Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease. - Wikidata - wikimedia - TriplyDB

Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q30581719

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In vivo imaging reveals sigmoidal growth kinetic of β-amyloid plaques.Role of presenilin 1 in structural plasticity of cortical dendritic spines in vivo Neural stem/progenitor cells in Alzheimer's disease Systematic Review of the Neurobiological Relevance of Chemokines to Psychiatric Disorders Implications of glial nitric oxide in neurodegenerative diseases Innate immunity in the central nervous system Neuroinflammation in Alzheimer's disease: chemokines produced by astrocytes and chemokine receptors Innate immunity in Alzheimer's disease: a complex affair Chemokine receptor CXCR2: physiology regulator and neuroinflammation controller?HIV Associated Neurocognitive Disorders Fibrillar amyloid plaque formation precedes microglial activation Multiple events lead to dendritic spine loss in triple transgenic Alzheimer's disease mice Microglia development and function Accumulating microglia phagocytose injured neurons in hippocampal slice cultures: involvement of p38 MAP kinase IVIg protects the 3xTg-AD mouse model of Alzheimer’s disease from memory deficit and Aβ pathology Innate Immunity Fights Alzheimer's Disease The ischemic environment drives microglia and macrophage function Neuroinflammation in Alzheimer's disease Statins promote the degradation of extracellular amyloid {beta}-peptide by microglia via stimulation of exosome-associated insulin-degrading enzyme (IDE) secretion Glial modulators as potential treatments of psychostimulant abuse Ten years of Nature Reviews Neuroscience: insights from the highly cited Cortical microhemorrhages cause local inflammation but do not trigger widespread dendrite degeneration Analysis of the Role of CX3CL1 (Fractalkine) and Its Receptor CX3CR1 in Traumatic Brain and Spinal Cord Injury: Insight into Recent Advances in Actions of Neurochemokine Agents.Fractalkine Signaling Regulates Macrophage Recruitment into the Cochlea and Promotes the Survival of Spiral Ganglion Neurons after Selective Hair Cell Lesion Microglia as a critical player in both developmental and late-life CNS pathologies.CX3CR1 deficiency leads to impairment of hippocampal cognitive function and synaptic plasticity.A neurodegenerative disease mutation that accelerates the clearance of apoptotic cells Cerebrospinal fluid biomarkers for Parkinson disease diagnosis and progression Microfluidic chemotaxis platform for differentiating the roles of soluble and bound amyloid-β on microglial accumulation.In vivo dynamics of innate immune sentinels in the CNS.A multidisciplinary approach unravels early and persistent effects of X-ray exposure at the onset of prenatal neurogenesis Eliminating microglia in Alzheimer's mice prevents neuronal loss without modulating amyloid-β pathology.Contributions of microglia to structural synaptic plasticity Dual induction of TREM2 and tolerance-related transcript, Tmem176b, in amyloid transgenic mice: implications for vaccine-based therapies for Alzheimer's disease.Modulating neurotoxicity through CX3CL1/CX3CR1 signaling.Alterations to the frequency and function of peripheral blood monocytes and associations with chronic disease in the advanced-age, frail elderly.Longitudinal in vivo two-photon fluorescence imaging.In vivo imaging of the neurovascular unit in CNS disease.Ontogeny and functions of central nervous system macrophages.Regulation of tau pathology by the microglial fractalkine receptor.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

http://www.wikidata.org/entity/Q30581719

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Nature Neuroscience

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Microglial Cx3cr1 knockout prevents neuron loss in a mouse model of Alzheimer's disease.

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Christian K E Jung

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Frank M LaFerla

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Gerda Mitteregger

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Hans Kretzschmar

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Jochen Herms

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Martin Fuhrmann

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Richard M Page

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Steffen Burgold

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Tobias Bittner

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P2860

Resting microglial cells are highly dynamic surveillants of brain parenchyma in vivo

Control of microglial neurotoxicity by the fractalkine receptor

Role for neuronally derived fractalkine in mediating interactions between neurons and CX3CR1-expressing microglia

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

ATP mediates rapid microglial response to local brain injury in vivo

Analysis of fractalkine receptor CX(3)CR1 function by targeted deletion and green fluorescent protein reporter gene insertion

Formation and maintenance of Alzheimer's disease beta-amyloid plaques in the absence of microglia

Inflammation in Alzheimer disease: driving force, bystander or beneficial response?

Dynamics of the microglial/amyloid interaction indicate a role in plaque maintenance

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411-413

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10.1038/NN.2511

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4

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Christian Haass

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2010-03-21T00:00:00Z

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42372604

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20305648

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Alzheimer's disease

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4072212

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