Transcription factor SP4 is a susceptibility gene for bipolar disorder
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Restoration of Sp4 in Forebrain GABAergic Neurons Rescues Hypersensitivity to Ketamine in Sp4 Hypomorphic Mice.Toxicogenomic and phenotypic analyses of bisphenol-A early-life exposure toxicity in zebrafishNovel loci for major depression identified by genome-wide association study of Sequenced Treatment Alternatives to Relieve Depression and meta-analysis of three studiesAnalysis of 94 candidate genes and 12 endophenotypes for schizophrenia from the Consortium on the Genetics of Schizophrenia.Prolonged Ketamine Effects in Sp4 Hypomorphic Mice: Mimicking Phenotypes of Schizophrenia.Genetic models of sensorimotor gating: relevance to neuropsychiatric disorders.Prepulse inhibition and genetic mouse models of schizophrenia.Reduced NMDAR1 expression in the Sp4 hypomorphic mouse may contribute to endophenotypes of human psychiatric disordersStore-operated calcium entry promotes the degradation of the transcription factor Sp4 in resting neurons.GlyT-1 Inhibition Attenuates Attentional But Not Learning or Motivational Deficits of the Sp4 Hypomorphic Mouse Model Relevant to Psychiatric DisordersThe genetics of major depression: moving beyond the monoamine hypothesis.Overview of the genetics of major depressive disorderAre Bipolar Disorder and Schizophrenia Neuroanatomically Distinct? An Anatomical Likelihood Meta-analysis.Mutation of murine Sox4 untranslated regions results in partially penetrant perinatal lethality.Transcription factor Sp4 regulates expression of nervous wreck 2 to control NMDAR1 levels and dendrite patterning.Rare variants in neuronal excitability genes influence risk for bipolar disorder.The transcription factor SP4 is reduced in postmortem cerebellum of bipolar disorder subjects: control by depolarization and lithium.Phosphorylation of transcription factor specificity protein 4 is increased in peripheral blood mononuclear cells of first-episode psychosisA composite peripheral blood gene expression measure as a potential diagnostic biomarker in bipolar disorder.Transcription factor SP4 phosphorylation is altered in the postmortem cerebellum of bipolar disorder and schizophrenia subjectsModeling neurodevelopmental cognitive deficits in tasks with cross-species translational validityFurther evidence for linkage of bipolar disorder to chromosomes 6 and 17 in a new independent pedigree series.Neuronal STIMulation at rest.miR-124, -128, and -137 Orchestrate Neural Differentiation by Acting on Overlapping Gene Sets Containing a Highly Connected Transcription Factor Network.Phosphorylation of the transcription factor Sp4 is reduced by NMDA receptor signaling.Mechanisms regulating dendritic arbor patterning.Species composition and environmental adaptation of indigenous Chinese cattle.Hot Genes in Schizophrenia: How Clinical Datasets Could Help to Refine their Role.Ketamine independently modulated power and phase-coupling of theta oscillations in Sp4 hypomorphic mice.
P2860
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P2860
Transcription factor SP4 is a susceptibility gene for bipolar disorder
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2009 nî lūn-bûn
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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2009 թվականի ապրիլին հրատարակված գիտական հոդված
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2009年の論文
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年学术文章
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2009年學術文章
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Transcription factor SP4 is a susceptibility gene for bipolar disorder
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Transcription factor SP4 is a susceptibility gene for bipolar disorder
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type
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Transcription factor SP4 is a susceptibility gene for bipolar disorder
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Transcription factor SP4 is a susceptibility gene for bipolar disorder
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prefLabel
Transcription factor SP4 is a susceptibility gene for bipolar disorder
@ast
Transcription factor SP4 is a susceptibility gene for bipolar disorder
@en
P2093
P2860
P1433
P1476
Transcription factor SP4 is a susceptibility gene for bipolar disorder
@en
P2093
John R Kelsoe
Mark A Geyer
Shengzhen Guo
Xianjin Zhou
P2860
P356
10.1371/JOURNAL.PONE.0005196
P407
P577
2009-04-09T00:00:00Z