vein Cell-Adhesion Molecule Profile and Potentiates Experimental Stasis Venous Thrombosis.Inducible RasGEF1B circular RNA is a positive regulator of ICAM-1 in the TLR4/LPS pathway.p53- and PAI-1-mediated induction of C-X-C chemokines and CXCR2: importance in pulmonary inflammation due to cigarette smoke exposureICAM-1-expressing neutrophils exhibit enhanced effector functions in murine models of endotoxemia.Platelet secretion of CXCL4 is Rac1-dependent and regulates neutrophil infiltration and tissue damage in septic lung damageDel-1, an endogenous leukocyte-endothelial adhesion inhibitor, limits inflammatory cell recruitment.Real-time imaging reveals endothelium-mediated leukocyte retention in LPS-treated lung microvessels.Overexpression of sICAM-1 in the alveolar epithelial space results in an exaggerated inflammatory response and early death in Gram negative pneumonia.
P2860
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P2860
ICAM-1 and LFA-1 play critical roles in LPS-induced neutrophil recruitment into the alveolar space.
description
2006 nî lūn-bûn
@nan
2006 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@ast
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@en
type
label
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@ast
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@en
prefLabel
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@ast
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@en
P2093
P2860
P1476
ICAM-1 and LFA-1 play critical ...... tment into the alveolar space.
@en
P2093
Abdul Basit
C Edward Rose
Joerg Reutershan
Margaret A Morris
P2860
P304
P356
10.1152/AJPLUNG.00346.2005
P577
2006-02-03T00:00:00Z