Switch to anaerobic glucose metabolism with NADH accumulation in the beta-cell model of mitochondrial diabetes. Characteristics of betaHC9 cells deficient in mitochondrial DNA transcription.
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The central role of calcium in the effects of cytokines on beta-cell function: implications for type 1 and type 2 diabetesPrenatal programming of insulin secretion in intrauterine growth restrictionThe malate-aspartate NADH shuttle member Aralar1 determines glucose metabolic fate, mitochondrial activity, and insulin secretion in beta cellsIntracellular coenzymes as natural biomarkers for metabolic activities and mitochondrial anomaliesMitochondrial GTP regulates glucose-stimulated insulin secretionImpaired expression of NADH dehydrogenase subunit 1 and PPARgamma coactivator-1 in skeletal muscle of ZDF rats: restoration by troglitazoneSelective actions of mitochondrial fission/fusion genes on metabolism-secretion coupling in insulin-releasing cellsRegulation of insulin secretion: role of mitochondrial signalling.Glucose sensing in the pancreatic beta cell: a computational systems analysis.Developmental origins of adult disease.Developmental origins of diabetes: The role of oxidative stressChronic pulsatile hyperglycemia reduces insulin secretion and increases accumulation of reactive oxygen species in fetal sheep islets.ESCI Award 2006. Mitochondrial function and endocrine diseases.Decreased NAA in gray matter is correlated with decreased availability of acetate in white matter in postmortem multiple sclerosis cortex.Altered expression of uncoupling protein 2 in GLP-1-producing cells after chronic high glucose exposure: implications for the pathogenesis of diabetes mellitus.DAPK2 regulates oxidative stress in cancer cells by preserving mitochondrial function.Self-immolative bioluminogenic quinone luciferins for NAD(P)H assays and reducing capacity-based cell viability assays.Inhibition of mitochondrial gene transcription suppresses neurotensin secretion in the human carcinoid cell line BON.Hepatocyte nuclear factor-4alpha is essential for glucose-stimulated insulin secretion by pancreatic beta-cells.The loss of Sirt1 in mouse pancreatic beta cells impairs insulin secretion by disrupting glucose sensing.Depletion of homeodomain-interacting protein kinase 3 impairs insulin secretion and glucose tolerance in mice.A common mechanism explains the induction of aerobic fermentation and adaptive antioxidant response in Phaffia rhodozyma.
P2860
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P2860
Switch to anaerobic glucose metabolism with NADH accumulation in the beta-cell model of mitochondrial diabetes. Characteristics of betaHC9 cells deficient in mitochondrial DNA transcription.
description
2002 nî lūn-bûn
@nan
2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年学术文章
@wuu
2002年学术文章
@zh-cn
2002年学术文章
@zh-hans
2002年学术文章
@zh-my
2002年学术文章
@zh-sg
2002年學術文章
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name
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@ast
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@en
type
label
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@ast
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@en
prefLabel
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@ast
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@en
P2093
P2860
P921
P356
P1476
Switch to anaerobic glucose me ...... tochondrial DNA transcription.
@en
P2093
Geoffrey W G Sharp
Haruo Kasai
Kazuhiro Eto
Kazuo Izumi
Lin-Ming Shen
Masamitsu Iino
Mitsuhiko Noda
Noriko Takahashi
Samira Daniel
Shigeo Yamashita
P2860
P304
41817-41826
P356
10.1074/JBC.M207690200
P407
P577
2002-08-06T00:00:00Z