Focal adhesion features during myofibroblastic differentiation are controlled by intracellular and extracellular factors.
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Keratocyte phenotype mediates proteoglycan structure: a role for fibroblasts in corneal fibrosisMyofibroblast contraction activates latent TGF-beta1 from the extracellular matrixFunctional and phenotypical comparison of myofibroblasts derived from biopsies and bronchoalveolar lavage in mild asthma and sclerodermaThe myofibroblast in wound healing and fibrosis: answered and unanswered questionsActin cytoskeleton in myofibroblast differentiation: ultrastructure defining form and driving functionFibroblasts and myofibroblasts in wound healingHeading off with the herd: how cancer cells might maneuver supernumerary centrosomes for directional migrationThe wound healing, chronic fibrosis, and cancer progression triadS-nitrosoglutathione accelerates recovery from 5-fluorouracil-induced oral mucositisCancer cell invasion is enhanced by applied mechanical stimulationWhole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transitionAdhesion and proliferation of human periodontal ligament cells on poly(2-methoxyethyl acrylate)Alpha-smooth muscle actin is crucial for focal adhesion maturation in myofibroblasts.Focal adhesion size controls tension-dependent recruitment of alpha-smooth muscle actin to stress fibersMatrix stiffness reverses the effect of actomyosin tension on cell proliferation.PPFIA1 drives active α5β1 integrin recycling and controls fibronectin fibrillogenesis and vascular morphogenesis.Rac inhibition reverses the phenotype of fibrotic fibroblastsPDGF-Ralpha gene expression predicts proliferation, but PDGF-A suppresses transdifferentiation of neonatal mouse lung myofibroblasts.Nanoscale topography-induced modulation of fundamental cell behaviors of rabbit corneal keratocytes, fibroblasts, and myofibroblasts.Autocrine production of TGF-beta1 promotes myofibroblastic differentiation of neonatal lung mesenchymal stem cells.Cancer associated fibroblasts (CAFs) in tumor microenvironment.Cells lacking β-actin are genetically reprogrammed and maintain conditional migratory capacity.Modulation of fibroblast morphology and adhesion during collagen matrix remodeling.β2-adrenoceptor activation modulates skin wound healing processes to reduce scarringMechanosignaling through YAP and TAZ drives fibroblast activation and fibrosisTyrosine phosphatase PTPalpha regulates focal adhesion remodeling through Rac1 activationMyocardin-related transcription factors A and B are key regulators of TGF-β1-induced fibroblast to myofibroblast differentiation.Delayed stress fiber formation mediates pulmonary myofibroblast differentiation in response to TGF-β.Fibronectin extra domain-A promotes hepatic stellate cell motility but not differentiation into myofibroblasts.The role of osteopontin and osteopontin aptamer (OPN-R3) in fibroblast activity.SPOCK1 Is a Novel Transforming Growth Factor-β-Induced Myoepithelial Marker That Enhances Invasion and Correlates with Poor Prognosis in Breast Cancer.Improved throughput traction microscopy reveals pivotal role for matrix stiffness in fibroblast contractility and TGF-β responsiveness.Dermal Fibroblasts from the Red Duroc Pig Have an Inherently Fibrogenic Phenotype: An In Vitro Model of Fibroproliferative ScarringA Chemomechanical Model of Matrix and Nuclear Rigidity Regulation of Focal Adhesion Size.Matrix-driven Myosin II Mediates the Pro-fibrotic Fibroblast PhenotypeStretch-activated force shedding, force recovery, and cytoskeletal remodeling in contractile fibroblasts.Peptide-mediated inhibition of mitogen-activated protein kinase-activated protein kinase-2 ameliorates bleomycin-induced pulmonary fibrosisProtease-activated receptor 1 inhibition by SCH79797 attenuates left ventricular remodeling and profibrotic activities of cardiac fibroblasts.Cellular fibronectin expression in human trabecular meshwork and induction by transforming growth factor-β2Role of IGF-1 pathway in lung fibroblast activation.
P2860
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P2860
Focal adhesion features during myofibroblastic differentiation are controlled by intracellular and extracellular factors.
description
2001 nî lūn-bûn
@nan
2001 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Focal adhesion features during ...... lar and extracellular factors.
@ast
Focal adhesion features during ...... lar and extracellular factors.
@en
type
label
Focal adhesion features during ...... lar and extracellular factors.
@ast
Focal adhesion features during ...... lar and extracellular factors.
@en
prefLabel
Focal adhesion features during ...... lar and extracellular factors.
@ast
Focal adhesion features during ...... lar and extracellular factors.
@en
P2093
P1476
Focal adhesion features during ...... lar and extracellular factors.
@en
P2093
P304
P407
P577
2001-09-01T00:00:00Z