p120-catenin is essential for terminal end bud function and mammary morphogenesis.
about
Adhesion in mammary development: novel roles for E-cadherin in individual and collective cell migrationSRChing for the substrates of Src.p120-catenin prevents multinucleation through control of MKLP1-dependent RhoA activity during cytokinesis.DIPA-family coiled-coils bind conserved isoform-specific head domain of p120-catenin family: potential roles in hydrocephalus and heterotopia.Atbf1 regulates pubertal mammary gland development likely by inhibiting the pro-proliferative function of estrogen-ER signaling.p120 catenin is required for the stress response in DrosophilaA strategy for tissue self-organization that is robust to cellular heterogeneity and plasticityPhosphorylation and isoform use in p120-catenin during development and tumorigenesisp120-catenin binding masks an endocytic signal conserved in classical cadherins.Guilt by association: what p120-catenin has to hide.p120-catenin controls contractility along the vertical axis of epithelial lateral membranesManganese superoxide dismutase is dispensable for post-natal development and lactation in the murine mammary glandRhoA is down-regulated at cell-cell contacts via p190RhoGAP-B in response to tensional homeostasis.p120 Catenin is required for normal tubulogenesis but not epithelial integrity in developing mouse pancreas.p120-Catenin Is Critical for the Development of Invasive Lobular Carcinoma in Micep120-catenin down-regulation and epidermal growth factor receptor overexpression results in a transformed epithelium that mimics esophageal squamous cell carcinomaβ-Catenin haploinsufficiency promotes mammary tumorigenesis in an ErbB2-positive basal breast cancer model.p120 Catenin Suppresses Basal Epithelial Cell Extrusion in Invasive Pancreatic Neoplasia.Slug/Pcad pathway controls epithelial cell dynamics in mammary gland and breast carcinoma.Stromal matrix metalloproteinase-11 is involved in the mammary gland postnatal development.p120-Catenin is an obligate haploinsufficient tumor suppressor in intestinal neoplasia.ARVCF expression is significantly correlated with the malignant phenotype of non-small cell lung cancer.NFkB hyperactivation causes invasion of esophageal squamous cell carcinoma with EGFR overexpression and p120-catenin down-regulation.
P2860
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P2860
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մարտին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@ast
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@en
type
label
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@ast
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@en
prefLabel
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@ast
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@en
P2093
P2860
P50
P356
P1433
P1476
p120-catenin is essential for terminal end bud function and mammary morphogenesis.
@en
P2093
Albert B Reynolds
Ilse Hofmann
Michael A Davis
Nichole A Lobdell
Sarah J Kurley
William J Muller
P2860
P304
P356
10.1242/DEV.072769
P407
P577
2012-03-29T00:00:00Z