Loss of parietal cell expression of Sonic hedgehog induces hypergastrinemia and hyperproliferation of surface mucous cells. - Wikidata - wikimedia - TriplyDB

Loss of parietal cell expression of Sonic hedgehog induces hypergastrinemia and hyperproliferation of surface mucous cells.

Loss of parietal cell expression of Sonic hedgehog induces hypergastrinemia and hyperproliferation of surface mucous cells.

http://www.wikidata.org/entity/Q30883814

about

Stomach development, stem cells and disease Helicobacter pylori-induced Sonic Hedgehog expression is regulated by NFκB pathway activation: the use of a novel in vitro model to study epithelial response to infection Transgenic and gene knockout mice in gastric cancer research Mesenchymal stem cells induce epithelial proliferation within the inflamed stomach.Mouse models of gastric carcinogenesis.The transcription factor MIST1 is a novel human gastric chief cell marker whose expression is lost in metaplasia, dysplasia, and carcinoma.Gastric epithelial stem cells Hedgehog signaling regulates E-cadherin expression for the maintenance of the actin cytoskeleton and tight junctions.Cytodifferentiation of the postnatal mouse stomach in normal and Huntingtin-interacting protein 1-related-deficient mice.Crosstalks between cytokines and Sonic Hedgehog in Helicobacter pylori infection: a mathematical model.Anti-acid medication as a risk factor for food allergy.Epithelial BMP signaling is required for proper specification of epithelial cell lineages and gastric endocrine cells.Indian Hedgehog mediates gastrin-induced proliferation in stomach of adult mice.[Continual self-renewal of the gastric epithelium by cell differentiation: implications for carcinogenesis].A high-fat diet regulates gastrin and acid secretion through primary cilia.Gastric stem cells and gastric cancer stem cells.The hyaluronic acid receptor CD44 coordinates normal and metaplastic gastric epithelial progenitor cell proliferation Mouse models of gastric cancer.Stomach Organ and Cell Lineage Differentiation: from Embryogenesis to Adult Homeostasis.The acid-secreting parietal cell as an endocrine source of Sonic Hedgehog during gastric repair Mouse models for gastric cancer: Matching models to biological questions.Hedgehog signaling and gastrointestinal cancer The role of sonic hedgehog reemergence during gastric cancer Hedgehog signalling in gut development, physiology and cancer.Role of Sonic Hedgehog signaling during progression from inflammation to cancer in the stomach.The role of Sonic Hedgehog as a regulator of gastric function and differentiation.Gastric cancer stem cells: therapeutic targets.Hedgehog signaling in the stomach.Hedgehog Signaling Links Chronic Inflammation to Gastric Cancer Precursor Lesions.From inflammation to gastric cancer - the importance of Hedgehog/GLI signaling in Helicobacter pylori-induced chronic inflammatory and neoplastic diseases.Hedgehog signaling pathway as a potential target in the treatment of advanced gastric cancer.XBP1 controls maturation of gastric zymogenic cells by induction of MIST1 and expansion of the rough endoplasmic reticulum Intracellular calcium release and protein kinase C activation stimulate sonic hedgehog gene expression during gastric acid secretion.Gastric tuft cells express DCLK1 and are expanded in hyperplasia.Manifestation of spontaneous and early autoimmune gastritis in CCR7-deficient mice Gastric Sonic Hedgehog acts as a macrophage chemoattractant during the immune response to Helicobacter pylori.Sonic Hedgehog contributes to gastric mucosal restitution after injury.Gastritis promotes an activated bone marrow-derived mesenchymal stem cell with a phenotype reminiscent of a cancer-promoting cell.Directed transdifferentiation of Müller glial cells to photoreceptors using the sonic hedgehog signaling pathway agonist purmorphamine.Mechanisms of angiogenesis in microbe-regulated inflammatory and neoplastic conditions.

P2860

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P2860

Loss of parietal cell expression of Sonic hedgehog induces hypergastrinemia and hyperproliferation of surface mucous cells.

http://www.wikidata.org/entity/Q30883814

description

2009 nî lūn-bûn

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2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Loss of parietal cell expressi ...... ation of surface mucous cells.

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Loss of parietal cell expressi ...... ation of surface mucous cells.

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type

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Loss of parietal cell expressi ...... ation of surface mucous cells.

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Loss of parietal cell expressi ...... ation of surface mucous cells.

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Loss of parietal cell expressi ...... ation of surface mucous cells.

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J.GASTRO.2009.11.002

P1433

Gastroenterology

P1476

Loss of parietal cell expressi ...... ation of surface mucous cells.

@en

P2093

Andrea Varro

http://www.w3.org/2001/XMLSchema#string

Chang Xiao

http://www.w3.org/2001/XMLSchema#string

Frederic Hollande

http://www.w3.org/2001/XMLSchema#string

Marian L Miller

http://www.w3.org/2001/XMLSchema#string

Melissa A Orr-Asman

http://www.w3.org/2001/XMLSchema#string

Michael A Schumacher

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Nantaporn Lertkowit

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Sally A Ogle

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Yana Zavros

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P2860

The gastric epithelial progenitor cell niche and differentiation of the zymogenic (chief) cell lineage.

The maturation of mucus-secreting gastric epithelial progenitors into digestive-enzyme secreting zymogenic cells requires Mist1

Hip1r is expressed in gastric parietal cells and is required for tubulovesicle formation and cell survival in mice

Cholesterol modification of sonic hedgehog is required for long-range signaling activity and effective modulation of signaling by Ptc1

Snail induction is an early response to Gli1 that determines the efficiency of epithelial transformation

Convergence of Wnt, beta-catenin, and cadherin pathways

Snail and Slug promote epithelial-mesenchymal transition through beta-catenin-T-cell factor-4-dependent expression of transforming growth factor-beta3

A molecular signature of gastric metaplasia arising in response to acute parietal cell loss

Familial giant hypertrophic gastritis (Ménétrier's disease).

Gli1 acts through Snail and E-cadherin to promote nuclear signaling by beta-catenin

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550-61, 561.e1-8

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scholarly article

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10.1053/J.GASTRO.2009.11.002

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2

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138

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4269486

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