Mechanisms of tight junction dysregulation in the SAMP1/YitFc model of Crohn's disease-like ileitis.
about
Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human geneticsClaudins in intestines: Distribution and functional significance in health and diseasesIncrease in the tight junction protein claudin-1 in intestinal inflammationSAMP1/YitFc mouse strain: a spontaneous model of Crohn's disease-like ileitis.Probiotic bacteria regulate intestinal epithelial permeability in experimental ileitis by a TNF-dependent mechanismTargeting the A2B adenosine receptor during gastrointestinal ischemia and inflammationWhole-genome microarray analysis and functional characterization reveal distinct gene expression profiles and patterns in two mouse models of ileal inflammation.Immunopathogenesis of inflammatory bowel diseaseUnderstanding host-adherent-invasive Escherichia coli interaction in Crohn's disease: opening up new therapeutic strategies.Host-microbial interactions and regulation of intestinal epithelial barrier function: From physiology to pathology.A role for syndecan-1 and claudin-2 in microbial translocation during HIV-1 infection.Actin reorganization as the molecular basis for the regulation of apoptosis in gastrointestinal epithelial cells.Fermented Pueraria Lobata extract ameliorates dextran sulfate sodium-induced colitis by reducing pro-inflammatory cytokines and recovering intestinal barrier function.Increased Enterococcus faecalis infection is associated with clinically active Crohn disease.The digestive neuronal-glial-epithelial unit: a new actor in gut health and disease.CCR9 antagonism: potential in the treatment of Inflammatory Bowel Disease.Vasoactive intestinal peptide prevents PKCε-induced intestinal epithelial barrier disruption during EPEC infection.Inflammatory bowel disease therapies and gut function in a colitis mouse model.Wasabia japonica is a potential functional food to prevent colitis via inhibiting the NF-κB signaling pathway.Epithelial-specific Toll-like Receptor (TLR)5 Activation Mediates Barrier Dysfunction in Experimental Ileitis.Adherent-invasive Escherichia coli induce claudin-2 expression and barrier defect in CEABAC10 mice and Crohn's disease patients.
P2860
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P2860
Mechanisms of tight junction dysregulation in the SAMP1/YitFc model of Crohn's disease-like ileitis.
description
2009 nî lūn-bûn
@nan
2009 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
name
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@ast
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@en
type
label
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@ast
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@en
prefLabel
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@ast
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@en
P1476
Mechanisms of tight junction d ...... Crohn's disease-like ileitis.
@en
P2093
Brian K Reuter
Theresa T Pizarro
P2860
P304
P356
10.1111/J.1749-6632.2009.04035.X
P407
P577
2009-05-01T00:00:00Z