Stability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine. - Wikidata - wikimedia - TriplyDB

Stability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine.

Stability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine.

http://www.wikidata.org/entity/Q30490470

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Roles of the ubiquitin proteasome system in the effects of drugs of abuse The human epilepsy mutation GABRG2(Q390X) causes chronic subunit accumulation and neurodegeneration Tspyl2 Loss-of-Function Causes Neurodevelopmental Brain and Behavior Abnormalities in Mice.Constitutive knockout of kalirin-7 leads to increased rates of cocaine self-administration.Neuronal Abelson helper integration site-1 (Ahi1) deficiency in mice alters TrkB signaling with a depressive phenotype.CaMKIIalpha interacts with M4 muscarinic receptors to control receptor and psychomotor function Amphetamine alters Ras-guanine nucleotide-releasing factor expression in the rat striatum in vivo Setdb1 histone methyltransferase regulates mood-related behaviors and expression of the NMDA receptor subunit NR2B.Regulation of group I metabotropic glutamate receptor expression in the rat striatum and prefrontal cortex in response to amphetamine in vivo Behavioral and morphological responses to cocaine require kalirin7.Analysis of proteome dynamics in the mouse brain Immunogold electron microscopic evidence of differential regulation of GluN1, GluN2A, and GluN2B, NMDA-type glutamate receptor subunits in rat hippocampal CA1 synapses during benzodiazepine withdrawal.Methylphenidate and the juvenile brain: enhancement of attention at the expense of cortical plasticity?Down-regulation of synaptic GluN2B subunit-containing N-methyl-D-aspartate receptors: a physiological brake on CA1 neuron α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid hyperexcitability during benzodiazepine withdrawal Post-translational modification biology of glutamate receptors and drug addiction Reversible palmitoylation regulates surface stability of AMPA receptors in the nucleus accumbens in response to cocaine in vivo.Ubiquitination in postsynaptic function and plasticity Altered sensitivity of cerebellar granule cells to glutamate receptor overactivation in the Cln3(Δex7/8)-knock-in mouse model of juvenile neuronal ceroid lipofuscinosis.Cocaine increases phosphorylation of MeCP2 in the rat striatum in vivo: a differential role of NMDA receptors.New medications for drug addiction hiding in glutamatergic neuroplasticity.Roles of Ca(2+)/calmodulin-dependent protein kinase II in subcellular expression of striatal N-methyl-D-aspartate receptors in l-3, 4-dihydroxyphenylalanine-induced dyskinetic rats.Dynamic loss of surface-expressed AMPA receptors in mouse cortical and striatal neurons during anesthesia.Heroin relapse requires long-term potentiation-like plasticity mediated by NMDA2b-containing receptors.Cocaine facilitates PKC maturation by upregulating its phosphorylation at the activation loop in rat striatal neurons in vivo.Dynamic increases in AMPA receptor phosphorylation in the rat hippocampus in response to amphetamine.Half-life of DISC1 protein and its pathological significance under hypoxia stress Failure to Recognize Novelty after Extended Methamphetamine Self-Administration Results from Loss of Long-Term Depression in the Perirhinal Cortex Regulated internalization of NMDA receptors drives PKD1-mediated suppression of the activity of residual cell-surface NMDA receptors.Deletion of adenosine A2A receptors from astrocytes disrupts glutamate homeostasis leading to psychomotor and cognitive impairment: relevance to schizophrenia Treatment with a clinically-relevant dose of methylphenidate alters NMDA receptor composition and synaptic plasticity in the juvenile rat prefrontal cortex.NMDA hypofunction as a convergence point for progression and symptoms of schizophrenia Locomotor sensitization to ethanol impairs NMDA receptor-dependent synaptic plasticity in the nucleus accumbens and increases ethanol self-administration.A critical role for protein degradation in the nucleus accumbens core in cocaine reward memory.Short and long access to cocaine self-administration activates tyrosine phosphatase STEP and attenuates GluN expression but differentially regulates GluA expression in the prefrontal cortex.Differential regulation of locomotor activity to acute and chronic cocaine administration by acid-sensing ion channel 1a and 2 in adult mice.Changes in surface expression of N-methyl-D-aspartate receptors in the striatum in a rat model of Parkinson's disease.Protein degradation and memory formation.Cocaine-induced metaplasticity in the nucleus accumbens: silent synapse and beyond.Scaffolding proteins of the post-synaptic density contribute to synaptic plasticity by regulating receptor localization and distribution: relevance for neuropsychiatric diseases.Do specific NMDA receptor subunits act as gateways for addictive behaviors?

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P2860

Stability of surface NMDA receptors controls synaptic and behavioral adaptations to amphetamine.

http://www.wikidata.org/entity/Q30490470

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2009 nî lūn-bûn

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2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2009 թվականի ապրիլին հրատարակված գիտական հոդված

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2009年の論文

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Nature Neuroscience

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Christopher J Papasian

http://www.w3.org/2001/XMLSchema#string

Eugene E Fibuch

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Guo-Chi Zhang

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Jian-Guo Chen

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John Q Wang

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Li-Min Mao

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Michelle Haines

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Wei Wang

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P2860

NEURAL MECHANISMS OF ADDICTION: The Role of Reward-Related Learning and Memory

Brain reward circuitry: insights from unsensed incentives

Interaction with newly synthesized and retained proteins in the endoplasmic reticulum suggests a chaperone function for human integral membrane protein IP90 (calnexin)

Regulation of the NMDA receptor complex and trafficking by activity-dependent phosphorylation of the NR2B subunit PDZ ligand

Activity-dependent NMDA receptor degradation mediated by retrotranslocation and ubiquitination

Modulation of D2R-NR2B interactions in response to cocaine

Ubiquitination regulates PSD-95 degradation and AMPA receptor surface expression

Identification of PSD-95 as a regulator of dopamine-mediated synaptic and behavioral plasticity

PDZ domain proteins of synapses

Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome system

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602-610

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10.1038/NN.2300

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5

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19349975

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