Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injury
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Azithromycin drives alternative macrophage activation and improves recovery and tissue sparing in contusion spinal cord injuryInnate immune responses in the ageing lung.Protumoral TSP50 Regulates Macrophage Activities and Polarization via Production of TNF-α and IL-1β, and Activation of the NF-κB Signaling Pathway.Strong upregulation of inflammatory genes accompanies photoreceptor demise in canine models of retinal degeneration.Predictive screening of M1 and M2 macrophages reveals the immunomodulatory effectiveness of post spinal cord injury azithromycin treatmentNeuroinflammation as Fuel for Axonal Regeneration in the Injured Vertebrate Central Nervous System.Microglia in central nervous system repair after injury.Renal Aging: Causes and Consequences.Neuroinflammation: the devil is in the details.Advanced Age Alters Monocyte and Macrophage Responses.Macrophage polarization: a key event in the secondary phase of acute spinal cord injury.TRIM32 affects the recovery of motor function following spinal cord injury through regulating proliferation of glia.Age exacerbates microglial activation, oxidative stress, inflammatory and NOX2 gene expression, and delays functional recovery in a middle-aged rodent model of spinal cord injury.Age increases reactive oxygen species production in macrophages and potentiates oxidative damage after spinal cord injury.Pioglitazone treatment following spinal cord injury maintains acute mitochondrial integrity and increases chronic tissue sparing and functional recovery.Immunobiology of spinal cord injuries and potential therapeutic approaches.Compression Decreases Anatomical and Functional Recovery and Alters Inflammation after Contusive Spinal Cord Injury.Neuregulin-1 positively modulates glial response and improves neurological recovery following traumatic spinal cord injury.A cross-sectional comparison of ethanol-related cytokine expression in the hippocampus of young and aged Fischer 344 rats.Fibroblast fate regulation by time dependent TGF-β1 and IL-10 stimulation in biomimetic 3D matrices.Cell cycle and complement inhibitors may be specific for treatment of spinal cord injury in aged and young mice: Transcriptomic analyses.
P2860
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P2860
Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injury
description
2015 nî lūn-bûn
@nan
2015 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@ast
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@en
type
label
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@ast
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@en
prefLabel
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@ast
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@en
P2093
P2860
P1476
Age decreases macrophage IL-10 ...... e repair in spinal cord injury
@en
P2093
John C Gensel
Kaitlyn J Braun
William M Bailey
P2860
P356
10.1016/J.EXPNEUROL.2015.08.001
P407
P577
2015-08-08T00:00:00Z