Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis. - Wikidata - wikimedia - TriplyDB

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

http://www.wikidata.org/entity/Q30496231

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Tipping the balance from angiogenesis to fibrosis in CKD The luminal connection: from animal development to lumopathies Tips, stalks, tubes: notch-mediated cell fate determination and mechanisms of tubulogenesis during angiogenesis Molecular mechanisms controlling vascular lumen formation in three-dimensional extracellular matrices Endothelial cell-cell adhesion during zebrafish vascular development Molecular control of endothelial cell behaviour during blood vessel morphogenesis Clusters of circulating tumor cells traverse capillary-sized vessels.Single-cell analysis of endothelial morphogenesis in vivo.Endothelial cilia are essential for developmental vascular integrity in zebrafish E-cadherin roles in animal biology: A perspective on thyroid hormone-influence Wnt7b Signaling from the Ureteric Bud Epithelium Regulates Medullary Capillary Development.An apical actin-rich domain drives the establishment of cell polarity during cell adhesion.VE-PTP regulates VEGFR2 activity in stalk cells to establish endothelial cell polarity and lumen formation PP2A regulatory subunit Bα controls endothelial contractility and vessel lumen integrity via regulation of HDAC7.Synergy of cell-cell repulsion and vacuolation in a computational model of lumen formation.Guidance of subcellular tubulogenesis by actin under the control of a synaptotagmin-like protein and Moesin.Fusing VE-cadherin to α-catenin impairs fetal liver hematopoiesis and lymph but not blood vessel formation.Interferon-induced transmembrane protein 1 regulates endothelial lumen formation during angiogenesis.'In parallel' interconnectivity of the dorsal longitudinal anastomotic vessels requires both VEGF signaling and circulatory flow In vivo analysis of hyaloid vasculature morphogenesis in zebrafish: A role for the lens in maturation and maintenance of the hyaloid.VE-cadherin interacts with cell polarity protein Pals1 to regulate vascular lumen formation.A Zebrafish Model of Cryptococcal Infection Reveals Roles for Macrophages, Endothelial Cells, and Neutrophils in the Establishment and Control of Sustained Fungemia.A novel perivascular cell population in the zebrafish brain.Demystification of animal symmetry: symmetry is a response to mechanical forces.Intracellular lumen extension requires ERM-1-dependent apical membrane expansion and AQP-8-mediated flux.Somatic mutagenesis with a Sleeping Beauty transposon system leads to solid tumor formation in zebrafish.ERM proteins at a glance CBFβ and RUNX1 are required at 2 different steps during the development of hematopoietic stem cells in zebrafish Overlapping and opposing functions of G protein-coupled receptor kinase 2 (GRK2) and GRK5 during heart development.Genomic approach to identify factors that drive the formation of three-dimensional structures by EA.hy926 endothelial cells.Role of the cytoskeleton in formation and maintenance of angiogenic sprouts A distinct mechanism of vascular lumen formation in Xenopus requires EGFL7 Tip cell-specific requirement for an atypical Gpr124- and Reck-dependent Wnt/β-catenin pathway during brain angiogenesis.Anti-apoptosis effects of vascular endothelial cadherin in experimental corneal neovascularization.Formation of a PKCζ/β-catenin complex in endothelial cells promotes angiopoietin-1-induced collective directional migration and angiogenic sprouting.Imaging tumour cell heterogeneity following cell transplantation into optically clear immune-deficient zebrafish.Alk1 controls arterial endothelial cell migration in lumenized vessels Fast revascularization of the injured area is essential to support zebrafish heart regeneration.Molecular basis for endothelial lumen formation and tubulogenesis during vasculogenesis and angiogenic sprouting.Tubulogenesis during blood vessel formation.

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P2860

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

http://www.wikidata.org/entity/Q30496231

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@ast

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@en

type

label

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@ast

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@en

prefLabel

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@ast

Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@en

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Moesin1 and Ve-cadherin are required in endothelial cells during in vivo tubulogenesis.

@en

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Aidas Nasevicius

http://www.w3.org/2001/XMLSchema#string

Jeffrey J Essner

http://www.w3.org/2001/XMLSchema#string

Jon D Larson

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Mark S Kaiser

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Maura McGrail

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Perry B Hackett

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Shannon A Wadman

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Sharon E Roberg-Perez

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Stephen C Ekker

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Ying Wang

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P2860

Selective plane illumination microscopy techniques in developmental biology.

Rho-kinase phosphorylates COOH-terminal threonines of ezrin/radixin/moesin (ERM) proteins and regulates their head-to-tail association

Stable vascular connections and remodeling require full expression of VE-cadherin in zebrafish embryos.

Targeted deficiency or cytosolic truncation of the VE-cadherin gene in mice impairs VEGF-mediated endothelial survival and angiogenesis

Role of vascular endothelial-cadherin in vascular morphogenesis

Phosphorylation of ERM proteins at filopodia induced by Cdc42

Mechanisms controlling human endothelial lumen formation and tube assembly in three-dimensional extracellular matrices

Ezrin is a downstream effector of trafficking PKC-integrin complexes involved in the control of cell motility

Ezrin is essential for epithelial organization and villus morphogenesis in the developing intestine

The molecular basis of vascular lumen formation in the developing mouse aorta

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3119-3128

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10.1242/DEV.048785

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18

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137

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20736288

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2926960

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