Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose). - Wikidata - wikimedia - TriplyDB

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

http://www.wikidata.org/entity/Q30663823

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Interplay between Ubiquitin, SUMO, and Poly(ADP-Ribose) in the Cellular Response to Genotoxic Stress Prion-like domains as epigenetic regulators, scaffolds for subcellular organization, and drivers of neurodegenerative disease Roles of RNA-Binding Proteins in DNA Damage Response Readers of poly(ADP-ribose): designed to be fit for purpose Femtosecond near-infrared laser microirradiation reveals a crucial role for PARP signaling on factor assemblies at DNA damage sites.ADP-ribose-derived nuclear ATP synthesis by NUDIX5 is required for chromatin remodeling Are aberrant phase transitions a driver of cellular aging?Analyzing structure-function relationships of artificial and cancer-associated PARP1 variants by reconstituting TALEN-generated HeLa PARP1 knock-out cells.The DBHS proteins SFPQ, NONO and PSPC1: a multipurpose molecular scaffold Poly(ADP-ribosyl)ation-dependent Transient Chromatin Decondensation and Histone Displacement following Laser Microirradiation Staphylococcal Bap Proteins Build Amyloid Scaffold Biofilm Matrices in Response to Environmental Signals.Architectural plasticity of human BRCA2-RAD51 complexes in DNA break repair.The role of FLI-1-EWS, a fusion gene reciprocal to EWS-FLI-1, in Ewing sarcoma.The Structure and Dynamics of Higher-Order Assemblies: Amyloids, Signalosomes, and Granules.The LC Domain of hnRNPA2 Adopts Similar Conformations in Hydrogel Polymers, Liquid-like Droplets, and Nuclei.Higher-order oligomerization promotes localization of SPOP to liquid nuclear speckles.ADPriboDB: The database of ADP-ribosylated proteins.Efficacy of ATR inhibitors as single agents in Ewing sarcoma.PARPs and ADP-ribosylation: recent advances linking molecular functions to biological outcomes.Spatiotemporal regulation of posttranslational modifications in the DNA damage response.p53 induces formation of NEAT1 lncRNA-containing paraspeckles that modulate replication stress response and chemosensitivity.Insight into the machinery that oils chromatin dynamics.New directions in poly(ADP-ribose) polymerase biology.A glass menagerie of low complexity sequences.Droplet organelles?Cross-β Polymerization of Low Complexity Sequence Domains.Express or repress? The transcriptional dilemma of damaged chromatin.The code and beyond: transcription regulation by the RNA polymerase II carboxy-terminal domain.Phase Separation of C9orf72 Dipeptide Repeats Perturbs Stress Granule Dynamics.At the Interface of Three Nucleic Acids: The Role of RNA-Binding Proteins and Poly(ADP-ribose) in DNA Repair.Understanding co-polymerization in amyloid formation by direct observation of mixed oligomers.A lncRNA to repair DNA.Phosphorylation of the FUS low-complexity domain disrupts phase separation, aggregation, and toxicity.Coordinating cell cycle-regulated histone gene expression through assembly and function of the Histone Locus Body.A histone-mimicking interdomain linker in a multidomain protein modulates multivalent histone binding.Nucleic acid binding proteins affect the subcellular distribution of phosphorothioate antisense oligonucleotides.The C-terminal domain of p53 orchestrates the interplay between non-covalent and covalent poly(ADP-ribosyl)ation of p53 by PARP1.Phase behaviour of disordered proteins underlying low density and high permeability of liquid organelles.Specificity of reversible ADP-ribosylation and regulation of cellular processes.Impaired DNA damage response signaling by FUS-NLS mutations leads to neurodegeneration and FUS aggregate formation.

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Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

http://www.wikidata.org/entity/Q30663823

description

2015 nî lūn-bûn

@nan

2015 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2015年の論文

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2015年論文

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2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@ast

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@en

type

label

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@ast

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@en

prefLabel

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@ast

Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@en

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Nature Communications

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Liquid demixing of intrinsically disordered proteins is seeded by poly(ADP-ribose).

@en

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Irina Pozdnyakova

http://www.w3.org/2001/XMLSchema#string

Maj-Britt Druedahl Rask

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Merete Grøfte

http://www.w3.org/2001/XMLSchema#string

Stefania Pellegrino

http://www.w3.org/2001/XMLSchema#string

Stephanie Jungmichel

http://www.w3.org/2001/XMLSchema#string

Werner Streicher

http://www.w3.org/2001/XMLSchema#string

P2860

TRIP12 and UBR5 suppress spreading of chromatin ubiquitylation at damaged chromosomes

ATM-dependent chromatin changes silence transcription in cis to DNA double-strand breaks

Mutations in prion-like domains in hnRNPA2B1 and hnRNPA1 cause multisystem proteinopathy and ALS

Defining the RGG/RG motif

Self-propagation of pathogenic protein aggregates in neurodegenerative diseases

Getting RNA and protein in phase

A role for Q/N-rich aggregation-prone regions in P-body localization.

Spatial organization of the mammalian genome surveillance machinery in response to DNA strand breaks

Dual specificity kinase DYRK3 couples stress granule condensation/dissolution to mTORC1 signaling

Systematic identification of genomic markers of drug sensitivity in cancer cells

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8088

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scholarly article

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10.1038/NCOMMS9088

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6

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Michael Lund Nielsen

Matthias Altmeyer

Federico Teloni

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2015-08-19T00:00:00Z

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281172980

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1026790866

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26286827

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4560800

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