Interferon-gamma reduces melanosomal antigen expression and recognition of melanoma cells by cytotoxic T cells. - Wikidata - wikimedia - TriplyDB

Interferon-gamma reduces melanosomal antigen expression and recognition of melanoma cells by cytotoxic T cells.

Interferon-gamma reduces melanosomal antigen expression and recognition of melanoma cells by cytotoxic T cells.

http://www.wikidata.org/entity/Q30811481

about

Biological response modifiers in cancer MHC class II presentation of gp100 epitopes in melanoma cells requires the function of conventional endosomes and is influenced by melanosomes.microRNA-155, induced by interleukin-1ß, represses the expression of microphthalmia-associated transcription factor (MITF-M) in melanoma cells.Significance of the cytokine interferon gamma in clinical dermatology.The Silver locus product Pmel17/gp100/Silv/ME20: controversial in name and in function.Reduced skin homing by functional Treg in vitiligo.A Well-Controlled Experimental System to Study Interactions of Cytotoxic T Lymphocytes with Tumor Cells Immune escape mechanisms of intraocular tumors.IFN-γ signaling maintains skin pigmentation homeostasis through regulation of melanosome maturation Intravenous immunoglobulin-based immunotherapy: an arsenal of possibilities for patients and science.Achievements and challenges of adoptive T cell therapy with tumor-infiltrating or blood-derived lymphocytes for metastatic melanoma: what is needed to achieve standard of care?Interleukins 1alpha and 1beta secreted by some melanoma cell lines strongly reduce expression of MITF-M and melanocyte differentiation antigens.Prediction of pairwise gene interaction using threshold logic.Lysis of human chondrosarcoma cells by cytolytic T lymphocytes recognizing a MAGE-A3 antigen presented by HLA-A1 molecules.Interferon-gamma differentially regulates susceptibility of lung cancer cells to telomerase-specific cytotoxic T lymphocytes.A coreceptor-independent transgenic human TCR mediates anti-tumor and anti-self immunity in mice.Genetic variation in IRF4 expression modulates growth characteristics, tyrosinase expression and interferon-gamma response in melanocytic cells.Revisiting cancer immunoediting by understanding cancer immune complexity.The Dark Side of IFN-γ: Its Role in Promoting Cancer Immunoevasion.Sustained low-level expression of interferon-gamma promotes tumor development: potential insights in tumor prevention and tumor immunotherapy

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Interferon-gamma reduces melanosomal antigen expression and recognition of melanoma cells by cytotoxic T cells.

http://www.wikidata.org/entity/Q30811481

description

2002 nî lūn-bûn

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2002 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2002 թվականի փետրվարին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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Interferon-gamma reduces melan ...... ma cells by cytotoxic T cells.

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Interferon-gamma reduces melan ...... ma cells by cytotoxic T cells.

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Interferon-gamma reduces melan ...... ma cells by cytotoxic T cells.

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Interferon-gamma reduces melan ...... ma cells by cytotoxic T cells.

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The American Journal of Pathology

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Interferon-gamma reduces melan ...... ma cells by cytotoxic T cells.

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Adam I Riker

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Brian J Nickoloff

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Ena Wang

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Francesco M Marincola

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I Caroline Le Poole

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June K Robinson

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Lawrence S Stennett

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M Eugenia Quevedo

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W Martin Kast

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P2860

The p44S10 locus, encoding a subunit of the proteasome regulatory particle, is amplified during progression of cutaneous malignant melanoma

Analysis of MAGE-3-specific cytolytic T lymphocytes in human leukocyte antigen-A2 melanoma patients

Cellular responses to interferon-gamma

Melanomas with concordant loss of multiple melanocytic differentiation proteins: immune escape that may be overcome by targeting unique or undefined antigens.

alpha-melanocyte-stimulating hormone as a mediator of tolerance induction.

Processing of some antigens by the standard proteasome but not by the immunoproteasome results in poor presentation by dendritic cells.

Local immune response in skin of generalized vitiligo patients. Destruction of melanocytes is associated with the prominent presence of CLA+ T cells at the perilesional site.

Down-regulation of HLA class I antigen-processing molecules in malignant melanoma: association with disease progression

Developing recombinant and synthetic vaccines for the treatment of melanoma.

Aberrant retention of tyrosinase in the endoplasmic reticulum mediates accelerated degradation of the enzyme and contributes to the dedifferentiated phenotype of amelanotic melanoma cells

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521-528

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10.1016/S0002-9440(10)64871-7

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