Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity. - Wikidata - wikimedia - TriplyDB

Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

http://www.wikidata.org/entity/Q30500787

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Disruption of a GATA4/Ankrd1 signaling axis in cardiomyocytes leads to sarcomere disarray: implications for anthracycline cardiomyopathy The Kruppel-like transcription factor KLF13 is a novel regulator of heart development Doxorubicin cardiomyopathy Monoamine oxidase B prompts mitochondrial and cardiac dysfunction in pressure overloaded hearts Chemotherapy-Induced Cardiotoxicity: Overview of the Roles of Oxidative Stress Exercise Prevention of Cardiovascular Disease in Breast Cancer Survivors Mitogen-activated protein kinase signaling in the heart: angels versus demons in a heart-breaking tale.Transcription factors GATA-4 and GATA-6 in human adrenocortical tumors Maintenance of adult cardiac function requires the chromatin factor Asxl2 The transcription factor GATA-6 regulates pathological cardiac hypertrophy Diminished GATA4 protein levels contribute to hyperglycemia-induced cardiomyocyte injury Regulation of Bcl-xL expression in lung vascular smooth muscle Transcription factor GATA4 inhibits doxorubicin-induced autophagy and cardiomyocyte death Gata4 and Sp1 regulate expression of the erythropoietin receptor in cardiomyocytes The zinc finger-only protein Zfp260 is a novel cardiac regulator and a nuclear effector of alpha1-adrenergic signaling.Small heat shock proteins Hspb7 and Hspb12 regulate early steps of cardiac morphogenesis.GATA transcription factor required for immunity to bacterial and fungal pathogens.SIRT1 mediates the protective function of Nkx2.5 during stress in cardiomyocytes.GATA4 and GATA5 are essential for heart and liver development in Xenopus embryos.Molecular remodeling of left and right ventricular myocardium in chronic anthracycline cardiotoxicity and post-treatment follow up.Cardiomyocyte death in doxorubicin-induced cardiotoxicity Phosphorylation-dependent degradation of p300 by doxorubicin-activated p38 mitogen-activated protein kinase in cardiac cells.Cardiovascular Late Effects and Exercise Treatment in Breast Cancer: Current Evidence and Future Directions 20(S)-Ginsenoside Rg3 is a novel inhibitor of autophagy and sensitizes hepatocellular carcinoma to doxorubicin Mechanisms of anthracycline cardiac injury: can we identify strategies for cardioprotection?Long-acting phosphodiesterase-5 inhibitor tadalafil attenuates doxorubicin-induced cardiomyopathy without interfering with chemotherapeutic effect.An endocardial pathway involving Tbx5, Gata4, and Nos3 required for atrial septum formation.Paracrine factors released by GATA-4 overexpressed mesenchymal stem cells increase angiogenesis and cell survival Anthracycline cardiotoxicity: from bench to bedside.Alpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.Ageing is a risk factor in imatinib mesylate cardiotoxicity Weekly doxorubicin increases coronary arteriolar wall and adventitial thickness.Celastrol, an oral heat shock activator, ameliorates multiple animal disease models of cell death.Carboxy terminus of GATA4 transcription factor is required for its cardiogenic activity and interaction with CDK4.Mechanism of anthracycline-mediated down-regulation of GATA4 in the heart Attenuation of Doxorubicin-induced Cardiotoxicity by Tadalafil: A Long Acting Phosphodiesterase-5 Inhibitor.Alpha-1-adrenergic receptors: targets for agonist drugs to treat heart failure.Gata4 is required for maintenance of postnatal cardiac function and protection from pressure overload-induced heart failure Modulation of anthracycline-induced cardiotoxicity by aerobic exercise in breast cancer: current evidence and underlying mechanisms.Loss of Gata5 in mice leads to bicuspid aortic valve.

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P2860

Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

http://www.wikidata.org/entity/Q30500787

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2004 nî lūn-bûn

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2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年论文

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Proceedings of the National Academy of Sciences of the United States of America

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Essential role of GATA-4 in cell survival and drug-induced cardiotoxicity.

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Anne Aries

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Chantal Lefebvre

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Pierre Paradis

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Robert J Schwartz

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P2860

Serum response factor-GATA ternary complex required for nuclear signaling by a G-protein-coupled receptor

GATA-dependent recruitment of MEF2 proteins to target promoters

Angiotensin II type1a receptor gene expression in the heart: AP-1 and GATA-4 participate in the response to pressure overload

Mechanisms of caspase activation and inhibition during apoptosis.

Fetal anemia and apoptosis of red cell progenitors in Stat5a-/-5b-/- mice: a direct role for Stat5 in Bcl-X(L) induction

Transcriptional regulation of erythropoiesis: an affair involving multiple partners

Proteasome-mediated degradation of the coactivator p300 impairs cardiac transcription

Tissue-specific GATA factors are transcriptional effectors of the small GTPase RhoA

Targeted disruption of the GATA3 gene causes severe abnormalities in the nervous system and in fetal liver haematopoiesis

Transcription factor GATA-1 permits survival and maturation of erythroid precursors by preventing apoptosis

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