Different microcirculatory and interstitial matrix patterns in idiopathic dilated cardiomyopathy and Chagas' disease: a three dimensional confocal microscopy study - Wikidata - wikimedia - TriplyDB

Different microcirculatory and interstitial matrix patterns in idiopathic dilated cardiomyopathy and Chagas' disease: a three dimensional confocal microscopy study

Different microcirculatory and interstitial matrix patterns in idiopathic dilated cardiomyopathy and Chagas' disease: a three dimensional confocal microscopy study

http://www.wikidata.org/entity/Q30755828

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Pentoxifylline reverses chronic experimental Chagasic cardiomyopathy in association with repositioning of abnormal CD8+ T-cell response Coronary microvascular disease in chronic Chagas cardiomyopathy including an overview on history, pathology, and other proposed pathogenic mechanisms.Relationship between fibrosis and ventricular arrhythmias in Chagas heart disease without ventricular dysfunction.Tumor necrosis factor is a therapeutic target for immunological unbalance and cardiac abnormalities in chronic experimental Chagas' heart disease.Severity of chronic experimental Chagas' heart disease parallels tumour necrosis factor and nitric oxide levels in the serum: models of mild and severe disease.Uptake of host cell transforming growth factor-beta by Trypanosoma cruzi amastigotes in cardiomyocytes: potential role in parasite cycle completion.Chagas heart disease: report on recent developments.Altered Hypercoagulability Factors in Patients with Chronic Chagas Disease: Potential Biomarkers of Therapeutic Response.Trypanosoma cruzi invades host cells through the activation of endothelin and bradykinin receptors: a converging pathway leading to chagasic vasculopathy.Myocardial tissue characterization in Chagas' heart disease by cardiovascular magnetic resonance.The vasculature in chagas disease.Pharmacological inhibition of transforming growth factor beta signaling decreases infection and prevents heart damage in acute Chagas' disease Perspectives on Trypanosoma cruzi-induced heart disease (Chagas disease).Mast Cell Coupling to the Kallikrein-Kinin System Fuels Intracardiac Parasitism and Worsens Heart Pathology in Experimental Chagas Disease.Effects of Trypanosoma cruzi infection on myocardial morphology, single cardiomyocyte contractile function and exercise tolerance in rats.Host cell invasion by Trypanosoma cruzi is potentiated by activation of bradykinin B(2) receptors.The kallikrein-kinin system in experimental Chagas disease: a paradigm to investigate the impact of inflammatory edema on GPCR-mediated pathways of host cell invasion by Trypanosoma cruzi.Infectious myocarditis: the role of the cardiac vasculature.

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P2860

Different microcirculatory and interstitial matrix patterns in idiopathic dilated cardiomyopathy and Chagas' disease: a three dimensional confocal microscopy study

http://www.wikidata.org/entity/Q30755828

description

1999 nî lūn-bûn

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1999 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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1999 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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1999年論文

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1999年論文

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1999年論文

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1999年論文

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Different microcirculatory and ...... onal confocal microscopy study

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Different microcirculatory and ...... onal confocal microscopy study

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Different microcirculatory and ...... onal confocal microscopy study

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Different microcirculatory and ...... onal confocal microscopy study

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Different microcirculatory and ...... onal confocal microscopy study

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G Bellotti

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J A Ramires

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L C Parzianello

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M L Higuchi

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P2860

Differential staining of collagens type I, II and III by Sirius Red and polarization microscopy.

Structure and function of connective tissue in cardiac muscle: collagen types I and III in endomysial struts and pericellular fibers.

Enhanced platelet adherence and aggregation in Chagas' disease: a potential pathogenic mechanism for cardiomyopathy.

Morphology, composition, and function of struts between cardiac myocytes of rat and hamster.

Experimental Trypanosoma cruzi cardiomyopathy in BALB/c mice. The potential role of intravascular platelet aggregation in its genesis.

Enhanced deposition of predominantly type I collagen in myocardial disease.

Increased capacity of the coronary arteries in chronic Chagas' heart disease: further support for the neurogenic pathogenesis concept.

Patterns of myocardial fibrosis in idiopathic cardiomyopathies and chronic Chagasic cardiopathy.

A metallic impregnation method for the demonstration of cerebral vascular patterns.

Interstitial connective tissue matrix alterations in acute murine Chagas' disease

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