Phosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice. - Wikidata - wikimedia - TriplyDB

Phosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice.

Phosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice.

http://www.wikidata.org/entity/Q30497498

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Anger, emotion, and arrhythmias: from brain to heart Dysfunctional ryanodine receptors in the heart: new insights into complex cardiovascular diseases Ca(2+) signaling in the myocardium by (redox) regulation of PKA/CaMKII Abnormal Ca(2+) cycling in failing ventricular myocytes: role of NOS1-mediated nitroso-redox balance Mechanisms of altered Ca²⁺ handling in heart failure Role of leaky neuronal ryanodine receptors in stress-induced cognitive dysfunction Ca²⁺-dependent phosphorylation of Ca²⁺ cycling proteins generates robust rhythmic local Ca²⁺ releases in cardiac pacemaker cells.Axial tubule junctions control rapid calcium signaling in atria Functional abnormalities in iPSC-derived cardiomyocytes generated from CPVT1 and CPVT2 patients carrying ryanodine or calsequestrin mutations Role of RyR2 phosphorylation in heart failure and arrhythmias: Controversies around ryanodine receptor phosphorylation in cardiac disease.CaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Role of RyR2 phosphorylation in heart failure and arrhythmias: protein kinase A-mediated hyperphosphorylation of the ryanodine receptor at serine 2808 does not alter cardiac contractility or cause heart failure and arrhythmias The in vivo regulation of heart rate in the murine sinoatrial node by stimulatory and inhibitory heterotrimeric G proteins.Direct measurements of SR free Ca reveal the mechanism underlying the transient effects of RyR potentiation under physiological conditions.β-Adrenergic receptor stimulation causes cardiac hypertrophy via a Gβγ/Erk-dependent pathway.Mitochondrial calcium overload is a key determinant in heart failure.ß-Adrenergic stimulation increases RyR2 activity via intracellular Ca2+ and Mg2+ regulation Fractalkine depresses cardiomyocyte contractility Dysfunction in the βII spectrin-dependent cytoskeleton underlies human arrhythmia.A-kinase anchoring proteins: scaffolding proteins in the heart.Membrane potential and Ca2+ concentration dependence on pressure and vasoactive agents in arterial smooth muscle: A model.Cellular Hypertrophy and Increased Susceptibility to Spontaneous Calcium-Release of Rat Left Atrial Myocytes Due to Elevated Afterload.Nitric Oxide Synthase 1 Modulates Basal and β-Adrenergic-Stimulated Contractility by Rapid and Reversible Redox-Dependent S-Nitrosylation of the Heart Reactive oxygen species-targeted therapeutic interventions for atrial fibrillation.Effects of increased systolic Ca(2+) and β-adrenergic stimulation on Ca(2+) transient decline in NOS1 knockout cardiac myocytes.Crosstalk between RyR2 oxidation and phosphorylation contributes to cardiac dysfunction in mice with Duchenne muscular dystrophy.Modulation of myocardial contraction by peroxynitrite Calcium cycling proteins and heart failure: mechanisms and therapeutics Molecular mechanisms underlying cardiac protein phosphatase 2A regulation in heart.A Single Protein Kinase A or Calmodulin Kinase II Site Does Not Control the Cardiac Pacemaker Ca2+ Clock.Calstabin 2: An important regulator for learning and memory in mice.PKA phosphorylation of cardiac ryanodine receptor modulates SR luminal Ca2+ sensitivity.New evidence for coupled clock regulation of the normal automaticity of sinoatrial nodal pacemaker cells: bradycardic effects of ivabradine are linked to suppression of intracellular Ca²⁺ cycling.Calcium/calmodulin-dependent kinase II and nitric oxide synthase 1-dependent modulation of ryanodine receptors during β-adrenergic stimulation is restricted to the dyadic cleft PDE4B mediates local feedback regulation of β₁-adrenergic cAMP signaling in a sarcolemmal compartment of cardiac myocytes.Compartmentation of cAMP signalling in cardiomyocytes in health and disease.Calcium in the heart: from physiology to disease.Acute and training effects of resistance exercise on heart rate variability.Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer's disease-like pathologies and cognitive deficits.Regulation of sarcoplasmic reticulum Ca2+ release by serine-threonine phosphatases in the heart.

P2860

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P2860

Phosphorylation of the ryanodine receptor mediates the cardiac fight or flight response in mice.

http://www.wikidata.org/entity/Q30497498

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Phosphorylation of the ryanodi ...... ht or flight response in mice.

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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type

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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Journal of Clinical Investigation

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Phosphorylation of the ryanodi ...... ht or flight response in mice.

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P2093

Alexander Kushnir

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Andrew R Marks

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Jian Shan

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Jingdong Li

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Marco Mongillo

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Matthew J Betzenhauser

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Nicolas Lindegger

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Peter J Mohler

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Stephan E Lehnart

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Steven Reiken

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P2860

Transcriptional profile of isoproterenol-induced cardiomyopathy and comparison to exercise-induced cardiac hypertrophy and human cardiac failure

PKA phosphorylation dissociates FKBP12.6 from the calcium release channel (ryanodine receptor): defective regulation in failing hearts

FK506 binding protein associated with the calcium release channel (ryanodine receptor)

Phosphodiesterase 4D deficiency in the ryanodine-receptor complex promotes heart failure and arrhythmias

FKBP12.6 deficiency and defective calcium release channel (ryanodine receptor) function linked to exercise-induced sudden cardiac death

Cardiac excitation-contraction coupling

Role of CaMKIIdelta phosphorylation of the cardiac ryanodine receptor in the force frequency relationship and heart failure

Stress pathways and heart failure.

Ryanodine receptor/calcium release channel PKA phosphorylation: a critical mediator of heart failure progression.

Direct activation of cardiac pacemaker channels by intracellular cyclic AMP.

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4388-4398

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scholarly article

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10.1172/JCI32726

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P433

12

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120

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2010-11-22T00:00:00Z

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49630231

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21099118

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P921

adrenergic receptor signaling pathway involved in positive regulation of heart rate

positive regulation of ryanodine-sensitive calcium-release channel activity by adrenergic receptor signaling pathway involved in positive regulation of cardiac muscle contraction

phosphorylation

P932

2993575

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