c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer. - Wikidata - wikimedia - TriplyDB

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

http://www.wikidata.org/entity/Q30541608

about

A targetable GATA2-IGF2 axis confers aggressiveness in lethal prostate cancer Upregulation of miR-21 in cisplatin resistant ovarian cancer via JNK-1/c-Jun pathway Functional proteomic analysis of advanced serous ovarian cancer using reverse phase protein array: TGF-beta pathway signaling indicates response to primary chemotherapy Dickkopf-4 is frequently overexpressed in epithelial ovarian carcinoma and promotes tumor invasion A systems biology approach to the global analysis of transcription factors in colorectal cancer.JNK is a novel regulator of intercellular adhesion Overexpressed DNA polymerase iota regulated by JNK/c-Jun contributes to hypermutagenesis in bladder cancer.Silencing survivin splice variant 2B leads to antitumor activity in taxane--resistant ovarian cancer Inhibition of JNK Sensitizes Hypoxic Colon Cancer Cells to DNA-Damaging Agents.Mixed lineage kinase 3 is required for matrix metalloproteinase expression and invasion in ovarian cancer cells Targeting c-MYC in Platinum-Resistant Ovarian Cancer Sensitivity of ovarian cancer cells to acetaminophen reveals biological pathways that affect patient survival.Delivery strategies and potential targets for siRNA in major cancer types Inhibition of JNK3 promotes apoptosis induced by BH3 mimetic S1 in chemoresistant human ovarian cancer cells.Dab2 inhibits the cholesterol-dependent activation of JNK by TGF-β Roles of phosphorylated JNK in esophageal squamous cell carcinomas of Kazakh ethnic.Loss of tyrosine phosphatase-dependent inhibition promotes activation of tyrosine kinase c-Src in detached pancreatic cells.Inhibitory effect of black tea pigments, theaflavin‑3/3'-gallate against cisplatin-resistant ovarian cancer cells by inducing apoptosis and G1 cell cycle arrest.The prognostic significance of Jun transcription factors in ovarian cancer.Dickkopf-1 is frequently overexpressed in ovarian serous carcinoma and involved in tumor invasion.Drug Combinations to Enhance Therapeutic Efficacy and Edit Out Side Effects and Resistance to Inhibition of Drug Resistance Drug-Target Associations Inducing Protein Folding Epistructural Drug Design to Treat Cancer Metastasis and the Associated Drug Resistance Epistructural Re-engineering of Imatinib to Eliminate Adverse Side Effects

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c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

http://www.wikidata.org/entity/Q30541608

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@ast

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@en

type

label

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@ast

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@en

prefLabel

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@ast

c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@en

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1078-0432.CCR-09-1180

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Clinical Cancer Research

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c-Jun-NH2-kinase-1 inhibition leads to antitumor activity in ovarian cancer.

@en

P2093

Alpa M Nick

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Angela Sanguino

http://www.w3.org/2001/XMLSchema#string

Anil K Sood

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Bryan T J Hennessy

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Francois-Xavier Claret

http://www.w3.org/2001/XMLSchema#string

Gabriel Lopez-Berestein

http://www.w3.org/2001/XMLSchema#string

Hee-Dong Han

http://www.w3.org/2001/XMLSchema#string

Hye Sun Kim

http://www.w3.org/2001/XMLSchema#string

Juliana Maria Benito

http://www.w3.org/2001/XMLSchema#string

Lingegowda Mangala

http://www.w3.org/2001/XMLSchema#string

P2860

Signal transduction by the JNK group of MAP kinases

Mammalian MAP kinase signalling cascades

Signal transduction by the c-Jun N-terminal kinase (JNK)--from inflammation to development

Signalling for survival and death in neurones: the role of stress-activated kinases, JNK and p38

Mammalian mitogen-activated protein kinase signal transduction pathways activated by stress and inflammation

A small molecule-kinase interaction map for clinical kinase inhibitors

The stress-activated protein kinase subfamily of c-Jun kinases

Identification of an oncoprotein- and UV-responsive protein kinase that binds and potentiates the c-Jun activation domain

An anticancer C-Kit kinase inhibitor is reengineered to make it more active and less cardiotoxic.

Docetaxel versus paclitaxel for adjuvant treatment of ovarian cancer: case-control analysis of toxicity.

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184-194

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10.1158/1078-0432.CCR-09-1180

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1

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Ariel Fernández

Cristian Rodriguez-Aguayo

Arturo Chavez-Reyes

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2009-12-22T00:00:00Z

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20028751

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