Pertussis toxin stimulates IL-17 production in response to Bordetella pertussis infection in mice.
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Repetitive pertussis toxin promotes development of regulatory T cells and prevents central nervous system autoimmune diseasePertussis toxin and adenylate cyclase toxin: key virulence factors of Bordetella pertussis and cell biology toolsBacterial Toxins as Pathogen Weapons Against PhagocytesNovel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway DiseasesToward a mechanism-based in vitro safety test for pertussis toxinReview of the neutrophil response to Bordetella pertussis infection.Critical pertussis illness in children: a multicenter prospective cohort study.Contribution of Bordetella filamentous hemagglutinin and adenylate cyclase toxin to suppression and evasion of interleukin-17-mediated inflammation.Bordetella pertussis proteins dominating the major histocompatibility complex class II-presented epitope repertoire in human monocyte-derived dendritic cellsRole of IL-17A on resolution of pulmonary C. neoformans infectionPeripheral and central neuronal ATF3 precedes CD4+ T-cell infiltration in EAE.Bordetella pertussis infection exacerbates influenza virus infection through pertussis toxin-mediated suppression of innate immunityExperimental autoimmune encephalomyelitis as a testing paradigm for adjuvants and vaccinesEpithelial anion transporter pendrin contributes to inflammatory lung pathology in mouse models of Bordetella pertussis infectionAcellular pertussis vaccines protect against disease but fail to prevent infection and transmission in a nonhuman primate model.A Type VI secretion system encoding locus is required for Bordetella bronchiseptica immunomodulation and persistence in vivo.Interleukin-1 receptor signaling is required to overcome the effects of pertussis toxin and for efficient infection- or vaccination-induced immunity against Bordetella pertussis.Depletion of neutrophils in a protective model of pulmonary cryptococcosis results in increased IL-17A production by γδ T cells.Differential expression of alpha 4 integrins on effector memory T helper cells during Bordetella infections. Delayed responses in Bordetella pertussisRelative contribution of Th1 and Th17 cells in adaptive immunity to Bordetella pertussis: towards the rational design of an improved acellular pertussis vaccine.A live, attenuated Bordetella pertussis vaccine provides long-term protection against virulent challenge in a murine model.Resident microbiota affect Bordetella pertussis infectious dose and host specificity.Tailored immune responses: novel effector helper T cell subsets in protective immunity.Formulation of a killed whole cell pneumococcus vaccine - effect of aluminum adjuvants on the antibody and IL-17 response.Controlled inflammatory responses in the lungs are associated with protection elicited by a pneumococcal surface protein A-based vaccine against a lethal respiratory challenge with Streptococcus pneumoniae in mice.Pertussis toxin exacerbates and prolongs airway inflammatory responses during Bordetella pertussis infection.IL-1-induced Bhlhe40 identifies pathogenic T helper cells in a model of autoimmune neuroinflammation.Decreased leukocyte accumulation and delayed Bordetella pertussis clearance in IL-6-/- micePendrin, an anion exchanger on lung epithelial cells, could be a novel target for lipopolysaccharide-induced acute lung injury mice.Intra-amniotic IL-1β induces fetal inflammation in rhesus monkeys and alters the regulatory T cell/IL-17 balanceB cell-intrinsic and -extrinsic regulation of antibody responses by PARP14, an intracellular (ADP-ribosyl)transferase.Precarious balance: Th17 cells in host defense.Neutrophils and macrophages work in concert as inducers and effectors of adaptive immunity against extracellular and intracellular microbial pathogens.Attenuated Bordetella pertussis vaccine protects against respiratory syncytial virus disease via an IL-17-dependent mechanism.IL-17 family member cytokines: regulation and function in innate immunity.The ins and outs of pertussis toxin.Immunity to the respiratory pathogen Bordetella pertussis.The virulence factors of Bordetella pertussis: talented modulators of host immune response.Improved pertussis vaccines based on adjuvants that induce cell-mediated immunity.The baboon model of pertussis: effective use and lessons for pertussis vaccines.
P2860
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P2860
Pertussis toxin stimulates IL-17 production in response to Bordetella pertussis infection in mice.
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@ast
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@en
type
label
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@ast
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@en
prefLabel
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@ast
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@en
P2860
P1433
P1476
Pertussis toxin stimulates IL- ...... a pertussis infection in mice.
@en
P2093
Charlotte Andreasen
Nicholas H Carbonetti
P2860
P356
10.1371/JOURNAL.PONE.0007079
P407
P577
2009-09-17T00:00:00Z