Deletion of Rictor in neural progenitor cells reveals contributions of mTORC2 signaling to tuberous sclerosis complex.
about
The neurology of mTORmTOR signaling in tumorigenesisTricornered Kinase Regulates Synapse Development by Regulating the Levels of Wiskott-Aldrich Syndrome ProteinEvaluation of diffusion kurtosis imaging in ex vivo hypomyelinated mouse brains.Rictor/mTORC2 facilitates central regulation of energy and glucose homeostasis.Rictor is an independent prognostic factor for endometrial carcinoma.mTORC1-independent reduction of retinal protein synthesis in type 1 diabetes.Regulation of endothelial cell proliferation and vascular assembly through distinct mTORC2 signaling pathways.Hypomyelination following deletion of Tsc2 in oligodendrocyte precursors.mTOR: a link from the extracellular milieu to transcriptional regulation of oligodendrocyte development.mTORC1 is necessary but mTORC2 and GSK3β are inhibitory for AKT3-induced axon regeneration in the central nervous system.Advancing the discovery of medications for autism spectrum disorder using new technologies to reveal social brain circuitry in rodents.Diabetic retinopathy: loss of neuroretinal adaptation to the diabetic metabolic environment.mTOR signaling and its roles in normal and abnormal brain development.Neuron membrane trafficking and protein kinases involved in autism and ADHD.Evaluating g-ratio weighted changes in the corpus callosum as a function of age and sex.mTORC2 controls actin polymerization required for consolidation of long-term memory.Multi-compartment microscopic diffusion imaging.Quantitative analysis of mouse corpus callosum from electron microscopy images.NGF signaling in PC12 cells: the cooperation of p75(NTR) with TrkA is needed for the activation of both mTORC2 and the PI3K signalling cascade.A revised model for estimating g-ratio from MRI.Loss of mTORC2 signaling in oligodendrocyte precursor cells delays myelination.Experimental studies of g-ratio MRI in ex vivo mouse brain.Neural Progenitor Cells Rptor Ablation Impairs Development but Benefits to Seizure-Induced Behavioral Abnormalities.Dendrite Growth and the Effect of Ectopic Rheb Expression on Cortical Neurons.Developmental brain abnormalities in tuberous sclerosis complex: a comparative tissue analysis of cortical tubers and perituberal cortex.Myelin volume fraction imaging with MRI.Coordination of Necessary and Permissive Signals by PTEN Inhibition for CNS Axon Regeneration
P2860
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P2860
Deletion of Rictor in neural progenitor cells reveals contributions of mTORC2 signaling to tuberous sclerosis complex.
description
2012 nî lūn-bûn
@nan
2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@ast
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@en
type
label
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@ast
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@en
prefLabel
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@ast
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@en
P2093
P2860
P356
P1476
Deletion of Rictor in neural p ...... to tuberous sclerosis complex.
@en
P2093
Kevin C Ess
Peggy Winzenburger
P2860
P304
P356
10.1093/HMG/DDS414
P577
2012-10-09T00:00:00Z