The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene.
about
Mitochondrial Dysfunction and Autophagy in Hepatic Ischemia/Reperfusion InjuryDiscovery of a new function of cyclooxygenase (COX)-2: COX-2 is a cardioprotective protein that alleviates ischemia/reperfusion injury and mediates the late phase of preconditioningRole of nitric oxide synthases in the infarct size-reducing effect conferred by heat stress in isolated rat heartsAtorvastatin at reperfusion reduces myocardial infarct size in mice by activating eNOS in bone marrow-derived cells.Protein S-nitrosylation and cardioprotectionc-kit+ Cardiac stem cells alleviate post-myocardial infarction left ventricular dysfunction despite poor engraftment and negligible retention in the recipient heart.Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.Preconditioning of isoflurane on spinal cord ischemia can increase the number of inducible nitric oxide synthase-expressing motor neurons in ratAn essential role of the JAK-STAT pathway in ischemic preconditioning.Opposing cardioprotective actions and parallel hypertrophic effects of delta PKC and epsilon PKC.Dynamic regulation of metabolism and respiration by endogenously produced nitric oxide protects against oxidative stress.Dietary nitrate supplementation protects against Doxorubicin-induced cardiomyopathy by improving mitochondrial function.Surviving ischemia: adaptive responses mediated by hypoxia-inducible factor 1Protective responses in the ischemic myocardium.Effect of disruption of Akt-1 of lin(-)c-kit(+) stem cells on myocardial performance in infarcted heartPostischemic deactivation of cardiac aldose reductase: role of glutathione S-transferase P and glutaredoxin in regeneration of reduced thiols from sulfenic acidsNitric oxide donors.Coronary endothelial dysfunction after ischemia and reperfusion: a new therapeutic target?Protection of Tong-Sai-Mai Decoction against Apoptosis Induced by H2O2 in PC12 Cells: Mechanisms via Bcl-2-Mitochondria-ROS-INOS Pathway.Role of microRNAs in cardiac preconditioningToll-like receptor 4 signaling confers cardiac protection against ischemic injury via inducible nitric oxide synthase- and soluble guanylate cyclase-dependent mechanisms.Organ preconditioning: the past, current status, and related lung studiesRedox signaling in cardiac myocytes.The late phase of preconditioning and its natural clinical application--gene therapyRole of mitogen- and stress-activated kinases in ischemic injury.Preemptive conditioning of the swine heart by H11 kinase/Hsp22 provides cardiac protection through inducible nitric oxide synthase.Formation of protein kinase C(epsilon)-Lck signaling modules confers cardioprotection.Role of Endothelial Cells in Myocardial Ischemia-Reperfusion Injury.Modulation of cardiac contraction, relaxation and rate by the endothelial nitric oxide synthase (eNOS): lessons from genetically modified mice.Nitric oxide, superoxide, and peroxynitrite in myocardial ischaemia-reperfusion injury and preconditioning.The NHLBI-sponsored Consortium for preclinicAl assESsment of cARdioprotective therapies (CAESAR): a new paradigm for rigorous, accurate, and reproducible evaluation of putative infarct-sparing interventions in mice, rabbits, and pigsObligatory role of heat shock protein 90 in iNOS induction.Endoplasmic reticulum stress-dependent activation of ATF3 mediates the late phase of ischemic preconditioningAdministration of a CO-releasing molecule induces late preconditioning against myocardial infarction.Late preconditioning induced by NO donors, adenosine A1 receptor agonists, and delta1-opioid receptor agonists is mediated by iNOSMechanism of cyclooxygenase-2 upregulation in late preconditioningPreconditioning: a paradigm shift in the biology of myocardial ischemiaDelayed adaptation of the heart to stress: late preconditioning.Endothelial nitric oxide synthase plays an obligatory role in the late phase of ischemic preconditioning by activating the protein kinase C epsilon p44/42 mitogen-activated protein kinase pSer-signal transducers and activators of transcription1/3 paTadalafil, a Phosphodiesterase Inhibitor Protects Stem Cells over Longer Period Against Hypoxia/Reoxygenation Injury Through STAT3/PKG-I Signaling.
P2860
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P2860
The late phase of ischemic preconditioning is abrogated by targeted disruption of the inducible NO synthase gene.
description
1999 nî lūn-bûn
@nan
1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The late phase of ischemic pre ...... he inducible NO synthase gene.
@ast
The late phase of ischemic pre ...... he inducible NO synthase gene.
@en
type
label
The late phase of ischemic pre ...... he inducible NO synthase gene.
@ast
The late phase of ischemic pre ...... he inducible NO synthase gene.
@en
prefLabel
The late phase of ischemic pre ...... he inducible NO synthase gene.
@ast
The late phase of ischemic pre ...... he inducible NO synthase gene.
@en
P2093
P2860
P356
P1476
The late phase of ischemic pre ...... he inducible NO synthase gene.
@en
P2093
V E Laubach
P2860
P304
11507-11512
P356
10.1073/PNAS.96.20.11507
P407
P50
P577
1999-09-01T00:00:00Z