Ageing Fxr deficient mice develop increased energy expenditure, improved glucose control and liver damage resembling NASH - Wikidata - wikimedia - TriplyDB

Ageing Fxr deficient mice develop increased energy expenditure, improved glucose control and liver damage resembling NASH

Ageing Fxr deficient mice develop increased energy expenditure, improved glucose control and liver damage resembling NASH

http://www.wikidata.org/entity/Q30454451

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Farnesoid X receptor inhibits glucagon-like peptide-1 production by enteroendocrine L cells Farnesoid X Receptor an Emerging Target to Combat Obesity.Deletion of mouse FXR gene disturbs multiple neurotransmitter systems and alters neurobehavior Recent insights into farnesoid X receptor in non-alcoholic fatty liver disease.Metabolic effects of cholecystectomy: gallbladder ablation increases basal metabolic rate through G-protein coupled bile acid receptor Gpbar1-dependent mechanisms in mice.Gut microbiota-derived lipopolysaccharide uptake and trafficking to adipose tissue: implications for inflammation and obesity.Effects of chronic sleep deprivation on glucose homeostasis in rats FXR agonists as therapeutic agents for non-alcoholic fatty liver disease.The pharmacological management of NAFLD in children and adolescents.Western Diet-Induced Dysbiosis in Farnesoid X Receptor Knockout Mice Causes Persistent Hepatic Inflammation after Antibiotic Treatment.The Role of Nuclear Receptors in the Pathophysiology, Natural Course, and Drug Treatment of NAFLD in Humans.SAR studies on FXR modulators led to the discovery of the first combined FXR antagonistic/TGR5 agonistic compound.Intestinal bile acid receptors are key regulators of glucose homeostasis.The role of bile acids in nonalcoholic fatty liver disease and nonalcoholic steatohepatitis.The intrahepatic expression levels of bile acid transporters are inversely correlated with the histological progression of nonalcoholic fatty liver disease.On the Role of Illness Duration and Nutrient Restriction in Cholestatic Alterations that Occur During Critical Illness.Role of FXR in Liver Inflammation during Nonalcoholic Steatohepatitis.Differential modulation of FXR activity by chlorophacinone and ivermectin analogs.Obeticholic acid protects against hepatocyte death and liver fibrosis in a murine model of nonalcoholic steatohepatitis.Farnesoid X receptor agonist INT-767 attenuates liver steatosis and inflammation in rat model of nonalcoholic steatohepatitis Nuclear receptors: how do they position in non-alcoholic fatty liver disease treatment?

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Ageing Fxr deficient mice develop increased energy expenditure, improved glucose control and liver damage resembling NASH

http://www.wikidata.org/entity/Q30454451

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2013 nî lūn-bûn

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2013 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2013 թվականի մայիսին հրատարակված գիտական հոդված

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2013年の論文

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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JOURNAL.PONE.0064721

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Ageing Fxr deficient mice deve ...... d liver damage resembling NASH

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Daniel Lindén

http://www.w3.org/2001/XMLSchema#string

Gerhard Böttcher

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Jan Oscarsson

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Krister Bamberg

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Majlis Hermansson

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Marianne Wedin

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Melker Göransson

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Mikael Bjursell

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Mohammad Bohlooly-Y

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Therése Admyre

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P2860

A gene encoding a liver-specific ABC transporter is mutated in progressive familial intrahepatic cholestasis

Bile acids: natural ligands for an orphan nuclear receptor

Bile acids activate fibroblast growth factor 19 signaling in human hepatocytes to inhibit cholesterol 7alpha-hydroxylase gene expression

Generalized lacZ expression with the ROSA26 Cre reporter strain

Differential regulation of bile acid homeostasis by the farnesoid X receptor in liver and intestine

Progressive familial intrahepatic cholestasis, type 1, is associated with decreased farnesoid X receptor activity

Bile acids induce energy expenditure by promoting intracellular thyroid hormone activation

Identification of a nuclear receptor that is activated by farnesol metabolites

FXR regulates liver repair after CCl4-induced toxic injury

Regulation of antibacterial defense in the small intestine by the nuclear bile acid receptor

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e64721

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scholarly article

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10.1371/JOURNAL.PONE.0064721

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2013-05-20T00:00:00Z

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236934293

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23700488

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3659114

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