Physiologic regulation of a tetrodotoxin-sensitive sodium influx that mediates a slow afterdepolarization potential in gonadotropin-releasing hormone neurons: possible implications for the central regulation of fertility.
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Kisspeptin excites gonadotropin-releasing hormone neurons through a phospholipase C/calcium-dependent pathway regulating multiple ion channelsThe neurobiology of preovulatory and estradiol-induced gonadotropin-releasing hormone surges.Identified GnRH neuron electrophysiology: a decade of studyHyperpolarization-activated currents in gonadotropin-releasing hormone (GnRH) neurons contribute to intrinsic excitability and are regulated by gonadal steroid feedbackDiurnal in vivo and rapid in vitro effects of estradiol on voltage-gated calcium channels in gonadotropin-releasing hormone neurons.Estradiol suppresses glutamatergic transmission to gonadotropin-releasing hormone neurons in a model of negative feedback in miceNeurobiological mechanisms underlying oestradiol negative and positive feedback regulation of gonadotrophin-releasing hormone neuronesCritical roles for fast synaptic transmission in mediating estradiol negative and positive feedback in the neural control of ovulation.Differential regulation of gonadotropin-releasing hormone neuron activity and membrane properties by acutely applied estradiol: dependence on dose and estrogen receptor subtype.Age affects spontaneous activity and depolarizing afterpotentials in isolated gonadotropin-releasing hormone neurons.Vasoactive intestinal polypeptide can excite gonadotropin-releasing hormone neurons in a manner dependent on estradiol and gated by time of dayEstradiol attenuates multiple tetrodotoxin-sensitive sodium currents in isolated gonadotropin-releasing hormone neurons.Flufenamic acid modulates multiple currents in gonadotropin-releasing hormone neuronsRegulation of endogenous conductances in GnRH neurons by estrogensOptogenetic activation of GnRH neurons reveals minimal requirements for pulsatile luteinizing hormone secretion.Physiology of the gonadotrophin-releasing hormone (GnRH) neurone: studies from embryonic GnRH neuronesEstradiol directly attenuates sodium currents and depolarizing afterpotentials in isolated gonadotropin-releasing hormone neurons.Kisspeptin and Gonadotropin-Releasing Hormone Neuronal Excitability: Molecular Mechanisms Driven by 17β-Estradiol.Simulated GABA synaptic input and L-type calcium channels form functional microdomains in hypothalamic gonadotropin-releasing hormone neurons.Orexin a suppresses gonadotropin-releasing hormone (GnRH) neuron activity in the mouse17β-oestradiol regulation of gonadotrophin-releasing hormone neuronal excitability.Gonadotropin-releasing hormone-1 neuronal activity is independent of hyperpolarization-activated cyclic nucleotide-modulated channels but is sensitive to protein kinase a-dependent phosphorylationIon channels and information processing in GnRH neuron dendrites17Beta-estradiol regulation of T-type calcium channels in gonadotropin-releasing hormone neurons.Rapid nongenomic effects of oestradiol on gonadotrophin-releasing hormone neurones.A unified model for two modes of bursting in GnRH neurons.Activation of mGluR5 induces spike afterdepolarization and enhanced excitability in medium spiny neurons of the nucleus accumbens by modulating persistent Na+ currents.Small-conductance calcium-activated potassium channels control excitability and firing dynamics in gonadotropin-releasing hormone (GnRH) neurons.Gonadotropin-Releasing Hormone (GnRH) Neuron Excitability Is Regulated by Estradiol Feedback and Kisspeptin.Regulation of electrical bursting in a spatiotemporal model of a GnRH neuron.GnRH Neuron Activity and Pituitary Response in Estradiol-Induced vs Proestrous Luteinizing Hormone Surges in Female Mice.Burst firing in gonadotrophin-releasing hormone neurones does not require ionotrophic GABA or glutamate receptor activation.A simple integrative electrophysiological model of bursting GnRH neurons.
P2860
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P2860
Physiologic regulation of a tetrodotoxin-sensitive sodium influx that mediates a slow afterdepolarization potential in gonadotropin-releasing hormone neurons: possible implications for the central regulation of fertility.
description
2006 nî lūn-bûn
@nan
2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Physiologic regulation of a te ...... ntral regulation of fertility.
@ast
Physiologic regulation of a te ...... ntral regulation of fertility.
@en
type
label
Physiologic regulation of a te ...... ntral regulation of fertility.
@ast
Physiologic regulation of a te ...... ntral regulation of fertility.
@en
prefLabel
Physiologic regulation of a te ...... ntral regulation of fertility.
@ast
Physiologic regulation of a te ...... ntral regulation of fertility.
@en
P1476
Physiologic regulation of a te ...... entral regulation of fertility
@en
P2093
Suzanne M Moenter
Zhiguo Chu
P304
11961-11973
P356
10.1523/JNEUROSCI.3171-06.2006
P407
P577
2006-11-01T00:00:00Z