Anaerobic killing of mucoid Pseudomonas aeruginosa by acidified nitrite derivatives under cystic fibrosis airway conditions.
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Involvement of nitric oxide in biofilm dispersal of Pseudomonas aeruginosaDisruption and eradication of P. aeruginosa biofilms using nitric oxide-releasing chitosan oligosaccharides.Prediction and analysis of the protein interactome in Pseudomonas aeruginosa to enable network-based drug target selectionNitrate respiration protects hypoxic Mycobacterium tuberculosis against acid- and reactive nitrogen species stressesBdlA, DipA and induced dispersion contribute to acute virulence and chronic persistence of Pseudomonas aeruginosaProteomic, microarray, and signature-tagged mutagenesis analyses of anaerobic Pseudomonas aeruginosa at pH 6.5, likely representing chronic, late-stage cystic fibrosis airway conditionsCatalase (KatA) plays a role in protection against anaerobic nitric oxide in Pseudomonas aeruginosaA Putative ABC Transporter Permease Is Necessary for Resistance to Acidified Nitrite and EDTA in Pseudomonas aeruginosa under Aerobic and Anaerobic Planktonic and Biofilm Conditions.Determinants of exhaled breath condensate pH in a large population with asthma.Two-pronged survival strategy for the major cystic fibrosis pathogen, Pseudomonas aeruginosa, lacking the capacity to degrade nitric oxide during anaerobic respiration.T2R38 taste receptor polymorphisms underlie susceptibility to upper respiratory infectionSimple sequence repeats and mucoid conversion: biased mucA mutagenesis in mismatch repair-deficient Pseudomonas aeruginosa.Evidence for sigma factor competition in the regulation of alginate production by Pseudomonas aeruginosa.Inhibition of staphylococcal biofilm formation by nitrite.Pseudomonas aeruginosa biofilm infections in cystic fibrosis: insights into pathogenic processes and treatment strategies.Nutrient availability as a mechanism for selection of antibiotic tolerant Pseudomonas aeruginosa within the CF airway.A biophysical model for integration of electrical, osmotic, and pH regulation in the human bronchial epithelium.Contribution of cell elongation to the biofilm formation of Pseudomonas aeruginosa during anaerobic respirationEpistatic roles for Pseudomonas aeruginosa MutS and DinB (DNA Pol IV) in coping with reactive oxygen species-induced DNA damage.Pseudomonas aeruginosa in vitro phenotypes distinguish cystic fibrosis infection stages and outcomesAcidosis potentiates the host proinflammatory interleukin-1β response to Pseudomonas aeruginosa infectionOral streptococci and nitrite-mediated interference of Pseudomonas aeruginosa.Nitric oxide protects bacteria from aminoglycosides by blocking the energy-dependent phases of drug uptake.Simple sequence repeats together with mismatch repair deficiency can bias mutagenic pathways in Pseudomonas aeruginosa during chronic lung infectionMyeloperoxidase-dependent oxidative metabolism of nitric oxide in the cystic fibrosis airway.Cytotoxicity of Pseudomonas secreted exotoxins requires OxyR expressionAn official ATS clinical practice guideline: interpretation of exhaled nitric oxide levels (FENO) for clinical applications.Inorganic nitrite therapy: historical perspective and future directions.Chronic rhinosinusitis pathogenesis.Vitamin B12-mediated restoration of defective anaerobic growth leads to reduced biofilm formation in Pseudomonas aeruginosa.A novel siderophore system is essential for the growth of Pseudomonas aeruginosa in airway mucusIdentification of essential genes of Pseudomonas aeruginosa for its growth in airway mucus.A Genetic Screen Reveals Novel Targets to Render Pseudomonas aeruginosa Sensitive to Lysozyme and Cell Wall-Targeting AntibioticsMicroenvironmental characteristics and physiology of biofilms in chronic infections of CF patients are strongly affected by the host immune response.Activation of cholera toxin production by anaerobic respiration of trimethylamine N-oxide in Vibrio choleraeUpdate in cystic fibrosis 2006Network-assisted investigation of virulence and antibiotic-resistance systems in Pseudomonas aeruginosaClearance of initial mucoid Pseudomonas aeruginosa in patients with cystic fibrosis.Dual promoters of the major catalase (KatA) govern distinct survival strategies of Pseudomonas aeruginosaFosfomycin and tobramycin in combination downregulate nitrate reductase genes narG and narH, resulting in increased activity against Pseudomonas aeruginosa under anaerobic conditions.
P2860
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P2860
Anaerobic killing of mucoid Pseudomonas aeruginosa by acidified nitrite derivatives under cystic fibrosis airway conditions.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@ast
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@en
type
label
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@ast
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@en
prefLabel
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@ast
Anaerobic killing of mucoid Ps ...... ic fibrosis airway conditions.
@en
P2093
P2860
P356
P1476
Anaerobic killing of mucoid Ps ...... tic fibrosis airway conditions
@en
P2093
Ahmet C Karabulut
Benjamin Gaston
Daniel J Hassett
David Speert
Jane L Burns
Joel E Mortensen
Michael J Schurr
Ray Coakley
Richard C Boucher
P2860
P304
P356
10.1172/JCI24684
P407
P577
2006-01-26T00:00:00Z