Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive decline - Wikidata - wikimedia - TriplyDB

Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive decline

Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive decline

http://www.wikidata.org/entity/Q30572684

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Overexpression of angiotensin-converting enzyme in myelomonocytic cells enhances the immune response Vascular contributions to cognitive impairment and dementia including Alzheimer's disease ACE overexpression in myelomonocytic cells: effect on a mouse model of Alzheimer's disease Therapeutic effects of glatiramer acetate and grafted CD115⁺ monocytes in a mouse model of Alzheimer's disease.New therapeutic approaches for Alzheimer's disease and cerebral amyloid angiopathy.Iron increases APP translation and amyloid-beta production in the retina.Angiotensin-converting enzyme overexpression in myelocytes enhances the immune response.Angiotensin-I converting enzyme (ACE): structure, biological roles, and molecular basis for chloride ion dependence.Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease.Angiotensin-converting enzyme enhances the oxidative response and bactericidal activity of neutrophils.Clearance of cerebral Aβ in Alzheimer's disease: reassessing the role of microglia and monocytes.Inhibition of ACE Retards Tau Hyperphosphorylation and Signs of Neuronal Degeneration in Aged Rats Subjected to Chronic Mild Stress.Common Impact of Chronic Kidney Disease and Brain Microhemorrhages on Cerebral Aβ Pathology in SHRSP.Renal tubular ACE-mediated tubular injury is the major contributor to microalbuminuria in early diabetic nephropathy.A novel role for osteopontin in macrophage-mediated amyloid-β clearance in Alzheimer's models.Within the Brain: The Renin Angiotensin System.Targeting Renin-Angiotensin System Against Alzheimer's Disease.Emerging Roles of Immune Cells in Postoperative Cognitive Dysfunction.

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P2860

Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive decline

http://www.wikidata.org/entity/Q30572684

description

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2014 թվականի մարտին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@ast

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@en

type

label

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@ast

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@en

prefLabel

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@ast

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

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Journal of Clinical Investigation

P1476

Angiotensin-converting enzyme ...... eimer's-like cognitive decline

@en

P2093

Brenda C Salumbides

http://www.w3.org/2001/XMLSchema#string

Dahabada H J Lopes

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Dieu-Trang Fuchs

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Ellen A Bernstein

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Jeff J-Y Yu

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Julia Sheyn

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Kandarp H Shah

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Keith L Black

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Lindsey Pelissier

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Maya Koronyo-Hamaoui

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P2860

A modern understanding of the traditional and nontraditional biological functions of angiotensin-converting enzyme

Catabolic attacks of membrane-bound angiotensin-converting enzyme on the N-terminal part of species-specific amyloid-beta peptides

Abeta42-to-Abeta40- and angiotensin-converting activities in different domains of angiotensin-converting enzyme

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Soluble oligomers of the amyloid beta-protein impair synaptic plasticity and behavior

Glatiramer acetate fights against Alzheimer's disease by inducing dendritic-like microglia expressing insulin-like growth factor 1

Large meta-analysis establishes the ACE insertion-deletion polymorphism as a marker of Alzheimer's disease

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Inflammation and Alzheimer's disease

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

P304

1000-1012

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10.1172/JCI66541

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3

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124

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Kenneth Bernstein

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2014-03-01T00:00:00Z

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24487585

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Alzheimer's disease

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3934162

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