Human cervicovaginal mucus contains an activity that hinders HIV-1 movement. - Wikidata - wikimedia - TriplyDB

Human cervicovaginal mucus contains an activity that hinders HIV-1 movement.

Human cervicovaginal mucus contains an activity that hinders HIV-1 movement.

http://www.wikidata.org/entity/Q30542085

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Sex hormones selectively impact the endocervical mucosal microenvironment: implications for HIV transmission Lactobacillus species as biomarkers and agents that can promote various aspects of vaginal health Preventing HIV infection: pre-exposure and postexposure prophylaxis Antibody responses to envelope glycoproteins in HIV-1 infection Increases in Endogenous or Exogenous Progestins Promote Virus-Target Cell Interactions within the Non-human Primate Female Reproductive Tract Tracking the luminal exposure and lymphatic drainage pathways of intravaginal and intrarectal inocula used in nonhuman primate models of HIV transmission HSV-2-driven increase in the expression of α4β7 correlates with increased susceptibility to vaginal SHIV(SF162P3) infection Modeling neutralization kinetics of HIV by broadly neutralizing monoclonal antibodies in genital secretions coating the cervicovaginal mucosa Antimicrobial and immune modulatory effects of lactic acid and short chain fatty acids produced by vaginal microbiota associated with eubiosis and bacterial vaginosis IgG in cervicovaginal mucus traps HSV and prevents vaginal herpes infections HIV-1 vaccines: challenges and new perspectives Transient antibody-mucin interactions produce a dynamic molecular shield against viral invasion Induction of Potent and Long-Lived Antibody and Cellular Immune Responses in the Genitorectal Mucosa Could be the Critical Determinant of HIV Vaccine Efficacy.HIV-1 specific IgA detected in vaginal secretions of HIV uninfected women participating in a microbicide trial in Southern Africa are primarily directed toward gp120 and gp140 specificities Nonneutralizing functional antibodies: a new "old" paradigm for HIV vaccines Vaccine-induced HIV-1 envelope gp120 constant region 1-specific antibodies expose a CD4-inducible epitope and block the interaction of HIV-1 gp140 with galactosylceramide.The effects of reproductive hormones on the physical properties of cervicovaginal fluid.Exogenous and endogenous hyaluronic acid reduces HIV infection of CD4(+) T cells.Vaginal pH and microbicidal lactic acid when lactobacilli dominate the microbiota Innate immunity in the vagina (Part II): Anti-HIV activity and antiviral content of human vaginal secretions Evolutionary distance of amino acid sequence orthologs across macaque subspecies: identifying candidate genes for SIV resistance in Chinese rhesus macaques Characterization of the Influence of Semen-Derived Enhancer of Virus Infection on the Interaction of HIV-1 with Female Reproductive Tract Tissues.Modeling of Virion Collisions in Cervicovaginal Mucus Reveals Limits on Agglutination as the Protective Mechanism of Secretory Immunoglobulin A.Randomized Cross-Sectional Study to Compare HIV-1 Specific Antibody and Cytokine Concentrations in Female Genital Secretions Obtained by Menstrual Cup and Cervicovaginal Lavage.HIV-Enhancing Factors Are Secreted by Reproductive Epithelia upon Inoculation with Bacterial Vaginosis-Associated Bacteria.HIV-1 vaginal transmission: cell-free or cell-associated virus?Diffusion of Immunoglobulin G in Shed Vaginal Epithelial Cells and in Cell-Free Regions of Human Cervicovaginal Mucus.Predicting first traversal times for virions and nanoparticles in mucus with slowed diffusion Antibody-Mediated Internalization of Infectious HIV-1 Virions Differs among Antibody Isotypes and Subclasses Enhanced Trapping of HIV-1 by Human Cervicovaginal Mucus Is Associated with Lactobacillus crispatus-Dominant Microbiota.New prospects for a preventive HIV-1 vaccine.Minimizing biases associated with tracking analysis of submicron particles in heterogeneous biological fluids Using Computational Modeling To Optimize the Design of Antibodies That Trap Viruses in Mucus The role of sex hormones in immune protection of the female reproductive tract.Broadly neutralizing antibody specificities detected in the genital tract of HIV-1 infected women Engineering and characterization of simplified vaginal and seminal fluid simulants.Mucosal immunology of HIV infection.Infectious virion capture by HIV-1 gp120-specific IgG from RV144 vaccinees Vaginal concentrations of lactic acid potently inactivate HIV Defining the interaction of HIV-1 with the mucosal barriers of the female reproductive tract

P2860

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P2860

Human cervicovaginal mucus contains an activity that hinders HIV-1 movement.

http://www.wikidata.org/entity/Q30542085

description

2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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Human cervicovaginal mucus contains an activity that hinders HIV-1 movement.

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Mucosal Immunology

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Human cervicovaginal mucus contains an activity that hinders HIV-1 movement

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C Hammond

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C J Gioia

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D J Stieh

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E J Spongberg

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S A Allen

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P2860

Methods of Digital Video Microscopy for Colloidal Studies

HIV-1 dynamics in vivo: virion clearance rate, infected cell life-span, and viral generation time

Rapid transport of large polymeric nanoparticles in fresh undiluted human mucus.

Improving defences at the portal of HIV entry: mucosal and innate immunity.

Electron tomography analysis of envelope glycoprotein trimers on HIV and simian immunodeficiency virus virions.

Differential incorporation of CD45, CD80 (B7-1), CD86 (B7-2), and major histocompatibility complex class I and II molecules into human immunodeficiency virus type 1 virions and microvesicles: implications for viral pathogenesis and immune regulation

Human beta-defensins suppress human immunodeficiency virus infection: potential role in mucosal protection.

Mucin gene expression in the human endocervix.

Mucins of the human endocervix.

A review of the physical and chemical properties of human semen and the formulation of a semen simulant.

P2888

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427-434

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10.1038/MI.2012.87

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2

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3732193

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