Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
about
Modulation of phagosomal pH by Candida albicans promotes hyphal morphogenesis and requires Stp2p, a regulator of amino acid transportStage specific assessment of Candida albicans phagocytosis by macrophages identifies cell wall composition and morphogenesis as key determinantsInhibitors of the glyoxylate cycle enzyme ICL1 in Candida albicans for potential use as antifungal agentsRecognition of yeast by murine macrophages requires mannan but not glucan.The interaction between Candida krusei and murine macrophages results in multiple outcomes, including intracellular survival and escape from killing.In vitro interactions of Candida parapsilosis wild type and lipase deficient mutants with human monocyte derived dendritic cellsThe functions of Mediator in Candida albicans support a role in shaping species-specific gene expression.Telomeric ORFs (TLOs) in Candida spp. Encode mediator subunits that regulate distinct virulence traits.Intracellular trafficking of AIP56, an NF-κB-cleaving toxin from Photobacterium damselae subsp. piscicidaG1/S transcription factor orthologues Swi4p and Swi6p are important but not essential for cell proliferation and influence hyphal development in the fungal pathogen Candida albicans.Regulation of innate immune response to Candida albicans infections by αMβ2-Pra1p interaction.IL-27 Induced by Select Candida spp. via TLR7/NOD2 Signaling and IFN-β Production Inhibits Fungal Clearance.Dual-species transcriptional profiling during systemic candidiasis reveals organ-specific host-pathogen interactions.Gene Ontology and the annotation of pathogen genomes: the case of Candida albicans.Fungal pathogens-a sweet and sour treat for toll-like receptorsHyphae-specific genes HGC1, ALS3, HWP1, and ECE1 and relevant signaling pathways in Candida albicans.The trophic life cycle stage of the opportunistic fungal pathogen Pneumocystis murina hinders the ability of dendritic cells to stimulate CD4(+) T cell responses.The pH-regulated antigen 1 of Candida albicans binds the human complement inhibitor C4b-binding protein and mediates fungal complement evasionCandida albicans cell surface superoxide dismutases degrade host-derived reactive oxygen species to escape innate immune surveillance.Candidalysin drives epithelial signaling, neutrophil recruitment, and immunopathology at the vaginal mucosa.Fungal Strategies to Evade the Host Immune Recognition.
P2860
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P2860
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
description
2008 nî lūn-bûn
@nan
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@ast
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@en
type
label
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@ast
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@en
prefLabel
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@ast
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions.
@en
P2093
P2860
P356
P1476
Analysis of PRA1 and its relationship to Candida albicans- macrophage interactions
@en
P2093
P2860
P304
P356
10.1128/IAI.00588-07
P407
P50
P577
2008-07-14T00:00:00Z