Microtubules underlie dysfunction in duchenne muscular dystrophy. - Wikidata - wikimedia - TriplyDB

Microtubules underlie dysfunction in duchenne muscular dystrophy.

Microtubules underlie dysfunction in duchenne muscular dystrophy.

http://www.wikidata.org/entity/Q30557507

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Taurine: the appeal of a safe amino acid for skeletal muscle disorders Dysferlin at transverse tubules regulates Ca(2+) homeostasis in skeletal muscle Loss of CENP-F results in distinct microtubule-related defects without chromosomal abnormalities.Detyrosinated microtubules modulate mechanotransduction in heart and skeletal muscle Genetic evidence in the mouse solidifies the calcium hypothesis of myofiber death in muscular dystrophy Age-related alterations in the sarcolemmal environment are attenuated by lifelong caloric restriction and voluntary exercise Microtubules that form the stationary lattice of muscle fibers are dynamic and nucleated at Golgi elements.Altered nuclear dynamics in MDX myofibers.The Dystrophin Complex: Structure, Function, and Implications for Therapy X-ROS signalling is enhanced and graded by cyclic cardiomyocyte stretch.Human skeletal muscle xenograft as a new preclinical model for muscle disorders Eccentric exercise in aging and diseased skeletal muscle: good or bad?Detyrosinated microtubules buckle and bear load in contracting cardiomyocytes Src-dependent impairment of autophagy by oxidative stress in a mouse model of Duchenne muscular dystrophy NADPH oxidase-2 inhibition restores contractility and intracellular calcium handling and reduces arrhythmogenicity in dystrophic cardiomyopathy.Diapocynin, a dimer of the NADPH oxidase inhibitor apocynin, reduces ROS production and prevents force loss in eccentrically contracting dystrophic muscle Mechanisms of Myofascial Pain A new directionality tool for assessing microtubule pattern alterations.Dystrobrevin increases dystrophin's binding to the dystrophin-glycoprotein complex and provides protection during cardiac stress Real-time imaging of NADPH oxidase activity in living cells using a novel fluorescent protein reporter Loss of nNOS inhibits compensatory muscle hypertrophy and exacerbates inflammation and eccentric contraction-induced damage in mdx mice.Recovery of altered neuromuscular junction morphology and muscle function in mdx mice after injury Microtubule Actin Cross-linking Factor 1 regulates cardiomyocyte microtubule distribution and adaptation to hemodynamic overload.Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells ROS Production via P2Y1-PKC-NOX2 Is Triggered by Extracellular ATP after Electrical Stimulation of Skeletal Muscle Cells.NOX2 As a Target for Drug Development: Indications, Possible Complications, and Progress.Abnormalities in brain structure and biochemistry associated with mdx mice measured by in vivo MRI and high resolution localized (1)H MRS.Utrophin suppresses low frequency oscillations and coupled gating of mechanosensitive ion channels in dystrophic skeletal muscle A new therapeutic effect of simvastatin revealed by functional improvement in muscular dystrophy.Modeling Local X-ROS and Calcium Signaling in the Heart.Nanospan, an alternatively spliced isoform of sarcospan, localizes to the sarcoplasmic reticulum in skeletal muscle and is absent in limb girdle muscular dystrophy 2F.Absence of Dystrophin Disrupts Skeletal Muscle Signaling: Roles of Ca2+, Reactive Oxygen Species, and Nitric Oxide in the Development of Muscular Dystrophy.2015 William Allan Award.The cell biology of disease: cellular and molecular mechanisms underlying muscular dystrophy.Calcium Sparks in the Heart: Dynamics and Regulation.Inhibition of iPLA2 β and of stretch-activated channels by doxorubicin alters dystrophic muscle function.Reactive oxygen species generation is not different during isometric and lengthening contractions of mouse muscle.Differential Expression of NADPH Oxidases Depends on Skeletal Muscle Fiber Type in Rats.Genetic silencing of Nrf2 enhances X-ROS in dysferlin-deficient muscle.X-ROS signaling in the heart and skeletal muscle: stretch-dependent local ROS regulates [Ca²⁺]i.

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P2860

Microtubules underlie dysfunction in duchenne muscular dystrophy.

http://www.wikidata.org/entity/Q30557507

description

2012 nî lūn-bûn

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2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2012 թվականի օգոստոսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Microtubules underlie dysfunction in duchenne muscular dystrophy.

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Science Signaling

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Microtubules underlie dysfunction in duchenne muscular dystrophy

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P2093

Anup Mahurkar

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Benjamin L Prosser

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Christopher W Ward

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Eric P Hoffman

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Francesca Sbrana

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Fredrick Sachs

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Guoli Shi

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Mark J Mazaitis

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Ramzi J Khairallah

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Roberto Raiteri

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P2860

Differential expression analysis for sequence count data

The mechanosensitive ion channel Piezo1 is inhibited by the peptide GsMTx4

SPP1 genotype is a determinant of disease severity in Duchenne muscular dystrophy

Piezo1 and Piezo2 are essential components of distinct mechanically activated cation channels

Mechanosensitive channel properties and membrane mechanics in mouse dystrophic myotubes

Angiotensin II and NGF differentially influence microtubule proteins in PC12W cells: role of the AT2 receptor

Mice lacking Homer 1 exhibit a skeletal myopathy characterized by abnormal transient receptor potential channel activity

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Axial stretch of rat single ventricular cardiomyocytes causes an acute and transient increase in Ca2+ spark rate

Calcium homeostasis and cell death in Sol8 dystrophin-deficient cell line in culture.

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10.1126/SCISIGNAL.2002829

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236

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Carlos J Borroto

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