ATP and NO dually control migration of microglia to nerve lesions.
about
Multiple sclerosis: molecular mechanisms and therapeutic opportunitiesBidirectional microglia-neuron communication in the healthy brainIn Vivo imaging reveals distinct inflammatory activity of CNS microglia versus PNS macrophages in a mouse model for ALSA Zebrafish Drug-Repurposing Screen Reveals sGC-Dependent and sGC-Independent Pro-Inflammatory Activities of Nitric OxideNitric Oxide Modulates Postnatal Bone Marrow-Derived Mesenchymal Stem Cell MigrationInnexin and pannexin channels and their signaling.Neuroglial ATP release through innexin channels controls microglial cell movement to a nerve injuryInfluence of methylene blue on microglia-induced inflammation and motor neuron degeneration in the SOD1(G93A) model for ALS.GCH1, BH4 and painThe evolving landscape of neurotoxicity by unconjugated bilirubin: role of glial cells and inflammation.The changing phenotype of microglia from homeostasis to diseaseMicroglia dynamics and function in the CNS.Central sensitization: a generator of pain hypersensitivity by central neural plasticity.Central nervous system regeneration: from leech to opossum.New insights regarding the regulation of chemotaxis by nucleotides, adenosine, and their receptors.Animal models for studying microglia: the first, the popular, and the new.The leech nervous system: a valuable model to study the microglia involvement in regenerative processes.ATP release through pannexon channelsRegulation of microglial migration, phagocytosis, and neurite outgrowth by HO-1/CO signaling.Calreticulin contributes to C1q-dependent recruitment of microglia in the leech Hirudo medicinalis following a CNS injuryMinocycline reduces neuronal death and attenuates microglial response after pediatric asphyxial cardiac arrestArachidonic acid closes innexin/pannexin channels and thereby inhibits microglia cell movement to a nerve injury.Microglia of medicinal leech (Hirudo medicinalis) express a specific activation marker homologous to vertebrate ionized calcium-binding adapter molecule 1 (Iba1/alias aif-1).Minocycline attenuates microglial response and reduces neuronal death after cardiac arrest and cardiopulmonary resuscitation in mice.Glial Cells and Integrity of the Nervous System.Involvement of nitric oxide through endocannabinoids release in microglia activation during the course of CNS regeneration in the medicinal leechExtracellular Degradation Into Adenosine and the Activities of Adenosine Kinase and AMPK Mediate Extracellular NAD-Produced Increases in the Adenylate Pool of BV2 Microglia Under Basal Conditions
P2860
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P2860
ATP and NO dually control migration of microglia to nerve lesions.
description
2009 nî lūn-bûn
@nan
2009 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
ATP and NO dually control migration of microglia to nerve lesions.
@ast
ATP and NO dually control migration of microglia to nerve lesions.
@en
type
label
ATP and NO dually control migration of microglia to nerve lesions.
@ast
ATP and NO dually control migration of microglia to nerve lesions.
@en
prefLabel
ATP and NO dually control migration of microglia to nerve lesions.
@ast
ATP and NO dually control migration of microglia to nerve lesions.
@en
P2093
P2860
P356
P1476
ATP and NO dually control migration of microglia to nerve lesions.
@en
P2093
Christie L Sahley
Kenneth J Muller
Yuanli Duan
P2860
P356
10.1002/DNEU.20689
P577
2009-01-01T00:00:00Z