Reducing excessive GABA-mediated tonic inhibition promotes functional recovery after stroke.
about
Pathophysiological power of improper tonic GABA(A) conductances in mature and immature models.NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currentsNon-Invasive Brain Stimulation to Enhance Post-Stroke RecoveryTraumatic Brain Injury and Neuronal Functionality Changes in Sensory CortexComputational neurorehabilitation: modeling plasticity and learning to predict recoveryNeuroplasticity in post-stroke gait recovery and noninvasive brain stimulationNovel Stroke Therapeutics: Unraveling Stroke Pathophysiology and Its Impact on Clinical TreatmentsThe Dark Side of the Force - Constraints and Complications of Cell Therapies for StrokeStress as necessary component of realistic recovery in animal models of experimental strokePriming the brain to capitalize on metaplasticity in stroke rehabilitationFunctional regulation of GABAA receptors in nervous system pathologiesThe interaction between training and plasticity in the poststroke brainEnhanced phasic GABA inhibition during the repair phase of stroke: a novel therapeutic targetRole of P38 MAPK on MMP Activity in Photothrombotic Stroke Mice as Measured using an Ultrafast MMP Activatable Probe.Reduced tonic inhibition after stroke promotes motor performance and epileptic seizures.Cortical hypoexcitation defines neuronal responses in the immediate aftermath of traumatic brain injuryTonic GABAA Receptors as Potential Target for the Treatment of Temporal Lobe Epilepsy.Emergent properties of neural repair: elemental biology to therapeutic conceptsCocktail treatment, a promising strategy to treat acute cerebral ischemic stroke?Alterations of the Ceramide Metabolism in the Peri-Infarct Cortex Are Independent of the Sphingomyelinase Pathway and Not Influenced by the Acid Sphingomyelinase Inhibitor FluoxetinePTEN degradation after ischemic stroke: a double-edged swordCross-modal plasticity results in increased inhibition in primary auditory cortical areas.GABA transporter-1 deficiency confers schizophrenia-like behavioral phenotypes.The GABA(A) receptor agonist THIP ameliorates specific behavioral deficits in the mouse model of fragile X syndrome.Beyond classical benzodiazepines: novel therapeutic potential of GABAA receptor subtypes.Targeted mini-strokes produce changes in interhemispheric sensory signal processing that are indicative of disinhibition within minutes.Closed-loop optogenetic control of thalamus as a tool for interrupting seizures after cortical injuryTransient focal cerebral ischemia induces long-term cognitive function deficit in an experimental ischemic stroke model.Novel test of motor and other dysfunctions in mouse neurological disease models.Studying the effects of transcranial direct-current stimulation in stroke recovery using magnetic resonance imaging.Optogenetic Approaches to Target Specific Neural Circuits in Post-stroke Recovery.Reducing GABAA-mediated inhibition improves forelimb motor function after focal cortical stroke in miceOptogenetic neuronal stimulation of the lateral cerebellar nucleus promotes persistent functional recovery after strokeAstrocytes convert network excitation to tonic inhibition of neuronsProtective efficacy of a single salvianolic acid A treatment on photothrombosis-induced sustained spatial memory impairmentsPEGylated insulin-like growth factor-I affords protection and facilitates recovery of lost functions post-focal ischemia.Histological, cellular and behavioral assessments of stroke outcomes after photothrombosis-induced ischemia in adult mice.Weakened rate-dependent depression of Hoffmann's reflex and increased motoneuron hyperactivity after motor cortical infarction in mice.GABA levels are decreased after stroke and GABA changes during rehabilitation correlate with motor improvement.Early and moderate sensory stimulation exerts a protective effect on perilesion representations of somatosensory cortex after focal ischemic damage.
P2860
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P2860
Reducing excessive GABA-mediated tonic inhibition promotes functional recovery after stroke.
description
2010 nî lūn-bûn
@nan
2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@ast
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@en
type
label
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@ast
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@en
prefLabel
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@ast
Reducing excessive GABA-mediat ...... ctional recovery after stroke.
@en
P2860
P50
P356
P1433
P1476
Reducing excessive GABA-mediat ...... nctional recovery after stroke
@en
P2093
Istvan Mody
Sarah E Macisaac
P2860
P2888
P304
P356
10.1038/NATURE09511
P407
P577
2010-11-03T00:00:00Z
P5875
P6179
1007638192