Two distant upstream regions containing cis-acting signals regulating splicing facilitate 3'-end processing of avian sarcoma virus RNA.
about
Gene trap mutagenesis of hnRNP A2/B1: a cryptic 3' splice site in the neomycin resistance gene allows continued expression of the disrupted cellular geneFrameshift mutations in the v-src gene of avian sarcoma virus act in cis to specifically reduce v-src mRNA levels.Inhibition of RNA splicing at the Rous sarcoma virus src 3' splice site is mediated by an interaction between a negative cis element and a chicken embryo fibroblast nuclear factor.Efficient polyadenylation of Rous sarcoma virus RNA requires the negative regulator of splicing elementSelective inhibition of splicing at the avian sarcoma virus src 3' splice site by direct-repeat posttranscriptional cis elementsRNA 3' readthrough of oncoretrovirus and lentivirus: implications for vector safety and efficacy.Alternative poly(A) site selection in complex transcription units: means to an end?The retrovirus RNA trafficking granule: from birth to maturity.The negative regulator of splicing element of Rous sarcoma virus promotes polyadenylationJuxtaposition of two distant, serine-arginine-rich protein-binding elements is required for optimal polyadenylation in Rous sarcoma virus.Silent point mutation in an avian retrovirus RNA processing element promotes c-myb-associated short-latency lymphomasSerine/arginine-rich proteins contribute to negative regulator of splicing element-stimulated polyadenylation in rous sarcoma virus.The human immunodeficiency virus type 1 Rev protein and the Rev-responsive element counteract the effect of an inhibitory 5' splice site in a 3' untranslated regionLack of an effect of the efficiency of RNA 3'-end formation on the efficiency of removal of either the final or the penultimate intron in intact cellsSequences homologous to 5' splice sites are required for the inhibitory activity of papillomavirus late 3' untranslated regions.Characterization of Rous sarcoma virus polyadenylation site use in vitroRNA processing control in avian retrovirusesPosttranscriptional regulation of retroviral gene expression: primary RNA transcripts play three roles as pre-mRNA, mRNA, and genomic RNA.Evidence that a threshold of serine/arginine-rich (SR) proteins recruits CFIm to promote rous sarcoma virus mRNA 3' end formationR region sequences in the long terminal repeat of a murine retrovirus specifically increase expression of unspliced RNAs.Genetic determinant of rapid-onset B-cell lymphoma by avian leukosis virus.Rous sarcoma virus direct repeat cis elements exert effects at several points in the virus life cycle.
P2860
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P2860
Two distant upstream regions containing cis-acting signals regulating splicing facilitate 3'-end processing of avian sarcoma virus RNA.
description
1992 nî lūn-bûn
@nan
1992 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@ast
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@en
type
label
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@ast
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@en
prefLabel
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@ast
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@en
P2860
P1433
P1476
Two distant upstream regions c ...... ng of avian sarcoma virus RNA.
@en
P2093
C M Stoltzfus
J T Miller
P2860
P304
P577
1992-07-01T00:00:00Z