Enterocyte loss of polarity and gut wound healing rely upon the F-actin-severing function of villin
about
Disruption of the C. elegans Intestinal Brush Border by the Fungal Lectin CCL2 Phenocopies Dietary Lectin Toxicity in MammalsBy moonlighting in the nucleus, villin regulates epithelial plasticity.The F-actin modifier villin regulates insulin granule dynamics and exocytosis downstream of islet cell autoantigen 512.Actin cytoskeleton regulates Hippo signalingNOD-like receptors in intestinal homeostasis and epithelial tissue repair.Apical invasion of intestinal epithelial cells by Salmonella typhimurium requires villin to remodel the brush border actin cytoskeletonProtection by enteral glutamine is mediated by intestinal epithelial cell peroxisome proliferator-activated receptor-γ during intestinal ischemia/reperfusion.Oral administration of Bifidobacterium bifidum G9-1 alleviates rotavirus gastroenteritis through regulation of intestinal homeostasis by inducing mucosal protective factors.Milk glucosidase activity enables suckled pup starch digestion.Taste cell-expressed α-glucosidase enzymes contribute to gustatory responses to disaccharides.Metabolic Interactions in the Gastrointestinal Tract (GIT): Host, Commensal, Probiotics, and Bacteriophage Influences.Manipulation of intestinal epithelial cell function by the cell contact-dependent type III secretion systems of Vibrio parahaemolyticusGelsolin: the tail of a molecular gymnast.Plasticity of the brush border - the yin and yang of intestinal homeostasis.Cryopreserved mouse fetal liver stromal cells treated with mitomycin C are able to support the growth of human embryonic stem cells.Mechanisms of Cell Polarity-Controlled Epithelial Homeostasis and Immunity in the Intestine.Deciphering the Complex Signaling Systems That Regulate Intestinal Epithelial Cell Death Processes and Shedding.Restoration of cytoskeletal and membrane tethering defects but not defects in membrane trafficking in the intestinal brush border of mice lacking both myosin Ia and myosin VI.Trafficking Ion Transporters to the Apical Membrane of Polarized Intestinal Enterocytes.Development of arrayed colonic organoids for screening of secretagogues associated with enterotoxins.Phenolic compounds increase the transcription of mouse intestinal maltase-glucoamylase and sucrase-isomaltase.AMPK improves gut epithelial differentiation and barrier function via regulating Cdx2 expression.
P2860
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P2860
Enterocyte loss of polarity and gut wound healing rely upon the F-actin-severing function of villin
description
2013 nî lūn-bûn
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2013 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մարտին հրատարակված գիտական հոդված
@hy
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Enterocyte loss of polarity an ...... in-severing function of villin
@ast
Enterocyte loss of polarity an ...... in-severing function of villin
@en
type
label
Enterocyte loss of polarity an ...... in-severing function of villin
@ast
Enterocyte loss of polarity an ...... in-severing function of villin
@en
prefLabel
Enterocyte loss of polarity an ...... in-severing function of villin
@ast
Enterocyte loss of polarity an ...... in-severing function of villin
@en
P2093
P2860
P356
P1476
Enterocyte loss of polarity an ...... in-severing function of villin
@en
P2093
Anthony Simon
Buford L Nichols
Céline Revenu
Daniel Louvard
Danijela Vignjevic
Fatima El-Marjou
Florent Ubelmann
Jeanne Netter
Mathias Chamaillard
Roberto Quezada-Calvillo
P2860
P304
P356
10.1073/PNAS.1218446110
P407
P577
2013-03-21T00:00:00Z