Reversal of gene dysregulation in cultured cytotrophoblasts reveals possible causes of preeclampsia. - Wikidata - wikimedia - TriplyDB

Reversal of gene dysregulation in cultured cytotrophoblasts reveals possible causes of preeclampsia.

Reversal of gene dysregulation in cultured cytotrophoblasts reveals possible causes of preeclampsia.

http://www.wikidata.org/entity/Q30576991

about

Pre-eclampsia: its pathogenesis and pathophysiolgy Placental origins of adverse pregnancy outcomes: potential molecular targets: an Executive Workshop Summary of the Eunice Kennedy Shriver National Institute of Child Health and Human Development Preeclampsia and Extracellular Vesicles Wingless ligand 5a is a critical regulator of placental growth and survival Provocative ideas on human placental biology: A prerequisite for prevention and treatment of neonatal health challenges.Abnormal oxidative stress responses in fibroblasts from preeclampsia infants.Genome-wide hypermethylation coupled with promoter hypomethylation in the chorioamniotic membranes of early onset pre-eclampsia.Ouabain inhibits placental sFlt1 production by repressing HSP27-dependent HIF-1α pathway.Bioinformatics approach reveals evidence for impaired endometrial maturation before and during early pregnancy in women who developed preeclampsia.Expression pattern and function of Notch2 in different subtypes of first trimester cytotrophoblast Gestational tissue transcriptomics in term and preterm human pregnancies: a systematic review and meta-analysis.Why is placentation abnormal in preeclampsia?Function and control of human invasive trophoblast subtypes: Intrinsic vs. maternal control.A curated transcriptome dataset collection to investigate the development and differentiation of the human placenta and its associated pathologies.Human placental trophoblast invasion and differentiation: a particular focus on Wnt signaling.Notch1 controls development of the extravillous trophoblast lineage in the human placenta.Class 3 semaphorins in cardiovascular development.Up-regulation of CD81 inhibits cytotrophoblast invasion and mediates maternal endothelial cell dysfunction in preeclampsia The Human Placenta Project: placental structure, development, and function in real time.Preeclampsia: a link between trophoblast dysregulation and an antiangiogenic state.Severe pre-eclampsia is associated with alterations in cytotrophoblasts of the smooth chorion.Prediction of preeclampsia-bench to bedside.Models of vertical cytomegalovirus (CMV) transmission and pathogenesis.Emerging role for dysregulated decidualization in the genesis of preeclampsia.Transcriptional Dynamics of Cultured Human Villous Cytotrophoblasts.Hypermethylation of the HLA-G promoter is associated with preeclampsia.Sema3F (Semaphorin 3F) Selectively Drives an Extraembryonic Proangiogenic Program.Placental transcriptomes in the common aneuploidies reveal critical regions on the trisomic chromosomes and genome-wide effects Defective decidualization during and after severe preeclampsia reveals a possible maternal contribution to the etiology.Neuregulin-1-mediated ErbB2-ErbB3 signalling protects human trophoblasts against apoptosis to preserve differentiation.Suppression of Fgf2 by ETS2 repressor factor (ERF) is required for chorionic trophoblast differentiation.Negative regulators of Wnt signaling pathway SFRP1 and SFRP3 expression in preterm and term pathologic placentas.Molecular Mechanisms of Preeclampsia.

P2860

Q26746841-09B77EA5-7441-462E-90E0-05B165BEA58F Q26766128-69A8B84A-F3EC-43D3-979C-6A0217B5BE3C Q28079349-EF698237-5FE7-4CF7-90CC-D2EA44F9A086 Q28829967-EFE33026-40C8-475B-9E03-9E204BFAE0D1 Q30235366-65930A63-E3B3-466A-AE3A-D55FD60DBE6A Q33946049-340D8A1E-F875-4B62-AB36-2B32D56C7E0D Q34040496-0B75C522-D307-43F3-AF87-D8B3ECEE35C2 Q34365552-FF0A70E0-78C8-4BD3-91F8-33AA77DF0FD1 Q34910971-C725A2A3-9485-4F01-8326-C3F87F44B9E2 Q35201773-242DD479-9B39-4C79-ACE7-2D3F04E83C7A Q35686547-944AFA82-BEC4-4B33-9126-E9E301A8F469 Q36119044-723CB3F1-8959-428E-A9BA-B43D340E40E3 Q36857915-5EA00764-211B-44F4-BAB3-529A17579CC1 Q36980531-2CAC7204-89B5-4E52-9DE0-AAB95932BD73 Q37199588-C5ED8E74-D4E5-45FC-9877-968C6003F83E Q37473736-3C9D5B01-4D78-4816-94C1-2113338E5205 Q37509074-588514F9-3B8D-4FDD-A682-741A79874A31 Q37682271-23DA6A7C-A9F5-4A6B-9B97-11A5AEC8F3AF Q37722793-6FF6A071-91AB-4DE5-AA9D-A8B92D728B22 Q37723245-693AFEA3-767B-4730-BBCF-550E87F487E2 Q37731214-37DE7A96-142B-4653-B905-6EC8F06CCB9B Q38252740-66672EF3-186D-44E3-9CC5-D215C64309D1 Q38257810-93D0D1FC-6A49-483F-B182-B8FADB9F96F2 Q38685664-77F642D8-C9F9-46BF-9EF6-6365143B81E5 Q38742215-8CB1FEA2-88FA-4887-A288-2199E62D45DD Q38859599-71B39073-FB47-4DF4-AFCE-3A3E3F42C3D5 Q41491009-0668A37B-8578-4DE3-9D52-E6E2074F94E9 Q41866713-7A4CBA56-E92A-4194-BC14-807880B9EBDB Q42263674-B334487B-89DE-4B10-8084-79FBEE94E14D Q42484071-0695DBFD-269E-4FBB-AAD6-8B55E1619D50 Q47339731-55062C70-D31C-41F4-85F3-C492A96A919F Q48356598-81176478-8218-45A1-8938-7E747F4B42A4 Q54901493-FF865F2D-9693-4435-94A0-3389A525122C

P2860

Reversal of gene dysregulation in cultured cytotrophoblasts reveals possible causes of preeclampsia.

http://www.wikidata.org/entity/Q30576991

description

2013 nî lūn-bûn

@nan

2013 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2013年の論文

@ja

2013年論文

@yue

2013年論文

@zh-hant

2013年論文

@zh-hk

2013年論文

@zh-mo

2013年論文

@zh-tw

2013年论文

@wuu

name

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@ast

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@en

type

label

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@ast

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@en

prefLabel

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@ast

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@en

P1433

Q30576991-66DFF52B-E392-44A7-8669-CA994692CB5D

P1476

Q30576991-2A8FA0C1-18B7-4CB5-996A-A7F1B8ED8702

P2093

Q30576991-19E17299-7158-41AA-B875-758E05471F2D

Q30576991-356251D4-0C35-49D1-84AE-322CE76415B6

Q30576991-363EAF6B-59BC-41F3-91A9-DACBA6855051

Q30576991-796290D4-4C22-4D58-9D42-48AA0EC0A4D8

Q30576991-8C294836-8E52-41C6-82BE-E032D90B8F9E

Q30576991-9AC70D3C-EB58-4BA3-97BD-FABAC81B03D2

Q30576991-9CE136E5-AEA7-4BE0-BB6B-872ABAED36A4

Q30576991-A8B6FA53-D653-4A68-A9AA-36053DE10FD4

Q30576991-ADBB1041-2421-488E-B1BE-54B16DCF3D4A

Q30576991-AFAE90F0-C041-4AF1-A826-C0470D89E3EE

P2860

Q30576991-009871B8-3EAA-4844-8B35-07C71757ADFB

Q30576991-084B6590-B9BF-4642-A8B3-6247C82CAA48

Q30576991-0AE55822-C354-4AA6-8A68-81153D229074

Q30576991-0CC8D16B-8A4F-45A3-8B50-FA189FDADD8C

Q30576991-0EEAFCE1-C553-45D7-BA3D-92AADE254C61

Q30576991-2330A4C9-A192-43CC-821F-5F0862D0687F

Q30576991-248F7E96-0082-4ED4-A3EA-6666D22B88B8

Q30576991-2CE44160-D15E-4DC6-B4B8-8D70C9FFBB68

Q30576991-3178807E-FE9D-4A6B-84CC-4200CAC6BD7B

Q30576991-340FF703-6AF6-455A-B9A3-1E63FA961099

P304

Q30576991-DFE1FA32-E502-49F8-8E20-BD04CC26E04C

P31

Q30576991-4B98F4A3-0F81-432C-88EA-F22EDDD087B0

P356

Q30576991-3F5D79D5-EE23-41A7-9783-2E5B580A1CEB

P407

Q30576991-E506CC34-7B5A-4A01-9677-A2BB763FB73C

P433

Q30576991-00FBC7F6-0960-4895-A27B-EEE02CD8BD55

P478

Q30576991-23C5F2F7-73B0-43E6-AD58-EB21C14C1C18

P577

Q30576991-DEF93087-F65C-43F8-AFB1-D64C21A919A1

P698

Q30576991-3685FC24-4BDC-46E7-B894-013F4D4F9953

P921

Q30576991-E8292C1C-089A-4763-907D-D5B7BF0D65C6

P932

Q30576991-BDD764FC-1DEB-4970-91AA-542247394DEE

P356

P1433

Journal of Clinical Investigation

P1476

Reversal of gene dysregulation ...... ssible causes of preeclampsia.

@en

P2093

Fei Wang

http://www.w3.org/2001/XMLSchema#string

Katherine Bianco

http://www.w3.org/2001/XMLSchema#string

Masakazu Nishida

http://www.w3.org/2001/XMLSchema#string

Matthew J Gormley

http://www.w3.org/2001/XMLSchema#string

Michael T McMaster

http://www.w3.org/2001/XMLSchema#string

Mirhan Kapidzic

http://www.w3.org/2001/XMLSchema#string

Nathan M Hunkapiller

http://www.w3.org/2001/XMLSchema#string

Penelope M Drake

http://www.w3.org/2001/XMLSchema#string

Susan J Fisher

http://www.w3.org/2001/XMLSchema#string

Victoria Feng

http://www.w3.org/2001/XMLSchema#string

P2860

The HELLP syndrome: clinical issues and management. A Review

Regulated expression of ADAMTS-12 in human trophoblastic cells: a role for ADAMTS-12 in epithelial cell invasion?

Further evidence that the inhibition of glycogen synthase kinase-3beta by IGF-1 is mediated by PDK1/PKB-induced phosphorylation of Ser-9 and not by dephosphorylation of Tyr-216

HOP/NECC1, a novel regulator of mouse trophoblast differentiation

Human cytotrophoblasts adopt a vascular phenotype as they differentiate. A strategy for successful endovascular invasion?

Hopx and Hdac2 interact to modulate Gata4 acetylation and embryonic cardiac myocyte proliferation

Pregnancy-specific glycoprotein 1 induces endothelial tubulogenesis through interaction with cell surface proteoglycans

GSK-3: tricks of the trade for a multi-tasking kinase

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

P304

2862-2872

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1172/JCI66966

http://www.w3.org/2001/XMLSchema#string

P407

P433

7

http://www.w3.org/2001/XMLSchema#string

P478

123

http://www.w3.org/2001/XMLSchema#string

P577

2013-06-24T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

23934129

http://www.w3.org/2001/XMLSchema#string

P921

P932

3999620

http://www.w3.org/2001/XMLSchema#string