Replication of Cryptococcus neoformans in macrophages is accompanied by phagosomal permeabilization and accumulation of vesicles containing polysaccharide in the cytoplasm
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The outcome of Cryptococcus neoformans intracellular pathogenesis in human monocytesCryptococcus neoformans gene expression during murine macrophage infectionThe Cryptococcus neoformans capsule: a sword and a shield.Cryptococcus neoformans: Tripping on Acid in the PhagolysosomeCryptococcus and Phagocytes: Complex Interactions that Influence Disease OutcomeDancing cheek to cheek: Cryptococcus neoformans and phagocytesFungal colonization of the brain: anatomopathological aspects of neurological cryptococcosisHost immunity to Cryptococcus neoformansThe outcome of phagocytic cell division with infectious cargo depends on single phagosome formationThe human fungal pathogen Cryptococcus neoformans escapes macrophages by a phagosome emptying mechanism that is inhibited by Arp2/3 complex-mediated actin polymerisationIdentification of Candida glabrata genes involved in pH modulation and modification of the phagosomal environment in macrophagesCryptococcus neoformans Intracellular Proliferation and Capsule Size Determines Early Macrophage Control of Infection.Functional analysis of host factors that mediate the intracellular lifestyle of Cryptococcus neoformansThe fatal fungal outbreak on Vancouver Island is characterized by enhanced intracellular parasitism driven by mitochondrial regulationGlobal transcriptome profile of Cryptococcus neoformans during exposure to hydrogen peroxide induced oxidative stressDifferential activation of peritoneal cells by subcutaneous treatment of rats with cryptococcal antigensThe spectrum of fungi that infects humansNovel cell-based in vitro screen to identify small-molecule inhibitors against intracellular replication of Cryptococcus neoformans in macrophagesCryptococcal interactions with the host immune system.Direct cell-to-cell spread of a pathogenic yeast.Cell-to-cell spread and massive vacuole formation after Cryptococcus neoformans infection of murine macrophagesPI3K signaling of autophagy is required for starvation tolerance and virulenceof Cryptococcus neoformans.Extracellular vesicles from Cryptococcus neoformans modulate macrophage functionsThe interaction between Candida krusei and murine macrophages results in multiple outcomes, including intracellular survival and escape from killing.Characterization of Cryptococcus neoformans variety gattii SOD2 reveals distinct roles of the two superoxide dismutases in fungal biology and virulence.Murine IgG1 and IgG3 isotype switch variants promote phagocytosis of Cryptococcus neoformans through different receptorsA putative P-type ATPase, Apt1, is involved in stress tolerance and virulence in Cryptococcus neoformans.Cryptococcus neoformans variants generated by phenotypic switching differ in virulence through effects on macrophage activation.Spleen deposition of Cryptococcus neoformans capsular glucuronoxylomannan in rodents occurs in red pulp macrophages and not marginal zone macrophages expressing the C-type lectin SIGN-R1Phospholipids trigger Cryptococcus neoformans capsular enlargement during interactions with amoebae and macrophages.Cryptococcus neoformans {alpha} strains preferentially disseminate to the central nervous system during coinfectionA sensitive high-throughput assay for evaluating host-pathogen interactions in Cryptococcus neoformans infectionCryptococcus neoformans hyperfilamentous strain is hypervirulent in a murine model of cryptococcal meningoencephalitis.Analysis of cell cycle and replication of mouse macrophages after in vivo and in vitro Cryptococcus neoformans infection using laser scanning cytometryRegulatory circuitry governing fungal development, drug resistance, and disease.Nonlytic exocytosis of Cryptococcus neoformans from macrophages occurs in vivo and is influenced by phagosomal pH.The vacuolar Ca²(+) exchanger Vcx1 is involved in calcineurin-dependent Ca²(+) tolerance and virulence in Cryptococcus neoformans.Cryptococcus interactions with macrophages: evasion and manipulation of the phagosome by a fungal pathogen.Mechanisms of microbial escape from phagocyte killing.Analysis of the secretomes of Paracoccidioides mycelia and yeast cells.
P2860
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P2860
Replication of Cryptococcus neoformans in macrophages is accompanied by phagosomal permeabilization and accumulation of vesicles containing polysaccharide in the cytoplasm
description
2002 nî lūn-bûn
@nan
2002 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի մարտին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@ast
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@en
type
label
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@ast
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@en
prefLabel
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@ast
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@en
P2860
P356
P1476
Replication of Cryptococcus ne ...... olysaccharide in the cytoplasm
@en
P2093
Stephanie C Tucker
P2860
P304
P356
10.1073/PNAS.052702799
P407
P577
2002-03-01T00:00:00Z