Combined toll-like receptor 3/7/9 deficiency on host cells results in T-cell-dependent control of tumour growth
about
Aberrant control of NF-κB in cancer permits transcriptional and phenotypic plasticity, to curtail dependence on host tissue: molecular mode.IFI16 restoration in hepatocellular carcinoma induces tumour inhibition via activation of p53 signals and inflammasome.Sipa1 deficiency unleashes a host-immune mechanism eradicating chronic myelogenous leukemia-initiating cells.
P2860
Combined toll-like receptor 3/7/9 deficiency on host cells results in T-cell-dependent control of tumour growth
description
2017 nî lūn-bûn
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2017 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2017 թվականի մարտին հրատարակված գիտական հոդված
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2017年の論文
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2017年論文
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2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
name
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@ast
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@en
type
label
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@ast
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@en
prefLabel
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@ast
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@en
P2093
P2860
P356
P1476
Combined toll-like receptor 3/ ...... ndent control of tumour growth
@en
P2093
Carsten J Kirschning
Gennadiy Zelinskyy
Iris Helfrich
Johanna C Klein
Katrin Moses
Simon Sody
Stephan Lang
Sven Brandau
P2860
P2888
P356
10.1038/NCOMMS14600
P407
P50
P577
2017-03-16T00:00:00Z
P6179
1084128837