The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis - Wikidata - wikimedia - TriplyDB

The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis

The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis

http://www.wikidata.org/entity/Q30544122

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Neuropilin 1 (NRP1) hypomorphism combined with defective VEGF-A binding reveals novel roles for NRP1 in developmental and pathological angiogenesis The neuropilin 1 cytoplasmic domain is required for VEGF-A-dependent arteriogenesis NRP1 function and targeting in neurovascular development and eye disease Vascularisation of the central nervous system Egfl7 is differentially expressed in arteries and veins during retinal vascular development.Quantitative assessment of angiogenesis, perfused blood vessels and endothelial tip cells in the postnatal mouse brain.BMP-SMAD signalling output is highly regionalized in cardiovascular and lymphatic endothelial networks.Placental growth factor deficiency is associated with impaired cerebral vascular development in mice.Regulation of embryonic neurogenesis by germinal zone vasculature.FOXO1 couples metabolic activity and growth state in the vascular endothelium.Novel insights into the development and maintenance of the blood-brain barrier.Neuropilin regulation of angiogenesis.VEGF189 binds NRP1 and is sufficient for VEGF/NRP1-dependent neuronal patterning in the developing brain.Nogo-A regulates vascular network architecture in the postnatal brain.NRP1 Regulates CDC42 Activation to Promote Filopodia Formation in Endothelial Tip Cells.Podoplanin and CLEC-2 drive cerebrovascular patterning and integrity during development.Neuropilin 1 sequestration by neuropathogenic mutant glycyl-tRNA synthetase is permissive to vascular homeostasis Intracellular adenosine regulates epigenetic programming in endothelial cells to promote angiogenesis.MEF2 transcription factors are key regulators of sprouting angiogenesis.Fascin 1 is dispensable for developmental and tumour angiogenesis.Crypto-rhombomeres of the mouse medulla oblongata, defined by molecular and morphological features.The Mouse Hindbrain As a Model for Studying Embryonic Neurogenesis.MPDZ promotes DLL4-induced Notch signaling during angiogenesis.Elevated Expression of miR302-367 in Endothelial Cells Inhibits Developmental Angiogenesis via CDC42/CCND1 Mediated Signaling Pathways.

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The embryonic mouse hindbrain as a qualitative and quantitative model for studying the molecular and cellular mechanisms of angiogenesis

http://www.wikidata.org/entity/Q30544122

description

2013 nî lūn-bûn

@nan

2013 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի փետրվարին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

The embryonic mouse hindbrain ...... lar mechanisms of angiogenesis

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Nature Protocols

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The embryonic mouse hindbrain ...... lar mechanisms of angiogenesis

@en

P2093

Alessandro Fantin

http://www.w3.org/2001/XMLSchema#string

Alice Plein

http://www.w3.org/2001/XMLSchema#string

Charlotte H Maden

http://www.w3.org/2001/XMLSchema#string

Christiana Ruhrberg

http://www.w3.org/2001/XMLSchema#string

Joaquim M Vieira

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P2860

Molecular and cellular properties of PECAM-1 (endoCAM/CD31): a novel vascular cell-cell adhesion molecule

Selective requirements for NRP1 ligands during neurovascular patterning

Cre recombinase: the universal reagent for genome tailoring

Neuropilin-1 is required for endothelial tip cell guidance in the developing central nervous system

Impaired myocardial angiogenesis and ischemic cardiomyopathy in mice lacking the vascular endothelial growth factor isoforms VEGF164 and VEGF188

Neuropilin-1 conveys semaphorin and VEGF signaling during neural and cardiovascular development

A computational tool for quantitative analysis of vascular networks

Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

Abnormal blood vessel development and lethality in embryos lacking a single VEGF allele

VEGF guides angiogenic sprouting utilizing endothelial tip cell filopodia

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418-429

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10.1038/NPROT.2013.015

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2

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8

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3763679

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