Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
about
Synaptic plasticity, neural circuits, and the emerging role of altered short-term information processing in schizophrenia.PKBγ/AKT3 loss-of-function causes learning and memory deficits and deregulation of AKT/mTORC2 signaling: Relevance for schizophrenia.Molecular substrates of schizophrenia: homeostatic signaling to connectivityPhosphorylation of AKT and abdominal aortic aneurysm formation.Pregnenolone sulfate normalizes schizophrenia-like behaviors in dopamine transporter knockout mice through the AKT/GSK3β pathway.Electrophysiological and behavioral responses to ketamine in mice with reduced Akt1 expression.Neurodevelopment in schizophrenia: the role of the wnt pathways.Cross-disorder comparative analysis of comorbid conditions reveals novel autism candidate genesThe NMDA Receptor and Schizophrenia: From Pathophysiology to Treatment.Modulation ofTcf7l2 expression alters behavior in mice.Multiple risk pathways for schizophrenia converge in serine racemase knockout mice, a mouse model of NMDA receptor hypofunctionFrontier of epilepsy research - mTOR signaling pathway.Perceptual training strongly improves visual motion perception in schizophreniaMicroRNA-derived network analysis of differentially methylated genes in schizophrenia, implicating GABA receptor B1 [GABBR1] and protein kinase B [AKT1]NMDA hypofunction as a convergence point for progression and symptoms of schizophreniaAkting up in the GABA hypothesis of schizophrenia: Akt1 deficiency modulates GABAergic functions and hippocampus-dependent functions.Molecular evidence for decreased synaptic efficacy in the postmortem olfactory bulb of individuals with schizophreniaMethylomic changes during conversion to psychosis.Neuroglialpharmacology: myelination as a shared mechanism of action of psychotropic treatments.Genetic Deletion of Akt3 Induces an Endophenotype Reminiscent of Psychiatric Manifestations in Mice.Altered CREB Binding to Activity-Dependent Genes in Serine Racemase Deficient Mice, a Mouse Model of Schizophrenia.Cognitive Deficits in Schizophrenia: Understanding the Biological Correlates and Remediation Strategies.Toward the Language OscillogenomeTafa-2 plays an essential role in neuronal survival and neurobiological function in mice
P2860
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P2860
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@ast
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@en
type
label
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@ast
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@en
prefLabel
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@ast
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@en
P2093
P2860
P356
P1433
P1476
Akt1 deficiency in schizophrenia and impairment of hippocampal plasticity and function.
@en
P2093
Darrick T Balu
Gregory C Carlson
Irwin Lucki
Konrad Talbot
Rachel M Easton
Tiffany E Hill-Smith
P2860
P304
P356
10.1002/HIPO.20887
P577
2010-11-03T00:00:00Z