TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms. - Wikidata - wikimedia - TriplyDB

TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.

TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.

http://www.wikidata.org/entity/Q30488724

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Absence of histamine-induced itch in the African naked mole-rat and "rescue" by Substance P Harnessing the Therapeutic Potential of Capsaicin and Its Analogues in Pain and Other Diseases Update and clinical utility of alcaftadine ophthalmic solution 0.25% in the treatment of allergic conjunctivitis Why we scratch an itch: the molecules, cells and circuits of itch The functional and anatomical dissection of somatosensory subpopulations using mouse genetics Genetic enhancement of behavioral itch responses in mice lacking phosphoinositide 3-kinase-γ (PI3Kγ)New insights into the mechanisms of itch: are pain and itch controlled by distinct mechanisms?Basic mechanisms of itch.The cell biology of acute itch Role of Ceramide from Glycosphingolipids and Its Metabolites in Immunological and Inflammatory Responses in Humans Neuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms?Tlx3 and Runx1 act in combination to coordinate the development of a cohort of nociceptors, thermoceptors, and pruriceptors Facilitation of TRPV4 by TRPV1 is required for itch transmission in some sensory neuron populations Atopic dermatitis: allergic dermatitis or neuroimmune dermatitis?The multiple pathways for itch and their interactions with pain Neural processing of itch.VGLUT2-dependent sensory neurons in the TRPV1 population regulate pain and itch.A subpopulation of nociceptors specifically linked to itch The cells and circuitry for itch responses in mice.Activity-dependent silencing reveals functionally distinct itch-generating sensory neurons.Chronic itch development in sensory neurons requires BRAF signaling pathways.Neural peptidase endothelin-converting enzyme 1 regulates endothelin 1-induced pruritus Three functionally distinct classes of C-fibre nociceptors in primates.TRPA1-dependent pruritus in IL-13-induced chronic atopic dermatitis.Distinct functions of opioid-related peptides and gastrin-releasing peptide in regulating itch and pain in the spinal cord of primates.The peripheral and central mechanisms underlying itch Transient receptor potential (TRP) channels: a clinical perspective Recent advances in the management of pruritus in chronic liver diseases Functional neurokinin and NMDA receptor activity in an animal naturally lacking substance P: the naked mole-rat Molecular signatures of mouse TRPV1-lineage neurons revealed by RNA-Seq transcriptome analysis.Tlx3 Function in the Dorsal Root Ganglion is Pivotal to Itch and Pain Sensations.Itch mechanisms and circuits.Pirt, a TRPV1 modulator, is required for histamine-dependent and -independent itch.A randomised trial evaluating the effects of the TRPV1 antagonist SB705498 on pruritus induced by histamine, and cowhage challenge in healthy volunteers Pain behavior in the formalin test persists after ablation of the great majority of C-fiber nociceptors Spontaneous itch in the absence of hyperalgesia in a mouse hindpaw dry skin model Recent advances in pruritus - what we have learned and where are we headed.Enhanced scratching evoked by PAR-2 agonist and 5-HT but not histamine in a mouse model of chronic dry skin itch.Labeled lines meet and talk: population coding of somatic sensations.CGRPα-expressing sensory neurons respond to stimuli that evoke sensations of pain and itch.

P2860

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P2860

TRPV1-expressing primary afferents generate behavioral responses to pruritogens via multiple mechanisms.

http://www.wikidata.org/entity/Q30488724

description

2009 nî lūn-bûn

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2009 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հունիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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type

label

TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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prefLabel

TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

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PNAS.0905605106

P1433

Proceedings of the National Academy of Sciences of the United States of America

P1476

TRPV1-expressing primary affer ...... ogens via multiple mechanisms.

@en

P2093

Allan I Basbaum

http://www.w3.org/2001/XMLSchema#string

David J Anderson

http://www.w3.org/2001/XMLSchema#string

Goon Ho Park

http://www.w3.org/2001/XMLSchema#string

Hyosang Lee

http://www.w3.org/2001/XMLSchema#string

Melvin I Simon

http://www.w3.org/2001/XMLSchema#string

Noritaka Imamachi

http://www.w3.org/2001/XMLSchema#string

Sang-Kyou Han

http://www.w3.org/2001/XMLSchema#string

P2860

Cowhage-evoked itch is mediated by a novel cysteine protease: a ligand of protease-activated receptors

Itch: scratching more than the surface

The neurobiology of itch

Primary afferent tachykinins are required to experience moderate to intense pain

Impaired nociception and pain sensation in mice lacking the capsaicin receptor

Frontiers in pruritus research: scratching the brain for more effective itch therapy

Direct activation of transient receptor potential vanilloid 1(TRPV1) by diacylglycerol (DAG).

Psychophysical and physiological evidence for parallel afferent pathways mediating the sensation of itch.

Spinothalamic lamina I neurons selectively sensitive to histamine: a central neural pathway for itch.

The itch-producing agents histamine and cowhage activate separate populations of primate spinothalamic tract neurons.

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27

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106

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2009-06-29T00:00:00Z

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26328634

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19564617

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2708751

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