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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

http://www.wikidata.org/entity/Q30579142

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Enzyme Biosensors for Biomedical Applications: Strategies for Safeguarding Analytical Performances in Biological Fluids Integrating Inflammasome Signaling in Sexually Transmitted Infections Inflammasome control of viral infection NLRP3 Inflammasome in Neurological Diseases, from Functions to Therapies Alarmins and central nervous system inflammation in HIV-associated neurological disorders.MicroRNAs upregulated during HIV infection target peroxisome biogenesis factors: Implications for virus biology, disease mechanisms and neuropathology Role of the immune system in HIV-associated neuroinflammation and neurocognitive implications.Sutherlandia frutescens may exacerbate HIV-associated neuroinflammation Methamphetamine abuse affects gene expression in brain-derived microglia of SIV-infected macaques to enhance inflammation and promote virus targets Neuroinflammation-Induced Interactions between Protease-Activated Receptor 1 and Proprotein Convertases in HIV-Associated Neurocognitive Disorder.Physical versus psychological social stress in male rats reveals distinct cardiovascular, inflammatory and behavioral consequences HIV-1 gp120 envelope glycoprotein determinants for cytokine burst in human monocytes.Strong upregulation of inflammatory genes accompanies photoreceptor demise in canine models of retinal degeneration.HIV alters neuronal mitochondrial fission/fusion in the brain during HIV-associated neurocognitive disorders.Microglial M1/M2 polarization and metabolic states.HIV-1 Tat Primes and Activates Microglial NLRP3 Inflammasome-Mediated Neuroinflammation.Osteopontin Impacts West Nile virus Pathogenesis and Resistance by Regulating Inflammasome Components and Cell Death in the Central Nervous System at Early Time Points.Chronic low-level expression of HIV-1 Tat promotes a neurodegenerative phenotype with aging.Immune activation in the central nervous system throughout the course of HIV infection.Fate of microglia during HIV-1 infection: From activation to senescence?What do we know about the inflammasome in humans?Effect of naringin on gp120-induced injury mediated by P2X7 receptors in rat primary cultured microglia.Methamphetamine potentiates HIV-1gp120-induced microglial neurotoxic activity by enhancing microglial outward K+ current.Modeling brain lentiviral infections during antiretroviral therapy in AIDS.Immediate initiation of cART is associated with lower levels of cerebrospinal fluid YKL-40, a marker of microglial activation, in HIV-1 infection.HIV-1 Viral Protein R Activates NLRP3 Inflammasome in Microglia: implications for HIV-1 Associated Neuroinflammation.HIV-1 protease cleaves the serine-threonine kinases RIPK1 and RIPK2.Role of microglial amylin receptors in mediating beta amyloid (Aβ)-induced inflammation.Reduced antiretroviral drug efficacy and concentration in HIV-infected microglia contributes to viral persistence in brain.Neurologic disease in feline immunodeficiency virus infection: disease mechanisms and therapeutic interventions for NeuroAIDS.Contrasting effect of the latency-reversing agents bryostatin-1 and JQ1 on astrocyte-mediated neuroinflammation and brain neutrophil invasion.In vitro modeling of HIV proviral activity in microglia.Inflammasomes in neurological diseases: emerging pathogenic and therapeutic concepts.Neurocognitive performances of visuospatial attention and the correlations with metabolic and inflammatory biomarkers in adults with obesity.Inflammasome in drug abuse.Suppressed oligodendrocyte steroidogenesis in multiple sclerosis: Implications for regulation of neuroinflammation.Neurodegeneration and NLRP3 inflammasome expression in the anterior thalamus of SOD1(G93A) ALS mice.Modulation of mitochondrial bioenergetics as a therapeutic strategy in Alzheimer's disease.HIV replication is associated to inflammasomes activation, IL-1β, IL-18 and caspase-1 expression in GALT and peripheral blood.

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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

http://www.wikidata.org/entity/Q30579142

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2014 nî lūn-bûn

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2014 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2014 թվականի մայիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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type

label

Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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prefLabel

Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDS

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Brienne A McKenzie

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Ferdinand Maingat

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John G Walsh

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William G Branton

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P2860

NLRP3 is activated in Alzheimer's disease and contributes to pathology in APP/PS1 mice

SAMHD1 is the dendritic- and myeloid-cell-specific HIV-1 restriction factor counteracted by Vpx

Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization

Neisseria gonorrhoeae activates the proteinase cathepsin B to mediate the signaling activities of the NLRP3 and ASC-containing inflammasome

Innate immunity in the central nervous system

Regulation of inflammasome signaling

Common transcriptional signatures in brain tissue from patients with HIV-associated neurocognitive disorders, Alzheimer's disease, and Multiple Sclerosis

IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection

HIV-1 restriction factor SAMHD1 is a deoxynucleoside triphosphate triphosphohydrolase

Gout-associated uric acid crystals activate the NALP3 inflammasome

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11

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Christopher Power

David I Broadhurst

Stacey N Reinke

Manmeet K Mamik

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2014-05-13T00:00:00Z

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