Administration of the soluble complement inhibitor, Crry-Ig, reduces inflammation and aquaporin 4 expression in lupus cerebritis.
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Do nutritional supplements have a role in age macular degeneration prevention?Novel variants in human Aquaporin-4 reduce cellular water permeabilityHuman Aquaporin-4 and Molecular Modeling: Historical Perspective and View to the FutureManagement of Neuropsychiatric Systemic Lupus Erythematosus: Current Approaches and Future PerspectivesThe MRL/lpr mouse strain as a model for neuropsychiatric systemic lupus erythematosus.The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.C5a alters blood-brain barrier integrity in experimental lupus.Targeted complement inhibition by C3d recognition ameliorates tissue injury without apparent increase in susceptibility to infection.Inhibition of C5a receptor alleviates experimental CNS lupus.C3aR inhibition reduces neurodegeneration in experimental lupus.TNF is a key mediator of septic encephalopathy acting through its receptor, TNF receptor-1.C5a/CD88 signaling alters blood-brain barrier integrity in lupus through nuclear factor-κBTransient life-threatening cerebral edema in a patient with systemic lupus erythematosus.The relative merits of therapies being developed to tackle inappropriate ('self'-directed) complement activationNeuroimmunopathology in a murine model of neuropsychiatric lupusPurine receptor antagonist modulates serology and affective behaviors in lupus-prone mice: evidence of autoimmune-induced pain?Pharmacotherapy of systemic lupus erythematosus.Chance, genetics, and the heterogeneity of disease and pathogenesis in systemic lupus erythematosus.Sugar-free, glycine-stabilized intravenous immunoglobulin prevents skin but not renal disease in the MRL/lpr mouse model of systemic lupus.Neuromyelitis optica: pathogenicity of patient immunoglobulin in vivo.Expression and function of water channels (aquaporins) in migrating malignant astrocytes.C5a alters blood-brain barrier integrity in a human in vitro model of systemic lupus erythematosus.Abnormal olfactory function demonstrated by manganese-enhanced MRI in mice with experimental neuropsychiatric lupus.C5a induces caspase-dependent apoptosis in brain vascular endothelial cells in experimental lupus.Absence of functional alternative complement pathway alleviates lupus cerebritis.Neuropsychiatric SLE: from animal model to human.Complement levels and anti-C1q autoantibodies in patients with neuropsychiatric systemic lupus erythematosus.
P2860
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P2860
Administration of the soluble complement inhibitor, Crry-Ig, reduces inflammation and aquaporin 4 expression in lupus cerebritis.
description
2003 nî lūn-bûn
@nan
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Administration of the soluble ...... xpression in lupus cerebritis.
@ast
Administration of the soluble ...... xpression in lupus cerebritis.
@en
type
label
Administration of the soluble ...... xpression in lupus cerebritis.
@ast
Administration of the soluble ...... xpression in lupus cerebritis.
@en
prefLabel
Administration of the soluble ...... xpression in lupus cerebritis.
@ast
Administration of the soluble ...... xpression in lupus cerebritis.
@en
P2093
P1476
Administration of the soluble ...... xpression in lupus cerebritis.
@en
P2093
Alexander Jacob
Damien M Kraus
Jessy J Alexander
Richard J Quigg
V Michael Holers
P304
P356
10.1016/J.BBADIS.2003.09.005
P407
P577
2003-11-01T00:00:00Z