Developmental changes in cell-extracellular matrix interactions limit proliferation in the mammalian inner ear.
about
A historical to present-day account of efforts to answer the question: "what puts the brakes on mammalian hair cell regeneration?"Responses to cell loss become restricted as the supporting cells in mammalian vestibular organs grow thick junctional actin bands that develop high stabilityOver half the hair cells in the mouse utricle first appear after birth, with significant numbers originating from early postnatal mitotic production in peripheral and striolar growth zonesIn vivo proliferative regeneration of balance hair cells in newborn mice.The postnatal accumulation of junctional E-cadherin is inversely correlated with the capacity for supporting cells to convert directly into sensory hair cells in mammalian balance organsVariations in shape-sensitive restriction points mirror differences in the regeneration capacities of avian and mammalian ears.Reinforcement of cell junctions correlates with the absence of hair cell regeneration in mammals and its occurrence in birds.Recent advances in hair cell regeneration research.Postnatal development, maturation and aging in the mouse cochlea and their effects on hair cell regeneration.Inner ear hair cells produced in vitro by a mesenchymal-to-epithelial transitionThe challenge of hair cell regeneration.Metalloproteinases and their associated genes contribute to the functional integrity and noise-induced damage in the cochlear sensory epithelium.MYC gene delivery to adult mouse utricles stimulates proliferation of postmitotic supporting cells in vitro.TAK1 expression in the cochlea: a specific marker for adult supporting cells.Activation of canonical Wnt/β-catenin signaling stimulates proliferation in neuromasts in the zebrafish posterior lateral line.Elastic force restricts growth of the murine utricle.Regeneration of hair cells in the mammalian vestibular system.Development and regeneration of vestibular hair cells in mammals.Specializations of intercellular junctions are associated with the presence and absence of hair cell regeneration in ears from six vertebrate classes.Cell-extracellular matrix versus cell-cell interactions during the development of the cochlear-vestibular ganglion.The miR-183/ItgA3 axis is a key regulator of prosensory area during early inner ear development.Junctional E-cadherin/p120-catenin Is Correlated with the Absence of Supporting Cells to Hair Cells Conversion in Postnatal Mice Cochleae.
P2860
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P2860
Developmental changes in cell-extracellular matrix interactions limit proliferation in the mammalian inner ear.
description
2007 nî lūn-bûn
@nan
2007 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Developmental changes in cell- ...... on in the mammalian inner ear.
@ast
Developmental changes in cell- ...... on in the mammalian inner ear.
@en
type
label
Developmental changes in cell- ...... on in the mammalian inner ear.
@ast
Developmental changes in cell- ...... on in the mammalian inner ear.
@en
prefLabel
Developmental changes in cell- ...... on in the mammalian inner ear.
@ast
Developmental changes in cell- ...... on in the mammalian inner ear.
@en
P2093
P1476
Developmental changes in cell- ...... on in the mammalian inner ear.
@en
P2093
Christopher Magnus
Dawn Davies
Jeffrey T Corwin
P304
P356
10.1111/J.1460-9568.2007.05355.X
P407
P577
2007-02-01T00:00:00Z