Neurological suppression of diaphragm electromyographs in hamsters infected with West Nile virus. - Wikidata - wikimedia - TriplyDB

Neurological suppression of diaphragm electromyographs in hamsters infected with West Nile virus.

Neurological suppression of diaphragm electromyographs in hamsters infected with West Nile virus.

http://www.wikidata.org/entity/Q30474961

about

Neurological approaches for investigating West Nile virus disease and its treatment in rodents Efficacy of the broad-spectrum antiviral compound BCX4430 against Zika virus in cell culture and in a mouse model.Fatal neurological respiratory insufficiency is common among viral encephalitides Autonomic nervous dysfunction in hamsters infected with West Nile virus.Inhibition of West Nile virus by calbindin-D28k.Respiratory insufficiency correlated strongly with mortality of rodents infected with West Nile virus.The contribution of rodent models to the pathological assessment of flaviviral infections of the central nervous system Autonomic deficit not the cause of death in West Nile virus neurological disease Phrenic nerve deficits and neurological immunopathology associated with acute West Nile virus infection in mice and hamsters.Comparative pathology of neurovirulent lineage 1 (NY99/385) and lineage 2 (SPU93/01) West Nile virus infections in BALBc mice.Treatment of spatial memory impairment in hamsters infected with West Nile virus using a humanized monoclonal antibody MGAWN1.

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Neurological suppression of diaphragm electromyographs in hamsters infected with West Nile virus.

http://www.wikidata.org/entity/Q30474961

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Neurological suppression of di ...... infected with West Nile virus.

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Neurological suppression of di ...... infected with West Nile virus.

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13550284.2010.501847

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Journal of NeuroVirology

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Neurological suppression of di ...... infected with West Nile virus.

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Hong Wang

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Jeffery O Hall

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Neil E Motter

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Ramona T Skirpstunas

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P2860

Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis

Defining Limits of Treatment with Humanized Neutralizing Monoclonal Antibody for West Nile Virus Neurological Infection in a Hamster Model

West Nile Virus-Induced Neuroinflammation: Glial Infection and Capsid Protein-Mediated Neurovirulence

West Nile Virus Infection Activates the Unfolded Protein Response, Leading to CHOP Induction and Apoptosis

West Nile virus–induced acute flaccid paralysis is prevented by monoclonal antibody treatment when administered after infection of spinal cord neurons

Persistent West Nile Virus Associated with a Neurological Sequela in Hamsters Identified by Motor Unit Number Estimation

Induction of inflammation by West Nile virus capsid through the caspase-9 apoptotic pathway

PHOX2B immunolocalization of the candidate human retrotrapezoid nucleus

Defective respiratory rhythmogenesis and loss of central chemosensitivity in Phox2b mutants targeting retrotrapezoid nucleus neurons

Treatment of late stage disease in a model of arenaviral hemorrhagic fever: T-705 efficacy and reduced toxicity suggests an alternative to ribavirin

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13550284.2010.501847

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318-329

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West Nile virus

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