Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination. - Wikidata - wikimedia - TriplyDB

Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.

Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.

http://www.wikidata.org/entity/Q30479247

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Neuroprotective effects of estrogens and androgens in CNS inflammation and neurodegeneration Estrogen or estrogen receptor agonist inhibits lipopolysaccharide induced microglial activation and death R-flurbiprofen attenuates experimental autoimmune encephalomyelitis in mice Pregnancy, sex and hormonal factors in multiple sclerosis.Nudging oligodendrocyte intrinsic signaling to remyelinate and repair: Estrogen receptor ligand effects.Estrogen receptor β ligand therapy activates PI3K/Akt/mTOR signaling in oligodendrocytes and promotes remyelination in a mouse model of multiple sclerosis.Estrogen receptor (ER) β expression in oligodendrocytes is required for attenuation of clinical disease by an ERβ ligand Effect of catalpol on remyelination through experimental autoimmune encephalomyelitis acting to promote Olig1 and Olig2 expressions in mice.Astrocytic TGF-β signaling limits inflammation and reduces neuronal damage during central nervous system Toxoplasma infection.Angiogenesis in multiple sclerosis and experimental autoimmune encephalomyelitis.Development of subtype-selective oestrogen receptor-based therapeutics.Multiple functional therapeutic effects of the estrogen receptor β agonist indazole-Cl in a mouse model of multiple sclerosis Restoration of axon conduction and motor deficits by therapeutic treatment with glatiramer acetate.Estriol preserves synaptic transmission in the hippocampus during autoimmune demyelinating disease.Sex-related factors in multiple sclerosis susceptibility and progression.Axon diameter and axonal transport: In vivo and in vitro effects of androgens.Estrogen treatment prevents gray matter atrophy in experimental autoimmune encephalomyelitis.The microstructural correlates of T1 in white matter Sex-specific differences in retinal nerve fiber layer thinning after acute optic neuritis.Therapeutic testosterone administration preserves excitatory synaptic transmission in the hippocampus during autoimmune demyelinating disease Attenuation of corpus callosum axon myelination and remyelination in the absence of circulating sex hormones Icariin exerts estrogen-like activity in ameliorating EAE via mediating estrogen receptor β, modulating HPA function and glucocorticoid receptor expression.Estrogen mediates neuroprotection and anti-inflammatory effects during EAE through ERα signaling on astrocytes but not through ERβ signaling on astrocytes or neurons.Simulations on the influence of myelin water in diffusion-weighted imaging.Tamoxifen accelerates the repair of demyelinated lesions in the central nervous system.Therapeutic laquinimod treatment decreases inflammation, initiates axon remyelination, and improves motor deficit in a mouse model of multiple sclerosis.17β-Estradiol Promotes Schwann Cell Proliferation and Differentiation, Accelerating Early Remyelination in a Mouse Peripheral Nerve Injury Model.Enhancing remyelination in disease--can we wrap it up?Neuroprotection and estrogen receptors.Neuroprotection in multiple sclerosis: a therapeutic approach.Sex disparities in neuro-ophthalmologic disorders.Intracellular signaling pathway regulation of myelination and remyelination in the CNS.Intraventricular injections of mesenchymal stem cells activate endogenous functional remyelination in a chronic demyelinating murine model.The importance of studying sex differences in disease: The example of multiple sclerosis.Role of Oligodendrocyte Dysfunction in Demyelination, Remyelination and Neurodegeneration in Multiple Sclerosis.Structure-based quantitative structure--activity relationship modeling of estrogen receptor β-ligands.The promyelinating properties of androstenediol in gliotoxin-induced demyelination in rat corpus callosum.Association between neurovascular contact and clinical characteristics in classical trigeminal neuralgia: A prospective clinical study using 3.0 Tesla MRI.Progesterone Enhanced Remyelination in the Mouse Corpus Callosum after Cuprizone Induced Demyelination.Bedside to bench to bedside research: Estrogen receptor beta ligand as a candidate neuroprotective treatment for multiple sclerosis.

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Oestrogen receptor beta ligand: a novel treatment to enhance endogenous functional remyelination.

http://www.wikidata.org/entity/Q30479247

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2010 nî lūn-bûn

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2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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Bingbing Song

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Daniel K Crawford

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Mario Mangiardi

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Michael V Sofroniew

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Rhonda R Voskuhl

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Rhusheet Patel

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Seema K Tiwari-Woodruff

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Sienmi Du

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P2860

Differential neuroprotective and antiinflammatory effects of estrogen receptor (ER)alpha and ERbeta ligand treatment

The immunopathology of multiple sclerosis: an overview

Activation of the mammalian target of rapamycin (mTOR) is essential for oligodendrocyte differentiation

Thyroid hormone administration enhances remyelination in chronic demyelinating inflammatory disease

Remyelination in the CNS: from biology to therapy

Neuroprotective and anti-inflammatory effects of estrogen receptor ligand treatment in mice.

Estrogen receptor beta protects against acoustic trauma in mice

IGF-1 protects oligodendrocyte progenitors against TNFalpha-induced damage by activation of PI3K/Akt and interruption of the mitochondrial apoptotic pathway.

MRI of the corpus callosum in multiple sclerosis: association with disability.

Insulin-like growth factor-I-stimulated Akt phosphorylation and oligodendrocyte progenitor cell survival require cholesterol-enriched membranes.

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10.1093/BRAIN/AWQ237

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