Identification of B6SJL mSOD1(G93A) mouse subgroups with different disease progression rates. - Wikidata - wikimedia - TriplyDB

Identification of B6SJL mSOD1(G93A) mouse subgroups with different disease progression rates.

Identification of B6SJL mSOD1(G93A) mouse subgroups with different disease progression rates.

http://www.wikidata.org/entity/Q30668718

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Outdoor Ambient Air Pollution and Neurodegenerative Diseases: the Neuroinflammation Hypothesis.Locomotor analysis identifies early compensatory changes during disease progression and subgroup classification in a mouse model of amyotrophic lateral sclerosis.

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Identification of B6SJL mSOD1(G93A) mouse subgroups with different disease progression rates.

http://www.wikidata.org/entity/Q30668718

description

2015 nî lūn-bûn

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2015 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2015 թվականի մայիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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The Journal of Comparative Neurology

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Identification of B6SJL mSOD1( ...... ent disease progression rates.

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Kathryn J Jones

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Kathryn P Mcmillan

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Melissa M Haulcomb

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Nichole A Mesnard-Hoaglin

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Rena M Meadows

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Richard J Batka

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Todd J Brown

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Virginia M Sanders

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Whitney M Miller

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Analyzing real-time PCR data by the comparative C(T) method

Control of microglial neurotoxicity by the fractalkine receptor

Age and founder effect of SOD1 A4V mutation causing ALS

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

El Escorial revisited: revised criteria for the diagnosis of amyotrophic lateral sclerosis

Use of laser microdissection in the investigation of facial motoneuron and neuropil molecular phenotypes after peripheral axotomy

Axotomy-induced target disconnection promotes an additional death mechanism involved in motoneuron degeneration in amyotrophic lateral sclerosis transgenic mice.

Differential gene expression in the axotomized facial motor nucleus of presymptomatic SOD1 mice

SOD1(G93A) transgenic mouse CD4(+) T cells mediate neuroprotection after facial nerve axotomy when removed from a suppressive peripheral microenvironment.

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2752-2768

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10.1002/CNE.23814

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523

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26010802

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