Smooth muscle cells and myofibroblasts use distinct transcriptional mechanisms for smooth muscle alpha-actin expression.
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Recent developments in myofibroblast biology: paradigms for connective tissue remodelingFibroblasts in fibrosis: novel roles and mediatorsFunctional vascular smooth muscle-like cells derived from adult mouse uterine mesothelial cellsPitx2 is functionally important in the early stages of vascular smooth muscle cell differentiationOverexpression of TEAD-1 in transgenic mouse striated muscles produces a slower skeletal muscle contractile phenotypeRegulation of myofibroblast differentiation by poly(ADP-ribose) polymerase 1Plakophilin-2 and the migration, differentiation and transformation of cells derived from the epicardium of neonatal rat heartsContext-dependent switch in chemo/mechanotransduction via multilevel crosstalk among cytoskeleton-regulated MRTF and TAZ and TGFβ-regulated Smad3Yap1 protein regulates vascular smooth muscle cell phenotypic switch by interaction with myocardin.WD repeat-containing protein 5, a ubiquitously expressed histone methyltransferase adaptor protein, regulates smooth muscle cell-selective gene activation through interaction with pituitary homeobox 2.Sp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.Ca(v)1.2 splice variant with exon 9* is critical for regulation of cerebral artery diameter.PDGF-Ralpha gene expression predicts proliferation, but PDGF-A suppresses transdifferentiation of neonatal mouse lung myofibroblasts.Genome-wide mapping of Sox6 binding sites in skeletal muscle reveals both direct and indirect regulation of muscle terminal differentiation by Sox6.Molecular regulation of contractile smooth muscle cell phenotype: implications for vascular tissue engineering.The endothelium-dependent effect of RTEF-1 in pressure overload cardiac hypertrophy: role of VEGF-BRTEF-1, an upstream gene of hypoxia-inducible factor-1α, accelerates recovery from ischemia.Endothelial differentiation gene-1, a new downstream gene is involved in RTEF-1 induced angiogenesis in endothelial cells.Vascular smooth muscle progenitor cells: building and repairing blood vessels.Mechanical stretch up-regulates the B-type natriuretic peptide system in human cardiac fibroblasts: a possible defense against transforming growth factor-β mediated fibrosis.TEAD transcription factors are required for normal primary myoblast differentiation in vitro and muscle regeneration in vivoThe induction of yes-associated protein expression after arterial injury is crucial for smooth muscle phenotypic modulation and neointima formation.Tissue Engineering of Blood Vessels: Functional Requirements, Progress, and Future Challenges.Transcriptional control of cardiac fibroblast plasticity.Transcriptional regulation of SM22alpha by Wnt3a: convergence with TGFbeta(1)/Smad signaling at a novel regulatory element.The effects of heparin releasing hydrogels on vascular smooth muscle cell phenotype.The transcription factor TEAD1 represses smooth muscle-specific gene expression by abolishing myocardin function.Sox10+ adult stem cells contribute to biomaterial encapsulation and microvascularization.Role of myofibroblasts in vascular remodelling: focus on restenosis and aneurysm.The role of transcription enhancer factors in cardiovascular biology.Targeting cardiac fibroblasts to treat fibrosis of the heart: focus on HDACs.The functional diversity of essential genes required for mammalian cardiac development.Bioresorbable Scaffolds for Atheroregression: Understanding of Transient ScaffoldingPossible Muscle Repair in the Human Cardiovascular System.Small-diameter vascular graft engineered using human embryonic stem cell-derived mesenchymal cells.Structural basis of multisite single-stranded DNA recognition and ACTA2 repression by purine-rich element binding protein B (Purβ).Related transcriptional enhancer factor 1 increases endothelial-dependent microvascular relaxation and proliferation.Hypoxia induced changes of SePP1 expression in rat preadipocytes and its impact on vascular fibroblastsDifferential topical susceptibility to TGFβ in intact and injured regions of the epithelium: key role in myofibroblast transitionLack of α8 integrin leads to morphological changes in renal mesangial cells, but not in vascular smooth muscle cells.
P2860
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P2860
Smooth muscle cells and myofibroblasts use distinct transcriptional mechanisms for smooth muscle alpha-actin expression.
description
2007 nî lūn-bûn
@nan
2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@ast
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@en
type
label
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@ast
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@en
prefLabel
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@ast
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@en
P2093
P1433
P1476
Smooth muscle cells and myofib ...... muscle alpha-actin expression.
@en
P2093
Gary K Owens
Tadashi Yoshida
P304
P356
10.1161/CIRCRESAHA.107.154831
P577
2007-09-06T00:00:00Z