G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility. - Wikidata - wikimedia - TriplyDB

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

http://www.wikidata.org/entity/Q30480512

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Numb links extracellular cues to intracellular polarity machinery to promote chemotaxis An excitable cortex and memory model successfully predicts new pseudopod dynamics Parathyroid Hormone Induces Bone Cell Motility and Loss of Mature Osteocyte Phenotype through L-Calcium Channel Dependent and Independent Mechanisms A continuum model of actin waves in Dictyostelium discoideum Activated membrane patches guide chemotactic cell motility A High-Throughput, Multi-Cell Phenotype Assay for the Identification of Novel Inhibitors of Chemotaxis/Migration.Gβ Regulates Coupling between Actin Oscillators for Cell Polarity and Directional Migration Ordered patterns of cell shape and orientational correlation during spontaneous cell migration Insulin granule recruitment and exocytosis is dependent on p110gamma in insulinoma and human beta-cells Curvature recognition and force generation in phagocytosis.Migrating fibroblasts reorient directionality by a metastable, PI3K-dependent mechanism Spontaneous phosphoinositide 3-kinase signaling dynamics drive spreading and random migration of fibroblasts Four key signaling pathways mediating chemotaxis in Dictyostelium discoideum.Open access microfluidic device for the study of cell migration during chemotaxis.Switching direction in electric-signal-induced cell migration by cyclic guanosine monophosphate and phosphatidylinositol signaling Self-organizing actin waves as planar phagocytic cup structures.A Rap/phosphatidylinositol 3-kinase pathway controls pseudopod formation [corrected].An unconventional myosin required for cell polarization and chemotaxis A Ras signaling complex controls the RasC-TORC2 pathway and directed cell migration.Self-organization of the phosphatidylinositol lipids signaling system for random cell migration An MEK-cofilin signalling module controls migration of human T cells in 3D but not 2D environments.The LRRK2-related Roco kinase Roco2 is regulated by Rab1A and controls the actin cytoskeleton Molecular basis of dynamic relocalization of Dictyostelium myosin IB Synthetic spatially graded Rac activation drives cell polarization and movement.Delineating the core regulatory elements crucial for directed cell migration by examining folic-acid-mediated responses Phase geometries of two-dimensional excitable waves govern self-organized morphodynamics of amoeboid cells.Dictyostelium Ric8 is a nonreceptor guanine exchange factor for heterotrimeric G proteins and is important for development and chemotaxis.The small GTPase HRas shapes local PI3K signals through positive feedback and regulates persistent membrane extension in migrating fibroblasts An excitable signal integrator couples to an idling cytoskeletal oscillator to drive cell migration.UNC-6 (netrin) stabilizes oscillatory clustering of the UNC-40 (DCC) receptor to orient polarity.Rectified directional sensing in long-range cell migration Fast-crawling cell types migrate to avoid the direction of periodic substratum stretching.A plasma membrane template for macropinocytic cups.Coupled excitable Ras and F-actin activation mediates spontaneous pseudopod formation and directed cell movement Cooperation of Mtmr8 with PI3K regulates actin filament modeling and muscle development in zebrafish Directional persistence of cell migration coincides with stability of asymmetric intracellular signaling.Rapid induction of apoptosis by PI3K inhibitors is dependent upon their transient inhibition of RAS-ERK signaling.Moving in the right direction: how eukaryotic cells migrate along chemical gradients.Involvement of the cytoskeleton in controlling leading-edge function during chemotaxis.A comparison of mathematical models for polarization of single eukaryotic cells in response to guided cues.

P2860

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P2860

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

http://www.wikidata.org/entity/Q30480512

description

2007 nî lūn-bûn

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2007 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2007 թվականի հուլիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@ast

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@en

type

label

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@ast

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@en

prefLabel

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@ast

G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

@en

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Journal of Cell Biology

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G protein-independent Ras/PI3K/F-actin circuit regulates basic cell motility.

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P2093

Atsuo T Sasaki

http://www.w3.org/2001/XMLSchema#string

Chris Janetopoulos

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Kosuke Takeda

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Lauren W Sundheimer

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Pascale G Charest

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Peter N Devreotes

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Richard A Firtel

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Ruedi Meili

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Susan Lee

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P2860

Roles of G beta gamma in membrane recruitment and activation of p110 gamma/p101 phosphoinositide 3-kinase gamma

Crystal structure and functional analysis of Ras binding to its effector phosphoinositide 3-kinase gamma

Cloning and characterization of a G protein-activated human phosphoinositide-3 kinase

p84, a new Gbetagamma-activated regulatory subunit of the type IB phosphoinositide 3-kinase p110gamma

The G beta gamma sensitivity of a PI3K is dependent upon a tightly associated adaptor, p101

The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism

Chemoattractant-mediated transient activation and membrane localization of Akt/PKB is required for efficient chemotaxis to cAMP in Dictyostelium

TOR complex 2 integrates cell movement during chemotaxis and signal relay in Dictyostelium

A novel phosphoinositide 3 kinase activity in myeloid-derived cells is activated by G protein beta gamma subunits.

The PI3K-mediated activation of CRAC independently regulates adenylyl cyclase activation and chemotaxis.

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185-191

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10.1083/JCB.200611138

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2

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178

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17635933

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2064438

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